Does Smoking by Pregnant Women Influence IQ, Birth Weight, and Developmental Disabilities in Their Infants? A Methodological Review and Multivariate Analysis

Neuropsychologists are asked frequently to address the issue of the cause of a variety of central nervous system problems that may affect higher cortical function. One such issue is the relationship of maternal smoking to adverse reproductive outcomes involving neocortical insult including mental retardation, learning disabilities, attention-deficit hyperactivity disorder, and other insults that may be related to prolonged hypoxic states in utero. The instant paper develops the issue of causation as a scientific inquiry, reviews several traditional, applicable models, and critiques these models. An additional model of motility is proposed and discussed. The issue of the relationship of maternal smoking to adverse reproductive outcomes is then addressed from a review perspective along with new empirical analyses, the latter demonstrating that researchers tend to draw causal conclusions independent of whether the respective design of their studies would support conclusions about the causation of an event. Causal conclusions in the absence of causal designs have often lead to incomplete and incorrect conclusions. It is necessary to match conclusions not only to the outcomes of a research project but also to its design and accompanying limitations.     

A puzzle about mental self-representation and causation

The paper articulates a puzzle that consists in the prima facie incompatibility between three widely accepted theses. The first thesis is, roughly, that there are intrinsically self-representational thoughts. The second thesis is, roughly, that there is a particular causal constraint on mental representation. The third thesis is, roughly, that nothing causes itself. In this paper, the theses are articulated in a less rough manner with the occurrence of the puzzle as a result. Finally, a number of solution strategies are considered, and a preliminary diagnosis is provided.     

Judging the Goring Ox: Retribution Directed Toward Animals

Prior research on the psychology of retribution is complicated by the difficulty of separating retributive and general deterrence motives when studying human offenders (Study 1). We isolate retribution by investigating judgments about punishing animals, which allows us to remove general deterrence from consideration. Studies 2 and 3 document a “victim identity” effect, such that the greater the perceived loss from a violent animal attack, the greater the belief that the culprit deserves to be killed. Study 3 documents a “targeted punishment” effect, such that the responsive killing of the actual “guilty” culprit is seen as more deserved than the killing of an almost identical yet “innocent” animal from the same species. Studies 4 and 5 extend both effects to participants' acceptance of inflicting pain and suffering on the offending animal at the time of its death, and show that both effects are mediated by measures of retributive sentiment, and not by consequentialist concerns.     

Moral inertia

I argue that, according to ordinary morality, there is moral inertia, that is, moral pressure to fail to intervene in certain circumstances. Moral inertia is manifested in scenarios with a particular causal structure: deflection scenarios, where a threatening or benefiting process is diverted from a group of people to another. I explain why the deflection structure is essential for moral inertia to be manifested. I argue that there are two different manifestations of moral inertia: strict prohibitions on interventions, and constraints on interventions. Finally, I discuss the connection between moral inertia and the distinction between killing and letting die (or doing and allowing harm).

The probability of causal conditionals

Conditionals in natural language are central to reasoning and decision making. A theoretical proposal called the Ramsey test implies the conditional probability hypothesis: that the subjective probability of a natural language conditional, P(if p then q), is the conditional subjective probability, P(q/p). We report three experiments on causal indicative conditionals and related counterfactuals that support this hypothesis. We measured the probabilities people assigned to truth table cases, P(pq), P(p notq), P( notpq) and P( notp notq). From these ratings, we computed three independent predictors, P(p), P(q/p) and P(q/ notp), that we then entered into a regression equation with judged P(if p then q) as the dependent variable. In line with the conditional probability hypothesis, P(q/p) was by far the strongest predictor in our experiments. This result is inconsistent with the claim that causal conditionals are the material conditionals of elementary logic. Instead, it supports the Ramsey test hypothesis, implying that common processes underlie the use of conditionals in reasoning and judgments of conditional probability in decision making.     

Three obstructions: forms of causation,chronotopoids, and levels of reality

The thesis is defended that the theories of causation, time and space, and levels of reality are mutually interrelated in such a way that the difficulties internal to theories of causation and to theories of space and time can be understood better, and perhaps dealt with, in the categorial context furnished by the theory of the levels of reality. The structural condition for this development to be possible is that the first two theories be opportunely generalized.

Structural equations and causation: six counterexamples

Hall [(2007), Philosophical Studies, 132, 109–136] offers a critique of structural equations accounts of actual causation, and then offers a new theory of his own. In this paper, I respond to Hall’s critique, and present some counterexamples to his new theory. These counterexamples are then diagnosed.

More Troubles for Epiphenomenalism

I have argued that to say qualia are epiphenomenal is to say a world without qualia would be physically identical to a world with qualia. Dan Cavedon-Taylor has offered an alternative interpretation of the commitments of qualia epiphenomenalism according to which qualia cause beliefs and those beliefs can and do cause changes to the physical world. I argue that neither of these options works for the qualia epiphenomenalist and thus that theory faces far more serious difficulties than has previously been recognized.

No Time, No Wholes: A Temporal and Causal-Oriented Approach to the Ontology of Wholes

What distinguishes a whole from an arbitrary sum of elements? I suggest a temporal and causal oriented approach. I defend two connected claims. The former is that existence is, by every means, coextensive with being the cause of a causal process. The latter is that a whole is the cause of a causal process with a joint effect. Thus, a whole is something that takes place in time. The approach endorses an unambiguous version of Restricted Composition that suits most commonsensical intuitions about wholes.