不熟悉材料伴随熟悉类别标签时儿童的归纳推理

对呈现在儿童面前的不熟悉的材料用儿童熟悉的、各不相同的类别标签命名之后,使儿童面临知觉相似和类别冲突的情形下,研究了儿童的归纳推理及其发展。研究结果显示,在此情形下,5岁儿童的归纳推理主要是基于知觉相似的,7岁儿童基于知觉相似和基于类别的归纳推理之间差异不显著,11岁儿童则主要基于类别进行归纳推理。     

双系统逃生决策的顺序替代效应

探讨当信息以不同方式呈现时双系统逃生决策的顺序替代效应。采用单因素完全随机设计,将180名被试平均分配到直觉、分析和直觉后分析三种条件下考察其逃生决策。结果表明：(1)信息直接呈现时,直觉和分析决策、以及直觉和直觉后分析决策均差异显著且趋势相同;(2)信息隐含呈现时,直觉和分析决策差异显著,但直觉和直觉后分析决策差异不显著。结论：当信息直接呈现时个体的逃生决策存在顺序替代;而当信息隐含呈现时存在反顺序替代。     

重复刺激的末位优势效应

探讨在RSVP任务中重复刺激的呈现位置和报告方式对重复知盲效应的影响, 检验该研究提出的注意资源最优化假设和末位优势效应。实验1操作了刺激性质和重复刺激位置, 得出了刺激性质和重复刺激位置的交互效应; 实验2操作了刺激性质和情景信息, 结果是交互效应不显著; 实验3操作了刺激性质和报告方式, 出现了刺激性质和报告方式的交互效应。该结果表明: (1)由于注意资源分配的优化, 重复刺激有末位优势效应。(2)重复知盲的产生不是发生在知觉阶段, 而是发生在报告阶段。(3)注意资源分配最优化理论比建构/归因理论能更合理地解释重复知盲的发生。     

词边界信息在中文阅读眼跳目标选择中的作用：来自中文二语学习者的证据

采用EyeLink 1000眼动仪, 选取韩、美、日、泰四国留学生各20名, 在正常无空格和词间空格两种呈现方式下阅读中文语句, 以此来探讨二语学习者在阅读中文时词边界信息在眼跳目标选择中的作用。结果发现, 二语学习者在中文阅读中存在着一致的眼动模式, 即在单次注视事件中, 倾向于注视词的中间部分, 在多次注视事件中, 倾向于注视其开端部分, 然后再计划一次词内再注视。词边界信息能够有效地引导中文二语学习者的眼动行为和眼跳计划, 更多地将首次注视位置落在词的中间部分。最后, 词边界信息在眼跳目标选择中的促进作用没有受到母语文本呈现方式熟悉性的调节。我们认为, 中文二语学习者在阅读中的眼跳目标选择可能采用的是战略-战术策略。     

相似和相同语言标签对幼儿归纳推理的影响

为进一步检验相似语言标签的效应,采用经典的三角属性归纳范式,通过两个实验研究了"相似/相同"的真实语言标签对4岁幼儿归纳推理的影响.在实验一中,三角范式中的三个刺激物属于同一类别,在实验二中,三个刺激物属于两种不同的类别.每个实验均有三种条件:在控制条件中,实验者用"这个"指代每个客体;在相似语言标签条件中,靶刺激与知觉不相似的比较刺激具有相同的、且暗示了类别成员关系的词素(例如,翠鸟—飞机—鸵鸟);在相同语言标签条件中,靶刺激和知觉不相似的比较刺激共享相同的语言标签(例如,鸟—飞机—鸟).结果一致显示,与控制条件相比,幼儿在相似语言标签条件下表现出了更多的基于概念的归纳,表明在真实语言标签条件下也存在相似语言标签效应.同时,研究结果也显示,幼儿在相似和相同语言标签条件下的归纳没有显著差异,说明语言标签在幼儿归纳中更可能传递了概念信息,从而挑战了Sloutsky等的理论.     

子团队利弊作用的调节机制

子团队是指在断裂带的作用之下, 团队成员在认同以及信息加工方式等方面的差异而自发形成的小群体, 因子团队广泛存在于团队之中, 因此对其利弊作用的研究受到了越来越多的关注。从子团队利弊作用出发, 即以子团队给团队运转过程带来的积极与消极作用为基础, 来研究调节子团队利弊作用的因素并提出其调节机制。最后提出未来的研究应从动态视角的结合、团队领导的介入、子团队过程的考虑、子团队距离以及社会关系等角度做进一步的探索。     

Ronald Reagan's Presidency: The Impact of an Alcoholic Parent

Ronald Reagan enjoyed a successful political career. Nevertheless, his political life was affected dramatically by the fact that he was the son of an alcoholic parent. Alcoholic parents leave deep marks on their children's lives, even after those children become adults. As president of the United States, Reagan clearly demonstrated these marks. He was aloof and distant, was often a disengaged leader, showed inordinate loyalty to associates even when such loyalty became problematic, was prone to live in a world of make‐believe, married compulsive women, and craved approval and applause. Each of these behavioral characteristics was part of the psychological legacy left to this president by his long‐dead alcoholic father. Some of them damaged his presidency greatly; others, however, may well have assisted it.     

Neuroticism and extraversion share genetic and environmental effects with negative and positive mood spillover in a nationally representative sample

Work-family spillover research focuses on how negative and positive moods in one life domain carry over to another. Domain-specific etiologies (e.g., family conflict) are often emphasized to explain spillover. Yet, strong correlations exist between spillover variables of the same emotional valence but originating from different domains, suggesting individual differences in the tendencies to prolong mood-states. The current study (N = 1143 individuals) examined whether these general tendencies are associated with neuroticism and extraversion and how genetic and environmental effects contribute to these associations. Findings revealed that neuroticism and extraversion are related to these tendencies through both genetic and environmental pathways.     

Pharmacological treatment effects on eye movement control

The increasing use of eye movement paradigms to assess the functional integrity of brain systems involved in sensorimotor and cognitive processing in clinical disorders requires greater attention to effects of pharmacological treatments on these systems. This is needed to better differentiate disease and medication effects in clinical samples, to learn about neurochemical systems relevant for identified disturbances, and to facilitate identification of oculomotor biomarkers of pharmacological effects. In this review, studies of pharmacologic treatment effects on eye movements in healthy individuals are summarized and the sensitivity of eye movements to a variety of pharmacological manipulations is established. Primary findings from these studies of healthy individuals involving mainly acute effects indicate that: (i) the most consistent finding across several classes of drugs, including benzodiazepines, first- and second- generation antipsychotics, anticholinergic agents, and anticonvulsant/mood stabilizing medications is a decrease in saccade and smooth pursuit velocity (or increase in saccades during pursuit); (ii) these oculomotor effects largely reflect the general sedating effects of these medications on central nervous system functioning and are often dose-dependent; (iii) in many cases changes in oculomotor functioning are more sensitive indicators of pharmacological effects than other measures; and (iv) other agents, including the antidepressant class of serotonergic reuptake inhibitors, direct serotonergic agonists, and stimulants including amphetamine and nicotine, do not appear to adversely impact oculomotor functions in healthy individuals and may well enhance aspects of saccade and pursuit performance. Pharmacological treatment effects on eye movements across several clinical disorders including schizophrenia, affective disorders, attention deficit hyperactivity disorder, Parkinson’s disease, and Huntington’s disease are also reviewed. While greater recognition and investigation into pharmacological treatment effects in these disorders is needed, both beneficial and adverse drug effects are identified. This raises the important caveat for oculomotor studies of neuropsychiatric disorders that performance differences from healthy individuals cannot be attributed to illness effects alone. In final sections of this review, studies are presented that illustrate the utility of eye movements for use as potential biomarkers in pharmacodynamic and pharmacogenetic studies. While more systematic studies are needed, we conclude that eye movement measurements hold significant promise as tools to investigate treatment effects on cognitive and sensorimotor processes in clinical populations and that their use may be helpful in speeding the drug development pathway for drugs targeting specific neural systems and in individualizing pharmacological treatments.     

Biological Implications of Gene–Environment Interaction

Gene–environment interaction (G × E) has been treated as both a statistical phenomenon and a biological reality. It is argued that, although there are important statistical issues that need to be considered, the focus has to be on the biological implications of G × E. Four reports of G × E deriving from the Dunedin longitudinal study are used as exemplars of the biological considerations that should lead to an hypothesis-driven choice of the specific genetic polymorphisms and the specific environmental influence to be investigated. The same four studies are used to discuss how the assessment of internal and external validity can be undertaken and how experimental approaches in humans and with animal models may be informative in the elucidation of the relevant operative biological mechanisms.