Effects of Subcortical Lesion Site on Human Emotional Behavior

This is the first quantitative group study of recognition, expression, and other social-emotional behaviors after third ventricle damage in humans. Hypothalamic damage often induced appetitive disorders without affecting other emotional functions. In contrast, lesions in several hemispheric sites, especially right medial temporal, impaired emotional recognition; frontal and basal ganglia damage impoverished facial emotional expressions, this deficit being dissociated from general facial-motor impairment. These functional dissociations indicate that social-emotional behavior is regulated by multiple neural circuits, many of which by-pass hypothalamic nuclei. This conclusion is shown to be consistent with (a) the considerable animal evidence linking appetitive-survival behaviors with hypothalamic activity and (b) the accumulating human data on the specific contributions of hemispheric cortical-subcortical systems in the regulation of emotional recognition and expression.     

Psychotherapie und Internet

Zusammenfassung  Die Außendarstellung im Internet ist wichtig und in der Zukunft unverzichtbar. Jedoch hat die Liberalisierung des Werberechts der freien Berufe keineswegs zur absoluten Schrankenlosigkeit geführt. Vielmehr sind die zu beachtenden Regeln auf verschiedene Gesetze und Rechtsgebiete verstreut, so dass ohne fachkundige Hilfe—etwa durch die Kammern oder durch juristische Beratung—die Selbstdarstellung im Internet schnell zu einem bösen Erwachen führen kann.

Victimization and PTSD in a Faroese youth total-population sample

Petersen, T., Elklit, A. & Olesen, J. G. (2010). Victimization and PTSD in a Faroese youth total-population sample. Scandinavian Journal of Psychology, 51, 56–62.
The prevalence of twenty traumatic events and negative life events in relation to posttraumatic stress disorder (PTSD) was studied in a Faroese total-population sample of 687 eighth-grade students with a mean age of 14.2 years. Ninety-four percent of the females and 89% of the males were directly exposed to or had witnessed at least one traumatic event or a negative life event. The odds ratios for PTSD after direct and indirect exposure to specific events are described. The lifetime prevalence of PTSD was 20%, whereas another 14% reached a subclinical level of PTSD. After exposure, females had PTSD more than twice as often as males. Being exposed to multiple traumatic events, living with a single parent, and having experienced a traumatic event or a negative life event within the last year were all associated with PTSD and its subscales.     

Sequential resolution of fragmented visual percepts: experimental investigation of a subject's perceptual experience after a right medial temporal stroke

This report concerns the fragmented visual percepts in a woman, TR, following a right entorhinal-perirhinal infarct. In a previous report, Weddell [Weddell, R. A. (2005). A visual disorder producing highly selective deletion of recurring letters. Cortex, 41, 471-485] linked TR's highly selective tendency to delete recurrent letters with her fragmented percepts. The conflation of same-identity form elements was attributed to anterior extrastriate damage, which reduced the amount of information sustainable in fully resolved visual percepts, and the present experimental investigation of her subjective account of segment formation and resolution completes the story. She said that complex objects and long words first appeared as blurred regions, which sometimes included form elements. It is argued that figure-centred attentional mechanisms subdivided this blurred region into up to 3-4 parts. String length, lexical status (word vs. nonword), and background colour and/or luminance determined fragment length. Two rules described the fragment resolution sequence: largest segments usually resolved first, left-to-right resolution accounting for a few sequences. This resolution sequence occurred when stimuli were exposed too briefly for saccadic exploration, implicating endogenous attentional shifts. Experiment 4 confirmed TR's assertion that spatial, orthographic, and phonological information were stored during the fragment resolution process. Moreover, TR exerted considerable voluntarily control over the fragment resolution sequence and some influence over fragment length. Finally, these findings were interpreted in terms of an extended version of a neural network model of vision largely derived from nonhuman primate studies.     

A dyscalculic patient with selectively impaired processing of the numbers 7, 9, and 0.

A patient (J.C) had particular difficulties with the numbers 7, 9, and 0 in aurally and visually presented calculations. These problems were explained in terms of a modified version of the McCloskey, Caramazza, & Basili (1985) model of calculation processes. His calculation difficulties were attributed to degraded semantic representations of the numbers 7 and 9 and to lost knowledge of rules governing calculations involving the number 0. Use of "noncalculation" number processing tasks confirmed that his 7/9 problem was not at the level of acoustic analysis, the auditory input lexicon, the phonological output lexicon, visual analysis, or the orthographic output lexicon.     

Subcortical modulation of spatial attention including evidence that the Sprague effect extends to man

The Sprague effect is well-established-small tectal lesions restore visual orientation in the hemianopic field of animals with extensive unilateral geniculo-striate lesions. Studies of human midbrain visual functions are rare. This man with a midbrain tumour developed left-neglect through subsequent right frontal damage. Bilateral orientation returned after clear evidence of damage to the superior colliculus contralateral to the cortical lesion (showing the Sprague effect extends to man). Sustained right-neglect developed after probable additional damage to right superior colliculus. The regulation of spatial attention by tecto-pulvinar circuits is discussed, and it is argued that the reduced right tecto-pulvinar activity (consequent to the additional right collicular damage) was offset by over-compensatory increase in thalamic reticular nucleus (TRN) suppression of left pulvinar activity.