复述、提取抑制与有意遗忘的实验研究

有意遗忘是关于记忆研究的另一种实验范式,对于在实验中出现的要求遗忘的学习项目的记忆成绩差于要求记忆的项目的结果,目前主人存在着两种理论解释：基于编码的理论和基于提取抑制的理论。该实验运用单字提示方式,采用控制较为严密的补笔测验方法和加入所谓的“无关项”,对有意遗忘的心理过程进行进一步的探讨,实验表明：在单了提示方式下,被试在进行有意遗忘的过程中,既对记忆项进行了有意的复述又对遗忘项产生了提取抑制。     

自我威胁刺激对返回抑制的影响

使用线索-靶子范式,分别将面孔刺激作为线索(实验1)和靶子(实验2)呈现,考察自我扭曲面孔对返回抑制的影响。结果发现:自我扭曲面孔为线索时,与他人扭曲、他人中性面孔产生了实验性分离,自我扭曲面孔的返回抑制效应量最小;自我扭曲面孔为靶子时,返回抑制消失。研究结果揭示:以自我扭曲面孔为代表的自我威胁刺激激活了威胁性自我概念,缩小了注意搜索范围,可能是导致人际紧张的潜在自我认知机制。     

自主性动机对创造力的预测：认知抑制的调节作用

执行功能和动机是构成创造力个体差异的重要因素。本研究采用Stroop任务、自主性动机和创造力量表对372名大学生进行问卷调查及实验室研究,以考察认知抑制在自主性动机预测创造力中的调节作用。结果发现：（1）自主性动机显著正向预测流畅性/灵活性/独创性。（2）认知抑制调节自主性动机对流畅性与灵活性的预测作用。具体表现为对高认知抑制能力者,自主性动机显著正向预测流畅性和灵活性,对低认知抑制能力者这一预测作用不显著。该研究深化了自主性动机与创造力关系的研究。     

指向性遗忘中认知抑制能力的发展

采用组块提示法,以小学二年级、五年级学生和大学生共104名为被试,考察语词记忆指向性遗忘效应的机制及其发展.结果表明:(1)各年龄段被试都明显存在着指向性遗忘效应;(2)指向性遗忘效应是提取抑制和选择性编码两种机制共同作用的结果,且年幼儿童与年长儿童、成人之间作用情况可能存在差异;(3)小学二年级、五年级学生和大学生之间认知抑制能力差异显著,随年龄增长,认知抑制能力不断增长.     

抑郁情绪大学生反应抑制的眼动特点

本研究采用反眼动实验范式,通过BDI和 SDS量表筛选11名抑郁情绪组和12名正常对照组被试,以高兴、中性和悲伤面孔图片作为实验材料,旨在探讨抑郁情绪大学生对情绪刺激的眼动抑制能力。结果发现：抑郁情绪组的正确眼跳反应时慢于正常组;反朝向眼跳任务中,抑郁情绪组在情绪面孔图片,尤其是悲伤面孔图片上的错误率比正常组高。研究表明,抑郁情绪个体的认知加工速度慢于正常个体,且对情绪面孔,尤其对悲伤面孔存在抑制缺陷。     

辨别任务中线索有效性对儿童返回抑制发展的影响

采用辨别任务范式,运用三个3×3×3的混合实验设计(线索有效性、年级、靶子出现位置),考察了3种线索-靶子时间间隔下线索有效性对辨别任务中儿童返回抑制发展的影响。结果表明:(1)随着年龄的增长,儿童完成任务所需的反应时逐渐缩短;(2)随着年龄的增长,儿童出现返回抑制所需的SOA缩短;(3)辨别任务的难度比较大,所需的认知技能和工作记忆更多,因此返回抑制出现的机制也就更复杂,线索有效性对辨别任务下的返回抑制的影响并没有呈现出规律性的变化。     

An equation for behavioral contrast.

Pigeons were trained on a three-component multiple schedule in which the rates of reinforcement in the various components were systematically varied. Response rates were described by an equation that posits that the response-strengthening effects of reinforcement are inversely related to the context of reinforcement in which it occurs, and that the context is calculated as the weighted average of the various sources of reinforcement in the situation. The quality of fits was comparable to that found with previous quantitative analyses of concurrent schedules, especially for relative response rates, with over 90% of the variance accounted for in every case. As with previous research, reinforcements in the component that was to follow received greater weights in determining the context than did reinforcements in the preceding component.     

Dynamic effects of food magnitude on interim-terminal interaction

We tested the assumption of a facilitatory relation between periodic food presentation and schedule-induced drinking by examination of (a) elicited drinking, (b) drinking in anticipation of food delivery, and (c) possible indirect effects of food delivery on drinking. We exposed rats to a fixed-time 60-second schedule in which interfood intervals ended in either one or four food pellets with equal probability. In Phases 1 and 3, a stimulus signaled the magnitude of upcoming food presentation. In Phase 2, the stimulus was eliminated. Changes in drinking and “head-in-feeder” distributions within interfood intervals demonstrated that head-in-feeder was controlled directly by food presentation, but drinking was not. Head-in-feeder increased and drinking was reduced when large meals began or ended an interval. In Phases 4 to 6, meal size was manipulated across sessions yielding a positive relation between meal size and schedule-induced drinking. We conclude: (1) Schedule-induced drinking is determined by distributions of food-related behavior and results from indirect effects of food delivery; and (2) the amount of schedule-induced drinking and the form of the drinking distributions in this experiment can be accurately explained by two assumptions: (a) Food presentation facilitates food-related behavior through elicitation and anticipation; and (b) food-related behavior and drinking are reciprocally, linearly related.     

SCHEDULE-INDUCED DRINKING: ELICITATION,ANTICIPATION, OR BEHAVIORAL INTERACTION?

We carried out five experiments with rats on fixed-time schedules in order to define the relation between drinking and individual food-pellet presentations. In Experiment 1, unsignaled extra food occurred at the end of occasional fixed intervals, and we compared subsequent drinking patterns with drinking before the extra food presentation. In Experiment 2 we presented signaled and unsignaled extra food and measured elicited and anticipatory drinking patterns. In Experiment 3, we observed the persistence of modified drinking patterns when several consecutive intervals ended with extra pellets. In Experiments 4 and 5, we varied the magnitude of food delivery across (rather than within) sessions to replicate published findings. Results show that schedule-induced drinking is neither elicited by food presentations nor induced by stimuli associated with a high food rate. All subjects seemed to follow a simple rule: during any stimulus signaling an increase in the local probability of food delivery within a session, engage in food-related behavior to the exclusion of drinking. Schedule-induced drinking appears to be the result of dynamic interactions among food-related behavior, drinking, and other motivated behavior, rather than a direct effect of the contingencies of food reinforcement.     

Emotional behavior as an agenda in neuropsychological evaluation

In an effort to objectify neuropsychologic evaluations, consideration of a patient's emotional behavior has often been neglected. An extensive literature review is undertaken in an effort to document lateralized emotional behaviors commonly found in brain injury populations. This evidence is contrasted with the psychiatric symptoms and lateralized neuropsychologic impairments seen in major depression and schizophrenia. A theoretical model is then proposed that attempts to integrate these functional vs. organic symptoms based upon reciprocal inhibition of lateralized emotional functioning in brain injury and psychiatric disorders. This opponent process model not only seems to account for some of the discrepant findings in the literature, but additionally provides a cogent and useful marker to neurophychologically differentiate neuronal vs. metabolic disorders. The model further suggests new ways of envisioning treatment and recovery from both psychiatric illness and brain injury.