No Evidence of Suicide Increase Following Terrorist Attacks in the United States: An Interrupted Time‐Series Analysis of September 11 and Oklahoma City

There is substantial evidence of detrimental psychological sequelae following disasters, including terrorist attacks. The effect of these events on extreme responses such as suicide, however, is unclear. We tested competing hypotheses about such effects by employing autoregressive integrated moving average techniques to model the impact of September 11 and the Oklahoma City bombing on monthly suicide counts at the local, state, and national level. Unlike prior studies that provided conflicting evidence, rigorous time series techniques revealed no support for an increase or decrease in suicides following these events. We conclude that while terrorist attacks produce subsequent psychological morbidity and may affect self and collective efficacy well beyond their immediate impact, these effects are not strong enough to influence levels of suicide mortality.     

The role of illness uncertainty on coping with fibromyalgia symptoms.

This study examined the role of illness uncertainty in pain coping among women with fibromyalgia (FM), a chronic pain condition of unknown origin. Fifty-one FM participants completed initial demographic and illness uncertainty questionnaires and underwent 10-12 weekly interviews regarding pain, coping difficulty, and coping efficacy. Main outcome measures included weekly levels of difficulty coping with FM symptoms and coping efficacy. Multilevel analyses indicated that pain elevations for those high in illness uncertainty predicted increases in coping difficulty. Furthermore, when participants had more difficulty coping, they reported lower levels of coping efficacy. Results were consistent with hypothesized effects. Illness uncertainty accompanied by episodic pain negatively influenced coping efficacy, an important resource in adaptation to FM.     

Methods for improving the signal-to-noise ratio of endogenous-evoked potentials

A stimulus leads to a cortical response (i.e., evoked potential [EP]) which may be recorded from electrodes attached to the scalp. However, background cortical activity, considered as noise (n), is typically of equal or greater magnitude than the response, which is considered as signal (s). This situation leads to the masking of the presence of the electrocortical signal. Two methods are described which enable the enhancement of the signal with respect to the noise. The first method outlined is time-domain averaging. Its relation to Fourier averaging is also presented. Time averaging can lead to an enhancement of the signal with respect to the noise, known as the signal-to-noise ratio (SNR), by a factor of root N; N being the number of trials recorded. However, latency variability (i.e., jitter) present in the signal leads to a decrement in this maximal potential enhancement. The second technique is an adaptive filter method of averaged cross-correlations, developed by Woody (1967), which deals with the variable latency problem. The development of a latency corrected average developed by McGillem and Aunon (1977) is also presented. The final section describes methods for data handling once the electrocortical signal has been enhanced. It is then necessary to describe the EP quantitatively. Principal Components Analysis (PCA) allows for the quantitative compact representation of the evoked potential waveform. The method also allows for the testing of the effects of explanatory variables on the EP.     

A brief neuropsychological screening battery to detect brain damage in a psychiatric population

Seventy-five patients admitted to the psychiatric unit of a general hospital were administered a brief neuropsychological screening battery. Discharge diagnoses obtained from the medical records revealed that 20% had brainbased pathology. A discriminant function analysis resulted in a five-variable model (Symbol-Digit Modalities Test, educational level, Psychiatric-Organic subscale of the MMPI, and Shipley Vocabulary and Abstraction scores) which correctly classified 84% of the subjects, with 87% correct identification of the brain-damaged group. A jackknife cross-validation procedure resulted in comparable hit rates.     

Divine Relations as a Basis of Subjective Social Status During Later-Life: Direct and Moderating Effects

Early social theorists, including W.E.B Du Bois, recognized the importance of religion and its links to inequality, particularly in how religious vocabularies are attuned to frictions with inimical concrete social and political realities. We apply these ideas to research on the intersection of religious beliefs and economic deprivation, examining how beliefs in divine relations not only structure subjective social status (SSS), but also the association between financial stress and SSS. Analyses of data from the Caregiving, Aging, and Financial Experiences Study—a national study of Canadian older adults (N = 4010)—show that financial strain is associated with lower SSS, but better divine relations (higher support from a divine power and lower troubled relationships with a higher power) benefit SSS. Divine support also attenuates the association between financial strain and SSS. This research suggests a Du Bosian “double consciousness,” in which relations with a divine power serve as a basis for individual conceptualizations of social status that run counter to predominant narratives based on economic standing. We suggest directions for future research to explore the nuances of SSS within the religious context informed by a Du Boisian mode of inquiry.     

Mapping physical characteristics in face images to social judgements

Sutherland and Young (Br. J. Psychol., 113, 2022, 1056) provide a comprehensive and timely overview of recent developments in research on social judgements of faces, emphasizing the utility of data-driven approaches. Here, we expand on this theme, focusing on how data-driven approaches can provide new insights into the physical characteristics in face images that best predict social judgements.     

Sind die klassische Mechanik und die spezielle Relativitätstheorie kommensurabel?

In its first part, this paper shows why a recently made attempt to reduce the special theory of relativity to Newtonian kinematics is bound to fail. In the second part, we propose a differentiated notion of incommensurability which enables us to amend the contention that the special theory of relatively and Newtonian kinematics are incommensurable.     

Effects of Acute Stress or Centrally Injected Interleukin-1beta on Neuropeptide Expression in the Immune System

Acute stress stimulates the expression and release of corticotropin-releasing hormone (CRH) and arginine vasopressin (AVP) from the hypothalamus, and the pro-opiomelanocortin products beta-endorphin and ACTH from the anterior pituitary. These neuropeptides are also expressed in immune tissues, and it has been proposed that they may modulate immune responses to stress through paracrine mechanisms. We subjected rats to restraint stress or central injection of interleukin (IL)-1beta to determine whether these acute stimuli can alter the expression of neuropeptides in the spleen and thymus. Restraint stress significantly increased the contents of all these neuropeptides in thymic, but not splenic, extracts. A single icv injection of IL-1beta increased contents of CRH, AVP, ACTH and beta-endorphin in the spleens of both sham-operated and adrenalectomised (ADX) rats. IL-1beta increased thymic contents of CRH and ACTH in sham-operated rats but these increases were not observed in ADX rats. These results suggest that the effects of IL-1beta on neuropeptide expression in the spleen are independent of glucocorticoids, whereas IL-1beta stimulation of neuropeptide expression in the thymus is dependent on circulating glucocorticoids. There were significant correlations between increases in CRH, ACTH and beta-endorphin in the spleen, and between CRH and ACTH in the thymus, consistent with the suggestion that IL-1beta-induced increases in ACTH and beta-endorphin may be mediated through CRH. These results provide evidence that stressors can directly influence neuropeptide expression in immune tissues. Thus stress may influence immune functions through paracrine mechanisms involving locally synthesised neuropeptides as well as through activation of the hypothalamo-pituitary-adrenal axis.