佛山市急性阑尾炎住院费用比较分析

对医保与非医保患者费用进行比较,分析医保病例的费用特点及相应的费用控制办法。采用协方差分析和秩和检验分析两组患者的费用情况。结果显示,在急性单纯性阑尾炎治疗费用上,医保患者的费用高于非医保患者。提倡政府应加强对医疗费用的监督,改革供方支付方式,建立公平竞争的医疗市场机制等。     

关于药物临床试验伦理问题的思考

医学伦理学是伴随着科学技术的发展而产生的事物,药物临床试验在促进人类健康事业发展的同时,也涉及到与之相驳的伦理学问题,运用哲学的矛盾原理,浅析药物临床试验与医学伦理之间存在的矛盾及矛盾的两面性,以求解决目前药物临床试验中遇到的困难和问题。     

孔子的“义利观”与医院的经营之道

义利关系是中国传统思想中与政治、经济、文化最为密切的一种价值观。以孔子“义利观”为代表的中国传统企业经营的指导思想,对医院构建遵守道德规范、讲求诚信、以人为本、完善激励机制、“以质量为核心”等经营之道具有重大的理论价值和现实意义,它将引导医院实现其所追求的道德观念和法律价值,达到道义与功利综合的发展。     

孔子的"义利观"与医院的经营之道

义利关系是中国传统思想中与政治、经济、文化最为密切的一种价值观.以孔子"义利观"为代表的中国传统企业经营的指导思想,对医院构建遵守道德规范、讲求诚信、以人为本、完善激励机制、"以质量为核心"等经营之道具有重大的理论价值和现实意义,它将引导医院实现其所追求的道德观念和法律价值,达到道义与功利综合的发展.     

青少年期母子性格与家庭亲密度的关系:性格喜好的作用

研究旨在考察性格喜好与母子性格、家庭成员亲密度的关系以及性格喜好在其他两个变量之间的中介作用。成都市632对初高中学生及其母亲分别自评并填写了艾森克人格问卷,同时评价喜欢对方性格的程度。另外,青少年还报告了与家庭成员的亲密程度。通过相关分析、多元方差分析、层级回归分析以及结构方程模型建模,结果发现：性格喜好与母亲性格、青少年性格均有显著关系,青少年对母亲的性格喜好程度与家庭成员间的亲密感有显著正相关。青少年性格和青少年对母亲的性格喜好程度能直接预测家庭成员亲密度,同时青少年和母亲的性格还通过青少年对母亲的性格喜好程度间接作用于家庭亲密度,提示性格喜好在二者的关系中发挥了重要的中介作用。     

负启动效应的认知神经科学研究

近几年,对负启动效应的神经机制研究发现,干扰项抑制与情节提取都是引起负启动的原因,究竟哪种机制起主要作用与实验任务有很大的关系。从现有的研究结果来看,位置负启动与前额皮层有关,EPR上表现为P1和N1波幅减小或N2波幅的增大,支持抑制机制。特性负启动P3成分仍存在不一致的结果,脑区的广泛激活模式使得研究者越来越倾向采取整合的观点。     

原始佛教的心理思想

本文以四部汉译阿含经为主要依据,从心理学角度探索了原始佛教教义。原始佛教认为心性本净,被其执着妄动生出的有为心法染污,从而产生各种心理结构和心理机能,即由眼、耳、鼻、舌、身、意五根和触、受、思、念、意、识等功能构成人类有情的心理整体;根据这些结构和机能对修行解脱的作用,分为染污烦恼和善心净法两种,并以后者对治前者。修行者借助这些心理机能修习戒、定、慧,依靠止心、观心、勤修习心这三个原则,达到止息心念妄动,离欲解脱的目的。     

《道德经》与现代管理

老子哲学对改善现代管理具有深刻的智慧启迪。在无为与自然的关系中,老子把无为视为实现自然理想的道路、方法,把自然视为无为方式所要达到的目标或理想境界。老子依据道德本体论形成了“无为——自然”理念,然后又将其推演、贯彻于人类社会,作为人类社会中效法道的典范的圣人来看,圣人通过无为、好静、无事、无欲等方式、方法取得了民众白化、自正、自富、自朴的效果。     

Iconic gestures prime related concepts: An ERP study

To assess priming by iconic gestures, we recorded EEG (at 29 scalp sites) in two experiments while adults watched short, soundless videos of spontaneously produced, cospeech iconic gestures followed by related or unrelated probe words. In Experiment 1, participants classified the relatedness between gestures and words. In Experiment 2, they attended to stimuli, and performed an incidental recognition memory test on words presented during the EEG recording session. Event-related potentials (ERPs) time-locked to the onset of probe words were measured, along with response latencies and word recognition rates. Although word relatedness did not affect reaction times or recognition rates, contextually related probe words elicited less-negative ERPs than did unrelated ones between 300 and 500 msec after stimulus onset (N400) in both experiments. These findings demonstrate sensitivity to semantic relations between iconic gestures and words in brain activity engendered during word comprehension.     

Intrahippocampal administration of A beta(1-40) impairs spatial learning and memory in hyperglycemic mice

Age-related neurodegenerative dementia, particularly Alzheimer's disease (AD), may be exacerbated by several interacting risk factors including genetic predisposition, beta amyloid (A beta) protein accumulation, environmental toxins, head trauma, and abnormal glycolytic metabolism. We examined the spatial learning and memory effects of A beta(1-40) administration on hyperglycemic mice by their performance in the Morris water maze. Chronic hyperglycemia was induced in male C57BL/6J mice to mimic diabetes mellitus by intraperitoneal injection of streptozotocin (STZ), which specifically destroys pancreatic beta-islet cells. Ten days after STZ treatment, intrahippocampal infusion of vehicle, monomer, or oligomer A beta(1-40) was given to these hyperglycemic mice. Our results demonstrate that in comparison with vehicle or monomer A beta(1-40), oligomer A beta(1-40) induced significant deficits of spatial learning and memory in hyperglycemic mice. Apoptotic signals were identified in the CA1 and dentate gyrus of hippocampus in hyperglycemic mice. A beta accumulation, oxidative stress, and apoptosis in the CA1 region were more intensive in hyperglycemic mice than that in normoglycemic mice after acute treatment with oligomer A beta(1-40) peptide treatment. These results indicate that CA1 apoptosis was enhanced by oxidative stress resulting from accumulation of A beta. Considered together, these findings suggest that hyperglycemic mice are more vulnerable to the A beta-induced-oxidative stress than normal subjects. We therefore propose that A beta accumulation would be enhanced by hyperglycemia, and the oxidative stress caused by A beta accumulation would in turn enhance the AD symptoms.