单一被试实验与元分析技术

单一被试实验是特殊儿童心理与教育研究中的一种常用方法,但其外部效度往往受到质疑。运用元分析技术,能对大量单一被试实验研究结果进行定量综述,既可综合评价各研究的总效应,也能探讨、揭示出各类研究特征与相应研究结果的关系,这无疑为单一被试实验信度与效度的检验提供了更多的信息。本文结合国外一个较新的研究实例,对元分析技术的基本原理及分析步骤作简要介绍,并对单一被试实验与元分析技术的结合应用问题提出几点看法与建议。     

The Moral Foreign-Language Effect

Many have argued that moral judgment is driven by one of two types of processes. Rationalists argue that reasoned processes are the source of moral judgments, whereas sentimentalists argue that emotional processes are. We provide evidence that both positions are mistaken; there are multiple mental processes involved in moral judgment, and it is possible to manipulate which process is engaged when considering moral dilemmas by presenting them in a non-native language. The Foreign-Language Effect (FLE) is the activation of systematic reasoning processes by thinking in a foreign language. We demonstrate that the FLE extends to moral judgment. This indicates that different types of processes can lead to the formation of differing moral judgments. One implication of the FLE is that it raises the possibility that moral judgments can be made more systematic, and that the type of processing used to form them might be relevant to normative and applied ethics.     

Addressing the “Replication Crisis”: Using Original Studies to Design Replication Studies with Appropriate Statistical Power

Psychology is undergoing a replication crisis. The discussion surrounding this crisis has centered on mistrust of previous findings. Researchers planning replication studies often use the original study sample effect size as the basis for sample size planning. However, this strategy ignores uncertainty and publication bias in estimated effect sizes, resulting in overly optimistic calculations. A psychologist who intends to obtain power of .80 in the replication study, and performs calculations accordingly, may have an actual power lower than .80. We performed simulations to reveal the magnitude of the difference between actual and intended power based on common sample size planning strategies and assessed the performance of methods that aim to correct for effect size uncertainty and/or bias. Our results imply that even if original studies reflect actual phenomena and were conducted in the absence of questionable research practices, popular approaches to designing replication studies may result in a low success rate, especially if the original study is underpowered. Methods correcting for bias and/or uncertainty generally had higher actual power, but were not a panacea for an underpowered original study. Thus, it becomes imperative that 1) original studies are adequately powered and 2) replication studies are designed with methods that are more likely to yield the intended level of power.     

Not all effect sizes are the same: Comments on Holden (2008)

Despite the common belief that response bias is a significant moderator of psychological tests in field settings, these biases have been notoriously difficult to identify. Holden (2008) has recently presented evidence suggesting this paradox may at least in part be explained by problems inherent to the use of moderated regression with self-report indicators of response bias. His article offers an innovative proposal for understanding a central issue in applied test use. However, the conclusions drawn about both moderated regression and the general validity of response bias indicators are open to alternative explanations. It would be premature to assume these factors are important contributors to the ephemeral character of response bias effects.     

Does performance on the standard antisaccade task meet the co-familiality criterion for an endophenotype?

The “co-familiality” criterion for an endophenotype has two requirements: (1) clinically unaffected relatives as a group should show both a shift in mean performance and an increase in variance compared with controls; (2) performance scores should be heritable. Performance on the antisaccade task is one of several candidate endophenotypes for schizophrenia. In this paper we examine whether the various measures of performance on the standard version of the antisaccade task meet the co-familiality criterion for an endophenotype. The three measures of performance—reflexive saccade errors, latency of correct antisaccades, and gain—show a wide range of effect sizes and variance ratios as well as evidence of significant or near significant heterogeneity. The estimated mean effect sizes [Cohen’s d: error rate: 0.34 (SD: 0.29); latency: 0.33 (SD: 0.30); gain: 0.54 (SD: 0.38)] are significantly greater than 0, but the magnitude of the departures from 0 is relatively small, corresponding to modest effect sizes. The width of the 95% confidence intervals for the estimated effect sizes (error rate: 0.2–0.49; latency: 0.17–0.50; gain: 0.23–0.85) and the coefficients of variation in effect sizes (error rate: 85.3%; latency: 90.9%; gain: 68.4%) reflect heterogeneity in effect sizes. The effect sizes for error rate showed statistically significant heterogeneity and those for latency (P = .07) and gain (P = .09) showed a trend toward heterogeneity. These results indicate that the effect sizes are not consistent with a single mean and that the average effect size may be a biased estimate of the magnitude of differences in performance between relatives of schizophrenics and controls. Relatives of schizophrenics show a small but significant increase in variance in error rate, but the confidence interval is broad, perhaps reflecting the heterogeneity in effect size. The variance ratios for latency and gain did not differ in relatives of schizophrenics and controls. Performance, as measured by error rate, is moderately heritable. The data do not provide compelling support for a consistent shift in mean or variance in relatives of schizophrenia patients compared with nonpsychiatric controls, both of which are required for a major gene involved in co-familial transmission. This set of findings suggests that although intra-familial resemblance in antisaccade performance is due in part to genetic factors, it may not be related to a schizophrenia genotype. Based on the current literature, it would be premature to conclude that any of the measures of antisaccade performance unambiguously meets the co-familiality criterion for an endophenotype.     

Accuracy of judging others’ traits and states: Comparing mean levels across tests

Tests of accuracy in interpersonal perception take many forms. Often, such tests use designs and scoring methods that produce overall accuracy levels that cannot be directly compared across tests. Therefore, progress in understanding accuracy levels has been hampered. The present article employed several techniques for achieving score equivalency. Mean accuracy was converted to a common metric, pi [Rosenthal, R., & Rubin, D. B. (1989). Effect size estimation for one-sample multiple-choice-type data: Design, analysis, and meta-analysis. Psychological Bulletin, 106, 332–337] in a database of 109 published results representing tests that varied in terms of scoring method (proportion accuracy versus correlation), content (e.g., personality versus affect), number of response options, item preselection, cue channel (e.g., face versus voice), stimulus duration, and dynamism. Overall, accuracy was midway between guessing level and a perfect score, with accuracy being higher for tests based on preselected than unselected stimuli. When item preselection was held constant, accuracy was equivalent for judging affect and judging personality. However, comparisons must be made with caution due to methodological variations between studies and gaps in the literature.     

自我概念在父母情感关爱与儿童发展间的完全中介效应

运用问卷法,对北京612名中小学生及其父母分别进行了儿童孤独退缩行为、自我概念及父母情感关爱行为测试,运用LISEREL软件对数据的分析结果表明:父母情感关爱行为通过儿童自我概念的完全中介作用,对儿童的孤独退缩行为起到明显的抑制作用;在父母情感关爱行为和儿童学业成就之间,儿童的学业自我概念也起到了明显的完全中介效果.从而支持了儿童自我概念在父母情感关爱与儿童发展间的完全中介地位.     

单侧化准备电位的含义和应用

单侧化准备电位（Lateralized readiness potential, LRP）是指在随意运动中,反应效应器方位所对应的对侧大脑皮层出现的准备电位。LRP分析的关键是确定启动时间点。LRP可以揭示刺激-反应不同阶段的认知加工时间特性。可以从反应准备的抽象性或具体性以及大脑控制运动输出的“中枢”机制或“外周”机制这两个思路去验证LRP。LRP在阈下知觉和内隐学习、运动准备和反应执行、西蒙效应等方面的认知心理学研究以及新兴的脑-机界面工程心理学研究得到了广泛的应用,但LRP的应用存在着某些限制因素,如：脑电干扰、测量的低信噪比和其本身对反应效应器的要求等     

Functions and Health at the Interface of Biology and Technology

Synthetic biology promises to eliminate the distinction between biology and engineering by delivering a philosophically interesting new kind of entity: a biological organism that is wholly designed and constructed by humans. The possibility of such organisms raises interesting questions in three domains: the analysis of (1) biological functions, (2) engineering functions, and (3) health and disease. This paper identifies and systematically answers these questions. This does not only establish how we should think about functions and health and disease in synthetic biological organisms, but it also reveals insights that are of broader relevance: (1) aetiological accounts of biological function need to omit or reinterpret reference to natural selection. This results in complete continuity between aetiological analyses of function in engineering and philosophy; (2) considering synthetic biology prompts interesting further questions about heritability, ancestry, and biological individuals; and (3) accounts of disease as biological dysfunction do not straightforwardly map onto our intuitive health and disease judgments regarding non-human animals. In response to the latter point I examine three possible avenues, and tentatively defend one on behalf of the ‘disease as dysfunction’ theorist.