论王阳明龙场所悟之“道”

明正德三年(1508)王阳明在贵州龙场悟道,这是他学术思想上的分水岭。阳明龙场所悟之道,从直接的内容看,不外是对格物致知之说的重新理解,而其深层的内涵则颇为复杂,主要包括了心外无理、心外无物和致知格物三个内容。     

先秦国家安全思想及其当代借鉴价值

在中国历史上,关于国家安全问题的战略性思考起源于先秦时期,许多重要的指导思想和原则,如以民为本、和而不同、慎战、亲仁善邻等思想已在那时得到了明晰。这些宝贵的思想具有久远而深厚的文化基础。他们不仅为历朝历代安内攘外,维系中国作为世界上唯一不曾中断几千年文明进程的国家发挥了重要的历史作用。而且对于我们今天研究安全问题、制定国家安全战略提供了可资借鉴的价值。     

社会判断偏差的分化研究：人物因素的影响

实验采用投射的方法,利用图片和问题材料,考察了128名本科生对物、事、人的社会判断偏差的分化现象。实验结果表明：1）社会判断内在的宽大效应存在局限性,它是有条件限制的,受到所介入的人物因素的影响而出现分化现象;2）对物与事的评价中,突出了对性别判断标准的差异;3）在社会人的对比中,容貌、性别、背景在对同一事件的判断中发挥着重要作用,使社会判断出现偏差分化。     

上清派炼养术的藏象学说内涵探讨

上清派是道教的一个重要流派,形成于魏晋时期,创始人相传为西晋女道士魏华存。该派以《上清经》、《黄庭经》、《真诰》等道书为主要经典,以存思、守神、行气、服食为主要修炼手段,对道学及中医学都有着深远的影响。魏晋时     

Acute starvation in C57BL/6J mice increases myocardial UCP2 and UCP3 protein expression levels and decreases mitochondrial bio-energetic function

Associations between uncoupling protein (UCP) expression and functional changes in myocardial mitochondrial bio-energetics have not been well studied during periods of starvation stress. Our aim was to study the effects of acute starvation, for 24 or 48 h, on combined cardiac mitochondrial function and UCP expression in mice. Isolated heart mitochondria from female mice starved for 48 h compared to that from mice fed revealed a significantly (p < 0.05) decreased adenosine diphosphate-to-oxygen ratio, a significantly increased proton leak and an increased GTP inhibition on palmitic acid-induced state 4 oxygen consumption (p < 0.05). These bio-energetic functional changes were associated with increases in mitochondrial UCP2 and UCP3 protein expression. In conclusion, our findings suggest that increased UCP2 and UCP3 levels may contribute to decreased myocardial mitochondrial bio-energetic function due to starvation.     

论犹太教对外邦人改宗的矛盾态度

犹太教是一个民族性的宗教。对改宗犹太教的外邦人,拉比们一方面给予他们积极的评价,另一方面又对他们表现出怀疑排斥的消极态度。从根本上说,这种矛盾态度是由犹太教对自身的不同理解造成的。一方面,犹太人将自身看成由律法塑造的宗教共同体;另一方面,犹太教又始终将自身理解为一个民族,甚至其改宗程序,也是为了在象征意义上建立改宗者与犹太人的血缘关系。犹太教对其民族性的坚持是她无法向外邦人敞开怀抱的主要原因。     

仪式过程与符号象征——索伦鄂温克火神祭祀仪式的田野研究

本文立足于对索伦鄂温克人岁时仪式中火神祭祀仪式的田野调查资料,借鉴了范·杰内普和特纳的仪式研究理论及方法,从象征的角度分析了火神祭祀的仪式过程、仪式结构与秩序,从而描述仪式所表达的深层意义:有形世界与无形世界并非毫不相干,人也并不总在被动地接受神的安排,神秘甚至是危险的世界在仪式中变得可以接触。更为重要的是,对于仪式的参与者来说,一切行为都是可以解释的,而不是毫无理性地随意而为。     

Emotion Regulation and Stress

This paper examines the similarities and differences between emotion regulation and stress coping and reviews research that suggests that the association between emotion regulation and stress may be explained by the common neural structures. Developmental changes related to emotion regulation and stress are also discussed. Overall, the research suggests that individuals vary in their ability to regulate emotions and cope with stress, and these abilities may differ across age. Little is known, however, about the factors that influence individual differences in emotion regulation and stress coping. We suggest that behavioral genetic designs may be an important avenue for future research. Such research would indicate the extent to which variation in emotion regulation and stress are due to genetic and/or environmental influences and further the extent to which common genetic and/or environmental factors explain the links between emotion regulation and stress.     

Ursolic acid attenuates lipopolysaccharide-induced cognitive deficits in mouse brain through suppressing p38/NF-κB mediated inflammatory pathways

Evidence indicates that systemic administration of lipopolysaccharide (LPS) induces brain inflammation, ultimately resulting in cognitive deficits. Ursolic acid (UA), a plant-derived pentacyclic triterpenoid, is well known to possess multiple biological functions, including antioxidant, anti-tumor and anti-inflammatory activities. In the present study, we assessed the protective effect of UA against the LPS-induced cognitive deficits in mice. We found that UA significantly improved cognitive deficits of LPS-treated mice in open field, step-through passive avoidance and Morris water maze tasks. One potential mechanism of this action was attributed to the decreased production of pro-inflammatory markers including COX-2, iNOS, TNF-α, IL-1β, IL-2 and IL-6 in LPS-treated mouse brain. Mechanistically, UA markedly inhibited LPS-induced IκBα phosphorylation and degradation, NF-κB p65 nuclear translocation and p38 activation in mouse brain, but did not affect the activation of TLR4, MyD88, ERK, JNK and Akt. Taken together, these results suggest that UA may be useful for mitigating inflammation-associated brain disorders by inhibiting pro-inflammatory factors production, at least in part, through blocking the p38/NF-κB signaling pathways.