首页 | 本学科首页   官方微博 | 高级检索  
文章检索
  按 检索   检索词:      
出版年份:   被引次数:   他引次数: 提示:输入*表示无穷大
  收费全文   5810篇
  免费   1139篇
  国内免费   35篇
  6984篇
  2019年   74篇
  2018年   107篇
  2017年   130篇
  2016年   121篇
  2015年   73篇
  2014年   102篇
  2013年   599篇
  2012年   149篇
  2011年   181篇
  2010年   187篇
  2009年   258篇
  2008年   230篇
  2007年   217篇
  2006年   146篇
  2005年   178篇
  2004年   171篇
  2003年   178篇
  2002年   130篇
  2001年   156篇
  2000年   97篇
  1999年   123篇
  1998年   135篇
  1997年   95篇
  1996年   131篇
  1995年   108篇
  1994年   117篇
  1993年   91篇
  1992年   137篇
  1991年   109篇
  1990年   111篇
  1989年   114篇
  1988年   103篇
  1987年   78篇
  1986年   89篇
  1985年   72篇
  1984年   88篇
  1982年   77篇
  1981年   73篇
  1980年   75篇
  1976年   69篇
  1975年   70篇
  1974年   66篇
  1973年   87篇
  1972年   80篇
  1971年   76篇
  1970年   65篇
  1968年   95篇
  1966年   68篇
  1964年   85篇
  1963年   79篇
排序方式: 共有6984条查询结果,搜索用时 15 毫秒
971.
972.
It has been hypothesized that some types of personality disorders tend to remit with age whereas others may become more prominent. The present study determined the prevalence and nature of 13 personality disorders with a self-report inventory (Coolidge Axis II Inventory) in an older group of chronically mentally ill inpatients (N = 30, mean age = 63 years) and a younger group of similar patients (N = 30, mean age = 39 years). All patients met DSM-IV criteria for either Schizophrenia, Schizoaffective Disorder, Bipolar Disorder, or recurrent severe Major Depression with psychotic features. The prevalence rate of personality disorders was high for both groups: 58% for the older sample and 66% for the younger group. The younger group was more likely to be diagnosed Antisocial, Borderline, Passive-Aggressive, Sadistic, and Schizotypal, but the groups were not different in the rates of Obsessive-Compulsive Disorder. This study supports the hypothesis that some personality disorders remit with age. However, no evidence was found to suggest that other personality disorders become more prominent in older adult psychiatric populations. Suggestions for future research are offered.  相似文献   
973.
974.
ANGER AND DEPRESSION IN GIRLS AND BOYS A Study of Gender Differences   总被引:1,自引:0,他引:1  
A growing body of literature supports the link between anger suppression and depression and females'greater likelihood than males of demonstrating both. Anger suppression was hypothesized to be involved in the development of gendered identity for girls, specifically by rendering girls more likely to experience depression. Employing an ethnically diverse sample of public school children, differences between fifth through ninth grade girls and boys in anger suppression and depression were investigated using self-report data. Results supported the hypothesis that girls suppress anger at higher rates than boys but not the related hypothesis that this suppression results in higher levels of depression. Age was not related to either anger suppression or depression, and no significant relationship was found between suppressed anger and depression for either sex. The impact of girls'anger suppression on their emotional and gender development is discussed.  相似文献   
975.
976.
977.
One of the hallmarks of the aging process is a loss of sensitivity in central neuronal receptors to agonist stimulation. This appears to be especially true in central (hippocampal, striatal) muscarinic cholinergic systems and in the striatal dopamine systems. For these two systems, any decline in their sensitivity can be of extreme importance in determining the behavioral capabilities of the organism. Decrements in the striatal dopamine system may be reflected as motor behavioral deficits, while the central cholinergic systems play a major role in the processing of memory through the activation of muscarinic receptors (mAChR). Declines in the function of these receptors appear to be at least partially responsible for the marked deterioration of cognitive function in normal aging and, more notably, in Alzheimer’s disease (AD). Previous work has indicated only minimal success in improving performance in tasks that assess memory in senescent animals or humans with pharmacological agents which enhance cholinergic functioning. The present review describes research that indicates that two of the factors involved in this decline in receptor sensitivity include: (a) decreased receptor concentrations and (b) age-related decrements in signal transduction pathways. Studies are reviewed that indicate that the oxidative neural damage that occurs via kainic acid or ionizing radiation parallel those seen in aging. It is suggested that the common mechanism that may exist among all of the age-, disease-, excitatory amino acid- or radiation-induced deficits in neuronal transmission may involve free-radical-mediated alterations in membrane integrity through lipid peroxidation.  相似文献   
978.
979.
The key pecking of two pigeons was reinforced on a variable-interval schedule of reinforcement during the presentation of each of two stimuli. In various phases of the experiment, punishment followed every response emitted in the presence of one of the stimuli. In general, when the rate of punished responding changed during the presentation of one stimulus, the rate of unpunished responding during the other stimulus changed in the opposite direction. This sort of change in rate is an example of behavioral contrast. When punishment was introduced, the rate of punished responding decreased and the rate of unpunished responding increased as functions of shock intensity. When the rate of previously punished responding increased after the termination of the shock, the rate of the always unpunished responding decreased. When the procedure correlated with a red key was changed from variable-interval reinforcement and punishment for each response to extinction and no punishment, the rate of reinforced responding during presentations of a green key decreased and then increased while the rate of the previously punished responding during red first increased and then decreased during extinction.  相似文献   
980.
设为首页 | 免责声明 | 关于勤云 | 加入收藏

Copyright©北京勤云科技发展有限公司  京ICP备09084417号