Robust affective priming effects in a conditional pronunciation task: Evidence for the semantic representation of evaluative information

Based on the hypothesis that information about the valence of words is encoded in a semantic system, we predicted that the match between the valence of a prime and the valence of a target word will influence the pronunciation of the target only if and to the extent that pronunciation is semantically mediated. In line with this prediction, we found affective priming effects (faster pronunciation when prime and target had the same valence than when they had a different valence) only when participants were instructed to read words but not nonwords (Experiment 1) or words that were not names of occupations (Experiment 2). Priming was not significant when participants were asked to read white but not red words (Experiment 1) or words that did not have a frame around them (Experiment 2).     

Führt die evolutionäre Erkenntnistheorie in einen Relativismus?

This essay is a discussion of Eve-Marie Engels' view on Evolutionary Epistemology (EE). In the first part two of the main doctrines of EE are criticized: (1.) that validity of human knowledge is to be explained as the result of evolutionary adaptation; yet (2.), that human cognitive capacities had been adequate to our ancestors life conditions but fail in relevant situations of modern world. In the second part the concept of reality underlying EE's adaptational view is discussed and compared with Jean Piaget's concept of reality. Finally, it is held that some of the questions recently raised by proponents of EE, far from being new, have been dealt with already in historical epistemology.

---

Diese Arbeit entstand im Rahmen des Projekts Nr. 11-2224.87 des Schweizerischen Nationalfonds.     

Association between the arginine vasopressin receptor 1A (AVPR1A) gene and preschoolers’ executive functioning

Recent evidence implicates the arginine vasopressin (AVP) system in complex neuropsychological disorders which are characterized by deficits in executive functioning (EF). Despite the genetic contribution to EF, little is currently known about its molecular genetic basis. Drawing on research from social neuroscience and the role of related physiological systems in psychopathology, the current study hypothesized that variability in the AVP receptor 1a gene (AVPR1A) would be associated with EF in an epidemiological sample of 323 normally developing preschool-aged children. Using a family-based association design, the current study found that variability in the rs7298346 marker, located in the 5′-flanking region, was significantly related to a composite measure of EF in 4-year-old children after controlling for a variety of covariates and children’s theory of mind. The converse association between AVPR1A and theory of mind (after controlling for EF) was not significant, suggesting a level of specificity in this relationship. The results are discussed in terms of the difficulties faced by genetic association studies in teasing apart the behavioral phenotypes that characterize complex psychological diseases and the involvement of multiple physiological systems in human behavior.     

Why is personality tied to sleep quality? A biometric analysis of twins

Despite consistent links between personality traits and poor sleep, little is known about genetic and environmental influences that may produce them. This study examined how much genetic background and environmental experiences contributed to phenotypic linkages between personality and subjective sleep quality. Seven hundred and thirty-four twin pairs from the Minnesota Study of Twin Aging and Development rated their sleep quality and provided personality reports. Bi-variate analyses revealed that genetic factors accounted for the majority of observed associations between subjective sleep quality and traits, but also that non-shared environmental experience played a role that varied across traits. The findings strongly implicate genotype in tying subjective sleep quality to personality variation, alongside non-shared environmental influences, and suggest indicate influences unique to individual traits.