A Rorschach investigation of mothers of behaviorally disturbed infants

This investigation focuses on one facet of the interactional family, parent-child, mother-infant matrix of behaviorally disturbed infants: the personality of mother as primary caregiver. Rorschach test behavior of mothers (N = 30) of behaviorally disturbed infants were compared with two normative samples. The Rorschach test was found to indicate certain atypical psychological patterns among these mothers. Differences emerged in intellectual sphere variables: number of responses (R), common detail responses (D), rare detail responses (Dd), number of accurate form percepts (F+), percentage of form percepts that are accurate (F+%), percentage of responses with animal content (A%), and popular percepts (P). Other response trends emerged in the affective sphere: percentage of pure form responses (F%) and in the interpersonal sphere: whole human content (H) and the ratio of the whole human content to human detail content (H:Hd), as well as to the test as a whole. Based on these results, hypotheses are generated for further investigation, particularly of traits in the areas of cognitive processing and the capacity for interpersonal relations.     

The problem of R in the Rorschach: the value of varying responses

A subject's freedom to give different numbers of responses to each inkblot has always been part of the Rorschach tradition. However, frequency of response (R) as a variable rather than a constant requires special attention when using the Rorschach psychometrically and when making clinical evaluations based in part or entirely on normative scores. In this article, I discuss the Rorschach tradition; the distinctiveness of the procedure; and the complexity of the technical, methodological, and theoretical problems. issues related to the integration of the empirical and conceptual approaches are discussed.     

McEwen-Induced Modulation of Endocrine History: A Partial Review

In endless facets of physiology, there are points of homeostatic balance, such that too much or too litttle of something can both be deleterious (i.e., an "inverse U" pattern). This is particularly true when considering glucocorticoids (GCs), the adrenals steroid secreted during stress. In the first part of this paper, I review a number of realms in which a paucity and an excess of GCs are both damaging. Some findings are classical (for example, concerning GC effects upon body weight), while some are quite recent and have considerable implications for both physiology and pathophysiology (for example, inverse U's of GC actions in the realm of immunity and neuronal survival). The second part of the review considers the far thornier issue of how such inverse U's of GC actions are generated on a cellular and molecular level. One solution that has evolved, primarily in the hippocampus within the nervous system, involves the presence of two different types of receptors for GCs within the same cells; so long as the two receptors have very different affinities and mediate opposing effects on some cellular endpoint, an inverse U will emerge. The second solution, found in a number of peripheral tissues, involves GCs having opposing effects on the amount of some signal being generated (e.g., an immune cytokine) and the sensitivity of target tissues to that signal; under conditions that appear to be physiologically relevant, inverse U's emerge from this pattern as well. The final section of this review considers the enormous role played by Bruce McEwen in the emergence of this literature. I suggest that while much of this obviously has to do with the facts that have come from his group, another substantial contribution is from his steadying and supportive personality, the veritable embodiment of homeostatic balance.     

Treatment of Time in the TAT

Part of a larger empirical investigation to identify and classify subjective probability notions and explore their relationship to personality variables (Lipgar, 1965). Four factors that distinguished four types of subjective probability notions had been obtained from a matrix of intercorrelations among 30 Ss' Q sort ratings on patterns of “head-tail” guesses. To investigate the possibility that subjective probability notions are associated with other orientational activities in situations of uncertainty, a typology of time orientations and rating scales was derived from TAT stories by those Ss who best represented the four subjective probability factors. These and other stories were rated by four judges independently, and an association between treatment of time in the TAT and subjective probability notions was substantiated.     

Developmental and sex-related differences in preschoolers' affective decision making

This study investigated developmental and sex-related differences in affective decision making, using a two-deck version of Children's Gambling Task administered to 3- and 4-year-old children. The main findings were that 4-year-old children displayed better decision-making performance than 3-year-olds. This effect was independent of developmental changes in inductive reasoning, language, and working memory. There were also sex differences in decision-making performance, which were apparent only in 3-year-old children and favored girls. Moreover, age predicted awareness of task and the correlation between the latter and decision-making performance was significant, but only in 4-year-old children. This study thus indicates that there is a remarkable developmental leap in affective decision making, whose effects are apparent around the age of 4, which according to our results, also marks the age when the correlation of declarative knowledge and decision-making performance becomes significant.     

Edelson's contributions to group psychology: a contemporary perspective

If you are already familiar with Marshall Edelson's writings, you may be expecting this article to be difficult and challenging or outmoded. Since his work with groups began in the 1950s, so much has changed in how we treat the mentally ill, how we train mental health professionals, how hospitals are managed, and how treatments for patients are designed and delivered. This article was written in the belief that nothing is of more lasting relevance than a story of courage and commitment in the search for knowledge to relieve human suffering. Edelson's career is such a story. Intent upon advancing our understanding of groups and organizations, Edelson embodied a distinctive concern with values and boundaries. This article was begun when his physical health was in rapid decline and completed while he was still able to review it. He died January 17, 2005.     

Stress, Glucocorticoids, and Damage to the Nervous System: The Current State of Confusion

An extensive literature demonstrates that glucocorticoids (GCs), the adrenal steroids secreted during stress, can have a broad range of deleterious effects in the brain. The actions occur predominately, but not exclusively, in the hippocampus, a structure rich in corticosteroid receptors and particularly sensitive to GCs. The first half of this review considers three types of GC effects: a) GC-induced atrophy, in which a few weeks' exposure to high GC concentrations or to stress causes reversible atrophy of dendritic processes in the hippocampus; b) GC neurotoxicity where, over the course of months, GC exposure kills hippocampal neurons; c) GC neuroendangerment, in which elevated GC concentrations at the time of a neurological insult such as a stroke or seizure impairs the ability of neurons to survive the insult. The second half considers the rather confusing literature as to the possible mechanisms underlying these deleterious GC actions. Five broad themes are discerned: a) that GCs induce a metabolic vulnerability in neurons due to inhibition of glucose uptake; b) that GCs exacerbate various steps in a damaging cascade of glutamate excess, calcium mobilization and oxygen radical generation. In a review a number of years ago, I concluded that these two components accounted for the deleterious GC effects. Specifically, the energetic vulnerability induced by GCs left neurons metabolically compromised, and less able to carry out the costly task of containing glutamate, calcium and oxygen radicals. More recent work has shown this conclusion to be simplistic, and GC actions are shown to probably involve at least three additional components: c) that GCs impair a variety of neuronal defenses against neurologic insults; d) that GCs disrupt the mobilization of neurotrophins; e) that GCs have a variety of electrophysiological effects which can damage neurons. The relevance of each of those mechanisms to GC-induced atrophy, neurotoxicity and neuroendangerment is considered, as are the likely interactions among them.