Analysing cycling as a social practice: An empirical grounding for behaviour change

Despite significant national and local efforts over the last decade to stimulate uptake of cycling in the UK, levels of cycling (particularly utility cycling) remain at around 2% of journeys. Understanding of cycling behaviour and subsequent development of interventions has typically been undertaken using an individualist approach, often relying on psychologically based models of behaviour. This paper argues that Social Practice Theory (SPT) may be a valuable addition to practitioner’s toolboxes by providing an alternative means of understanding the complex dynamics between the elements that constitute the practice of utility cycling, allowing it to be considered as a social issue, rather than focusing solely on individual behaviour. This is demonstrated within the paper by the use of SPT to reanalyse quantitative and qualitative datasets that explore views and experiences of both cyclists and non-cyclists. Therein, the practice of utility cycling is described according to its three elements; materials, meaning and competences and the potential benefits of this approach are discussed; particularly its ideological shift away from ‘victim blaming’ and its natural support of interdisciplinary intervention design.     

Step by step: A microgenetic study of the development of strategy choice in infancy

To examine patterns of strategy choice and discovery during problem‐solving of a novel locomotor task, 13.5‐ and 18‐month‐old infants were placed at the top of a staircase and encouraged to descend. Spontaneous stair descent strategy choices were documented step by step and trial by trial to provide a microgenetic account of problem‐solving in action. Younger infants tended to begin each trial walking, were more likely to choose walking with each successive step, and were more likely to lose their balance and have to be rescued by an experimenter. Conversely, older infants tended to begin each trial scooting, were more likely to choose scooting with each successive step, and were more likely to use a handrail to augment balance on stairs. Documenting problem‐solving microgenetically across age groups revealed striking similarities between younger infants' strategy development and older children's behaviour on more traditionally cognitive tasks, including using alternative strategies, mapping prior experiences with strategies to a novel task, and strengthening new strategies. As cognitive resources are taxed during a challenging task, resources available for weighing alternatives or inhibiting a well‐used strategy are reduced. With increased motor experience, infants can more easily consider alternative strategies and maintain those solutions over the course of the trial.     

Why employee psychopathy leads to counterproductive workplace behaviours : an analysis of the underlying mechanisms

ABSTRACT

The purpose of this study is to examine possible explanatory mechanisms linking employee secondary psychopathy to counterproductive workplace behaviour (CWB). Based on the emotion-centred model of voluntary work behaviour we argue that secondary psychopathy is characterized by unfavourable cognitive appraisal tendencies, which in turn positively relate to negative affectivity. We further assume that this cognitive-affective process enhances CWB. We also include primary psychopathy into our research model to test if the presumed mechanism applies to both psychopathy dimensions. We collected daily-survey data from 470 employees (1670 days) and analysed these data using multilevel structured equation modelling. We found strong support for the hypothesized serial mediation model, indicating that secondary psychopathy triggers dysfunctional cognitive-affective tendencies and consequently increases the likelihood of CWB. The proposed model did not hold up for primary psychopathy. Our study outlines the presence of distorted cognition-affective patterns in employee secondary psychopathy only. These patterns seem to play a key role in explaining the link between employee secondary psychopathy and deviant workplace behaviour. Based on this procedural knowledge relevant implications for theory and practice are provided.     

Problems in Psychiatric Genetic Research: A Reply to Faraone and Biederman

This article examines evidence cited in favor of the operation of genetic factors in attention-deficit hyperactivity disorder (ADHD). Like other psychiatric conditions, a belief in the genetic basis of ADHD is derived from the results of family, twin, and adoption studies. Because family studies are widely believed to be confounded by environmental factors, primary emphasis is placed on twin and adoption studies. ADHD twin studies depend on the validity of the equal environment assumption (EEA), which holds that the environments of identical (MZ) and fraternal (DZ) twins are the same. Here it is argued that however the EEA is defined, it cannot be accepted. Therefore, the greater similarity or concordance of MZ twins when compared to DZ twins is plausibly explained by environmental factors. Adoption studies constitute a third method for investigating the role of genetic factors in ADHD. It is argued that these studies are greatly flawed by factors including non blinded diagnoses and the failure to study the biological relatives of adoptees. After an examination of the total weight of evidence in favor of a genetic basis or predisposition for ADHD, it is concluded that a role for genetic factors is not supported and that future research should be directed toward psychosocial causes     

Support and compliance with 20 mph speed limits in Great Britain

There are a number of challenges relating to both the support of and compliance with speed limits. The introduction of 20 mph limits in Great Britain is no exception: the recent rise in the deployment of these limits in urban settings has created a need to understand these issues in more depth. This paper reports a study undertaken by the authors that used a population wide survey of GB drivers to explore how support and compliance were interlinked. Whilst as expected many supporters said they would comply with the limits, and many opponents might not comply, more surprisingly it was also found that some supporters claimed not to comply, while some opponents of 20 mph limits were compliers. Explanations included the strong likelihood of strong moral adherence to not breaking laws amongst opponent–compliers, and self-enhancement bias amongst supporter–non-compliers. This paper explores the incidence of these effects and their implications in detail.     

Triangulating causality between childhood obesity and neurobehavior: Behavioral genetic and longitudinal evidence

Childhood obesity is a serious health concern that is not yet fully understood. Previous research has linked obesity with neurobehavioral factors such as behavior, cognition, and brain morphology. The causal directions of these relationships remain mostly untested. We filled this gap by using the Adolescent Brain Cognitive Development study cohort comprising 11,875 children aged 9–10. First, correlations between the age- and sex-specific 95th BMI percentile (%BMIp95) and neurobehavioral measures were cross-sectionally analyzed. Effects were then aggregated by neurobehavioral domain for causal analyses. Behavioral genetic Direction of Causation modeling was used to test the direction of each relationship. Findings were validated by longitudinal cross-lagged panel modeling. %BMIp95 correlated with impulsivity, motivation, psychopathology, eating behavior, and cognitive tests (executive functioning, language, memory, perception, working memory). Greater %BMIp95 was also associated with reduced cortical thickness in frontal and temporal brain areas but with increased thickness in parietal and occipital areas. Similar although weaker patterns emerged for cortical surface area and volume. Behavioral genetic modeling suggested causal effects of %BMIp95 on eating behavior (β = 0.26), cognition (β = 0.05), cortical thickness (β = 0.15), and cortical surface area (β = 0.07). Personality/psychopathology (β = 0.09) and eating behavior (β = 0.16) appeared to influence %BMIp95. Longitudinal evidence broadly supported these findings. Results regarding cortical volume were inconsistent. Results supported causal effects of obesity on brain functioning and morphology. The present study highlights the importance of physical health for brain development and may inform interventions aimed at preventing or reducing pediatric obesity.

Research Highlights

• A continuous measure related to obesity, %BMIp95, has correlations with various measures of brain functioning and structure
• Behavioral genetic and longitudinal modeling suggest causal links from personality, psychopathology, and eating behavior to %BMIp95
• Results also indicate directional links from %BMIp95 to eating behavior, cognition, cortical thickness, and cortical surface area
• Obesity may play a role for healthy brain development during childhood