Pathways linking temperament and depressive symptoms: a short-term prospective diary study among adolescents

The current study examined mechanisms through which trait negative affectivity (NA) and effortful control (EC) prospectively predict increases in depressive symptoms in a community sample of 110 adolescents (mean age=16.40 years) in a 9-week, multi-wave diary study. We hypothesised youth with high NA and low EC would experience greater increases in depressive symptoms because they generate more maladaptive cognitive responses to stress. NA predicted greater event-specific rumination and negative cognitive style across the study period. After controlling for trait cognitive vulnerabilities, only event-specific rumination mediated the effect of NA on subsequent depressive symptoms, suggesting that high-NA youth are more likely to ruminate about stressful events, which then predicts increases in depressive symptoms. EC did not moderate the effects of NA on either depressive symptoms or cognitive responses to stress. Results are discussed in terms of vulnerability-stress models linking temperament and depressive symptoms in adolescence.     

Cognitive mechanisms linking low trait positive affect to depressive symptoms: A prospective diary study

Low trait positive affect represents an affective vulnerability to depression, but little research has examined mechanisms linking low trait positive affect to depressive symptoms. The current study investigated whether the cognitive strategies of dampening and positive rumination mediated the prospective association between low trait positive affect and depressive symptoms. Participants were 209 undergraduate students who participated in an eight-week online study. Depressive symptoms and trait temperament were assessed at baseline, followed by seven weekly questionnaires which assessed cognitive strategies in response to weekly events and weekly depressive symptoms. Data were analysed using multilevel modelling following the mediation approach proposed by Nezlek. Results indicated that low trait positive affect significantly predicted less positive rumination but not greater dampening in response to weekly positive events. Less positive rumination in response to weekly positive events partially mediated the association between low trait positive affect and greater depressive symptoms across the study.     

Pathways linking temperament and depressive symptoms: A short-term prospective diary study among adolescents

The current study examined mechanisms through which trait negative affectivity (NA) and effortful control (EC) prospectively predict increases in depressive symptoms in a community sample of 110 adolescents (mean age=16.40 years) in a 9-week, multi-wave diary study. We hypothesised youth with high NA and low EC would experience greater increases in depressive symptoms because they generate more maladaptive cognitive responses to stress. NA predicted greater event-specific rumination and negative cognitive style across the study period. After controlling for trait cognitive vulnerabilities, only event-specific rumination mediated the effect of NA on subsequent depressive symptoms, suggesting that high-NA youth are more likely to ruminate about stressful events, which then predicts increases in depressive symptoms. EC did not moderate the effects of NA on either depressive symptoms or cognitive responses to stress. Results are discussed in terms of vulnerability-stress models linking temperament and depressive symptoms in adolescence.     

The association between temperament and depressive symptoms in adolescence: brooding and reflection as potential mediators

This study examined whether rumination subtypes (brooding and reflection) mediated prospective associations between temperament (negative emotionality and positive emotionality) and depressive symptoms in a community sample of 423 adolescents. Effortful control and sex were examined as potential moderators of the mediated pathway. Youth self-reported negative emotionality (NE), positive emotionality (PE), and effortful control (EC) at age 12; brooding and reflection subtypes of rumination at age 14; and depressive symptoms at ages 12, 14, and 15. Hierarchical linear regression analyses indicated that, controlling for initial levels of depressive symptoms, high NE, but not low PE, predicted increases in depressive symptoms from age 12 to age 15. Brooding, but not reflection, mediated the association between NE and depressive symptoms. Neither sex nor EC moderated either indirect pathway in the mediated model. The results confirm and extend previous findings on the association between affective and cognitive vulnerability factors in predicting depressive symptoms in adolescence.     

Modeling the Gender Difference in Depression: A Commentary on Cambron, Acitelli, and Pettit

In reviewing Cambron, Acitelli, and Pettit’s interpersonal contingent self-esteem model of the gender difference in depression, we applaud their domain-match vulnerability-stress approach as well as their specification of the processes mediating self-esteem and depression. However, we argue that models of the gender difference in depression must address five key points in order to provide compelling and meaningful explanations. Such models should: consider that vulnerability, stress, or vulnerability-stress interactions may vary across domains; articulate the processes by which stress-reactive negative affect develops into depressive symptoms; take a developmental approach; be sensitive to social and political implications; and provide clear hypotheses regarding the mediating and moderating mechanisms implicated in the model. These suggestions apply to all researchers interested in explaining the gender difference in depression.     

Affective,Biological, and Cognitive Predictors of Depressive Symptom Trajectories in Adolescence

Heterogeneity in the longitudinal course of depressive symptoms was examined using latent growth mixture modeling among a community sample of 382 U.S. youth from ages 11 to 18 (52.1 % female). Three latent trajectory classes were identified: Stable Low (51 %; displayed low depressive symptoms at all assessments), Increasing (37 %; reported low depressive symptoms at age 11, but then significantly higher depressive symptoms than the Stable Low class at ages 13, 15, and 18), and Early High (12 %; reported high early depressive symptoms at age 11, followed by symptoms that declined over time yet remained significantly higher than those of the Stable Low class at ages 13, 15, and 18). By age 15, rates of Major Depressive Disorder diagnoses among the Early High (25.0 %) and Increasing (20.4 %) classes were more than twice that observed among the Stable Low class (8.8 %). Affective (negative affectivity), biological (pubertal timing, sex) and cognitive (cognitive style, rumination) factors were examined as predictors of class membership. Results indicated general risk factors for both high-risk trajectories as well as specific risk factors unique to each trajectory. Being female and high infant negative affectivity predicted membership in the Increasing class. Early puberty, high infant negative affectivity for boys, and high rumination for girls predicted membership in the Early High class. Results highlight the importance of examining heterogeneity in depression trajectories in adolescence as well as simultaneously considering risk factors across multiple domains.     

The developmental origins of cognitive vulnerability to depression: temperament, parenting, and negative life events in childhood as contributors to negative cognitive style

Cognitive models of depression have been well supported with adults, but the developmental origins of cognitive vulnerability are not well understood. The authors hypothesized that temperament, parenting, and negative life events in childhood would contribute to the development of cognitive style, with withdrawal negativity and negative parental feedback moderating the effects of negative life events to predict more depressogenic cognitive styles. These constructs were assessed in 289 children and their parents followed longitudinally from infancy to 5th grade; a subsample (n = 120) also participated in a behavioral task in which maternal feedback to child failure was observed. Results indicated that greater withdrawal negativity in interaction with negative life events was associated with more negative cognitive styles. Self-reported maternal anger expression and observed negative maternal feedback to child's failure significantly interacted with child's negative events to predict greater cognitive vulnerability. There was little evidence of paternal parenting predicting child negative cognitive style.     

The association between temperament and depressive symptoms in adolescence: Brooding and reflection as potential mediators

This study examined whether rumination subtypes (brooding and reflection) mediated prospective associations between temperament (negative emotionality and positive emotionality) and depressive symptoms in a community sample of 423 adolescents. Effortful control and sex were examined as potential moderators of the mediated pathway. Youth self-reported negative emotionality (NE), positive emotionality (PE), and effortful control (EC) at age 12; brooding and reflection subtypes of rumination at age 14; and depressive symptoms at ages 12, 14, and 15. Hierarchical linear regression analyses indicated that, controlling for initial levels of depressive symptoms, high NE, but not low PE, predicted increases in depressive symptoms from age 12 to age 15. Brooding, but not reflection, mediated the association between NE and depressive symptoms. Neither sex nor EC moderated either indirect pathway in the mediated model. The results confirm and extend previous findings on the association between affective and cognitive vulnerability factors in predicting depressive symptoms in adolescence.     

The ABCs of depression: integrating affective, biological, and cognitive models to explain the emergence of the gender difference in depression

In adulthood, twice as many women as men are depressed, a pattern that holds in most nations. In childhood, girls are no more depressed than boys, but more girls than boys are depressed by ages 13 to 15. Although many influences on this emergent gender difference in depression have been proposed, a truly integrated, developmental model is lacking. The authors propose a model that integrates affective (emotional reactivity), biological (genetic vulnerability, pubertal hormones, pubertal timing and development) and cognitive (cognitive style, objectified body consciousness, rumination) factors as vulnerabilities to depression that, in interaction with negative life events, heighten girls' rates of depression beginning in adolescence and account for the gender difference in depression.