Regret and Responsibility Resolved? Evaluating Ordóñez and Connolly's (2000) Conclusions

T. Connolly, L. D. Ordo;aan;atez, and R. Coughlan (1997, Organizational Behavior and Human Decision Processes, 70, 73-85) argued, on the basis of 5 experiments, that regret need not be related to a sense of responsibility for the regretted outcome. We (M. Zeelenberg, W. W. van Dijk, & A. S. R. Manstead, 1998, Organizational Behavior and Human Decision Processes, 74, 254-272) showed in 2 experiments that this conclusion was premature, because it was based on an indirect measure of regret (i.e., overall happiness with the decision outcome). When regret was directly measured, the predicted effects of responsibility were found. L. D. Ordo;aan;atez and T. Connolly (2000, Organizational Behavior and Human Decision Processes, 81, 132-142) replicated our findings in 2 experiments. Based on their findings they arrived at 4 conclusions. In this rejoinder we first discuss Ordó?ez and Connolly's new studies and we then discuss the validity of their 4 conclusions. Copyright 2000 Academic Press.     

Differences in spatial-temporal parameters and arm-leg coordination in butterfly stroke as a function of race pace, skill and gender

Spatial-temporal parameters (velocity, stroke rate, stroke length) and arm-leg coordination in the butterfly stroke were studied as a function of race pace, skill (due to technical level, age, and experience) and gender. Forty swimmers (ten elite men, ten elite women, ten less-skilled men, and ten less-skilled women) performed the butterfly stroke at four velocities corresponding to the appropriate paces for the 400-m, 200-m, 100-m, and 50-m, respectively. Arm and leg stroke phases were identified by video analysis and used to calculate four time gaps (T1: the time difference between the start of the arms' catch phase and the start of the legs' downward phase of the first leg kick; T2: the time difference between the start of the arms' pull phase and the start of the legs' upward phase of the first leg kick; T3: the time difference between the start of the arms' push phase and the start of the legs' downward phase of the second leg kick; and T4: the time difference between the start of the arms' recovery and the start of the legs' upward phase of the second leg kick) and the total time gap (TTG), i.e., the sum of the four discrete time gaps. These values described the changing coupling of arm to leg actions over an entire stroke cycle. A significant race pace effect indicated that the synchronization between the key motor points of the arms and legs, which determine the starts and ends of the arm and leg stroke phases, increased with pace for all participants. A significant skill effect indicated that the elite swimmers had greater velocity, stroke length, and stroke rate and stronger synchronization of the arm and leg stroke phases than the less-skilled swimmers, due to smaller T2 and T3 and greater T1. A significant gender effect revealed greater velocity and stroke length for the men, and smaller T1 for the less-skilled women. These time gap differences between skill levels were related to the capacity of elite swimmers to assume a more streamlined position of trunk, head and upper limbs during leg actions, adopt a shorter glide and higher stroke rate to overcome great forward resistance, and generate higher forces and use better technique during the arm pull. Thus, coaches are advised to begin monitoring arm-leg coordination earlier in swimmers' careers to ensure that they attain their highest possible skill levels.     

Is time limit at the minimum swimming velocity of VO2 max influenced by stroking parameters?

The aim of this study was to observe the relationship between time limit at the minimum velocity that elicits maximal oxygen consumption (TLim-v VO2 max) and stroke rate, stroke length, and stroke index. 13 men and 10 women, highly trained swimmers, performed an intermittent incremental test for v VO2 max assessment and an all-out swim to estimate TLim-v VO2 max. The mean +/- SD TLim-v VO2 max, v VO2 max, stroke rate, stroke length, and stroke index values were 233.36 +/- 53.92 sec., 1.40 +/- .06 meter/sec., 35.58 +/- 2.89 cycles/min., 2.39 +/- .22 meter/cycle, and 3.36 +/- .41 meter2/(cycle x sec.), respectively. The correlation between TLim-v VO2 max and stroke rate was -.51 (p < .01), and values for TLim-v VO2 max with stroke length (r = .52, p < .01) and stroke index (r = .45, p < .05). These results seem to suggest that technical skill is a key factor in typical efforts requiring prolonged aerobic power.     

Modifications in basal and stress-induced hypothalamic AMP-activated protein kinase (AMPK) activity in rats chronically treated with an angiotensin II receptor blocker

5' adenosine monophosphate-activated protein kinase (AMPK) plays a prominent role as a metabolic stress sensor. The role of hypothalamic AMPK in response to restraint and surgical stress has not been previously investigated. It has been recently suggested that the renin-angiotensin system, in addition to its role in stress regulation, may play a significant role in regulating metabolic pathways including the regulation of the AMPK system. This study was thus aimed to evaluate the effects of candesartan, an angiotensin II AT1 receptor blocker drug, on hypothalamic AMPK activity under basal conditions and after restraint in conscious rats or after surgical stress under general anesthesia. Male Wistar rats were treated with 5 mg/kg/day candesartan in the drinking water for 2 weeks. The hypothalamic AMPK activity was determined under basal and stress conditions, using a kinase activity assay. Chronic administration of candesartan significantly increased hypothalamic AMPK activity. Hypothalamic AMPK activity was also increased by restraint stress whereas no change was observed during surgical stress under anesthesia. The high levels of hypothalamic AMPK activation observed in candesartan-treated rats were not changed by restraint stress but were reduced to control levels by anesthesia and surgery. In conclusion, chronic candesartan treatment and restraint stress in conscious rats stimulate the hypothalamic AMPK activity, whereas surgical stress under anesthesia inhibits pathways regulating the AMPK activity even in candesartan-treated rats.