Vocal changes in stutterers and nonstutterers when speaking to children

Within the last few years, considerable attention has been devoted to the possibility that the amelioration of stuttering in many novel speaking conditions is a function of vocal changes with or without accompanying modifications in prosodic expression. These formulations have made reference primarily to conditions in which stutterers were induced into one or another novel speech pattern by an external stimulus such as masking noise, a rhythmic cue, or by instructions to sing, or read in unison with another speaker. In contrast, far less attention has been paid to other ameliorative conditions that lack a novel external stimulus or special instructions that direct the stutterer to speak in some unique manner. Speaking or reading to a child is an example of this latter type of condition. The present study was conducted to see if nine adult stutterers and a matched group of nonstutterers would evince vocal changes as they read aloud to another adult, a child, and a child and adult together. There was also a fourth control condition wherein the subjects read to one of the experimenters. Dependent measures of disfluency and stuttering, fundamental frequency, fundamental frequency deviation, fluent reading rate, and peak vocal sound pressure level were made and treated statistically. The major findings of this study indicated that, relative to the control condition, both subject groups exhibited several vocal changes when reading to a child alone and to a child and adult together. In addition, the stutterers experienced a reduction in disfluency, but only when reading to the child alone. The normal speakers exhibited too few disfluencies in the control condition for any meaningful change to occur in that dependent variable. The fact that the stutterers evinced vocal changes in both experimental conditions but a significant drop in disfluency in just one bears importantly on hypotheses that emphasize vocal changes in conditions that ameliorate stuttering.     

A study of language functioning in Alzheimer patients

Language functioning in Alzheimer's disease is reviewed and the performance of 25 Alzheimer patients on a standard battery is reported. All these hospitalized patients were aphasic to some degree. As a group, they differed from normals on all language variables, and from stroke patients in terms of higher fluency and lower comprehension. Spontaneous speech showed high incidence of circumlocutions and semantic jargon, but no phonemic paraphasias or target approximations. Syllabic perseverations, shouting, inappropriate laughter, and mutism were late-appearing features. Transcortical Sensory, and Wernicke's aphasias were frequent, but Broca's and Transcortical Motor aphasias notably absent. Extent of language impairment correlated with current length of hospitalization but not age. Reading, writing, and performance scores except praxis, were lower than oral language scores. Findings were discussed in relation to previous results, methodology, and language organization in the brain.     

Deep agraphia

A case of agraphia is documented which resembles in every respect the pattern of results obtained in deep dyslexia. Pronounceable pseudowords could not be written to dictation while concrete nouns were more accurately transcribed than abstract nouns. Verbs and functions words were written very poorly and oral spelling was impaired. In addition, semantic paragraphias were present. In contrast to the deficit observed in writing, no such impairment was found for reading. It is concluded that writing can occur without phoneme-grapheme conversion and that codes generated for reading are functionally distinct from codes generated for writing.     

Aphasics' selective deficits in appreciating grammatical agreements

This study attempted to dissociate in aphasic patients different aspects of grammatical capacity. Subjects were asked to evaluate sentences containing violations of agreements between pronouns and verbs. Agreements which are considered primarily structural in nature were violated (e.g., a surface object case like “them” placed in a surface subject sentential slot) while other violated agreements seemed to involve both structural and semantic information (e.g., lack of agreement between a pronoun and a verb in terms of the number of people performing the act). These violations were couched in one of three types of sentential frames which varied in terms of syntactic complexity (e.g., active declarative vs. passive syntactic voices). The results revealed that Broca's aphasics found agreement violations difficult to detect in complex syntactic frames. They were quite successful at detecting even violations of the largely structural “case” type of agreement, however, when couched in simpler syntactic frames. Fluent aphasics encountered more difficulty detecting violations of agreements involving both semantic and structural information than agreements which were primarily structural in nature, that is, regardless of the syntactic frame. These unique performance profiles suggest that Broca's aphasics may be agrammatic only with respect to certain aspects of a sentence's structure, and that fluent aphasics may also experience some selective—but different—grammatical deficits.     

Toward an integrated approach to early childhood stuttering

This article proposes a model that integrates various factors influencing early childhood stuttering. The factors interact in a synergistic manner and can be subsumed under three macrofactors: physiological, psycholinguistic, and psychosocial. Such a model can be clinically applied toward an integrated approach to the assessment and treatment of the young stutterer.     

The problem of relapse in stuttering: Some thoughts on what might cause it and how to deal with it

Bloodstein recently wrote that “the most vital part of science is not accurate measurement or the insistence on empiricism. It is an attitude of inquiry into the how and why of things” [Bloodstein, O. Foreword. In: Maintenance of Fluency (E. Boberg ed.). New York: Elsevier, 1981, p. vii]. In the paper below, I present some unsubstantiated and no doubt controversial thoughts on the how and why of relapse in stuttering. The overall thrust of these thoughts is that stutterers have different propensities to stutter because of the inherent variability in their speech production systems. For this reason, for therapy to have long-range success, stutterers must learn to accept and deal with the inherent variability in their speech production mechanisms.