Narrative self-constitution and vulnerability to co-authoring

All people are vulnerable to having their self-concepts shaped by others. This article investigates that vulnerability using a theory of narrative self-constitution. According to narrative self-constitution, people depend on others to develop and maintain skills of self-narration and they are vulnerable to having the content of their self-narratives co-authored by others. This theoretical framework highlights how vulnerability to co-authoring is essential to developing a self-narrative and, thus, the possibility of autonomy. However, this vulnerability equally entails that co-authors can undermine autonomy by contributing disvalued content to the agent’s self-narrative and undermining her authorial skills. I illustrate these processes with the first-hand reports of several women who survived sexual abuse as children. Their narratives of survival and healing reveal the challenges involved in (re)developing the skills required to manage vulnerability to co-authoring and how others can help in this process. Finally, I discuss some of the implications of co-authoring for the healthcare professional and the therapeutic relationship.     

Taboo desires,creativity, and career choice

Two studies suggest that Protestants are more likely than Catholics or Jews to sublimate taboo desires into motives to pursue creative careers. The results are consistent with a synthesis of psychological and classic sociological theories. In Study 1, Protestants induced to have taboo sexual desires were likely to express a preference for creative careers (as opposed to prosocial ones). In Study 2, a national probability sample revealed that “conflicted” Protestants—who had taboo desires but tried to rule their sexual behavior according to their religious beliefs—worked in the most creative jobs. The effects in both studies did not hold for Catholics and Jews. Results suggest that intrapsychic conflict can partially motivate important real-world decisions, such as the choice to pursue a creative career.     

Differences between traces of negative emotions in smile judgment

While a smile can reflect felt happiness, it can also be voluntarily produced, for instance, to mask negative emotions. Masking strategies are not always perfect and traces of the negative emotion can leak. The current study examined the role of traces of anger, sadness, fear and disgust in the judgment of authenticity of smiles. Participants judged the authenticity of the smiles while their eye movements were recorded. They were also asked if the stimuli comprised another emotion and, if so, what the emotion was. Results revealed that participants were sensitive to traces of negative emotions. Variations were observed between emotions with performance being best for traces of fear and lowest for traces of anger in the eyebrows in the judgment task. However, when the presence of a negative emotion was reported, participants were less accurate in identifying fear but more accurate in identifying anger. Furthermore, variations were observed as a function of the location of the trace whether in the mouth or eyes as a function of the emotion. Traces in the eyebrows were associated with better performance than traces in the mouth for sadness but the opposite was observed for anger. The performance at the judgment task was not linked to eye movement measures or explicit knowledge of the masked emotion. Future research should explore other explanation for the variations in performance in the judgments of authenticity of masking smiles such as emotional contagion.     

Primal emotional-affective expressive foundations of human facial expression

Emotional facial expressions provide important insights into various valenced feelings of humans. Recent cross-species neuroscientific advances offer insights into molecular foundations of mammalian affects and hence, by inference, the related emotional/affective facial expressions in humans. This is premised on deep homologies based on affective neuroscience studies of valenced primary emotional systems across species. Thus, emerging theoretical perspectives suggest that ancient cross-species emotional systems are intimately linked not only to emotional action patterns evident in all mammals, but also, by inference, distinct emotional facial expressions studied intensively in humans. Thus, the goal of the present theoretical work was to relate categories of human emotional facial expressions—e.g. especially of anger fear, joy and sadness—to respective underlying primary cross-mammalian emotional circuits. This can potentially provide coherent theoretical frameworks for the eventual molecular study of emotional facial expressions in humans.     

Why are attitude–behaviour inconsistencies uncomfortable? Using motivational theories to explore individual differences in dissonance arousal and motivation to compensate

Using cognitive dissonance theories and self-determination theory, we explored the role of individual differences in global and contextual motivational orientations on dissonance arousal processes following spontaneous attitude–behaviour inconsistencies (ABIs). Study 1 (N = 382) showed that individual differences in global motivation relate to the frequency of ABIs and dissonance arousal across important life domains. Studies 2 (N = 282) and 3 (N = 202) showed that individual differences in contextual motivation toward the environment predict the relative frequency of ABIs and the quantity and quality of proximal motivation to compensate for ABIs in that context. Autonomous motivation was associated with a tendency to compensate for ABIs to both reduce dissonance and restore self-integrity. Controlled motivation disposed individuals to reduce dissonance to protect ego-invested self-structures, and to be indifferent to non self-threatening ABIs. Amotivation left people indifferent to ABIs. Individual differences in motivational orientations could explain why ABIs are uncomfortable and motivate people to compensate differently when they face ABIs.     

A Comparison of Structural Brain Imaging Findings in Autism Spectrum Disorder and Attention-Deficit Hyperactivity Disorder

ASD and ADHD are regarded as distinct disorders in the current DSM-5. However, recent research and the RDoC initiative are recognizing considerable overlap in the clinical presentation of ASD, ADHD, and other neurodevelopmental disorders. In spite of numerous neuroimaging findings in ASD and ADHD, the extent to which either of the above views are supported remains equivocal. Here we compare structural MRI and DTI literature in ASD and ADHD. Our main findings reveal both distinct and shared neural features. Distinct expressions were in total brain volume (ASD: increased volume, ADHD: decreased volume), amygdala (ASD: overgrowth, ADHD: normal), and internal capsule (ASD: unclear, ADHD: reduced FA in DTI). Considerable overlap was noted in the corpus callosum and cerebellum (lower volume in structural MRI and decreased FA in DTI), and superior longitudinal fasciculus (reduced FA in DTI). In addition, we identify brain regions which have not been studied in depth and require more research. We discuss relationships between brain features and symptomatology. We conclude by addressing limitations of current neuroimaging research and offer approaches that account for clinical heterogeneity to better distinguish brain-behavior relationships.     

The Underlying Role of Negative Affect in the Association between PTSD,Major Depressive Disorder,and Generalized Anxiety Disorder

Clinicians and researchers have found differential diagnosis to be difficult, particularly for conceptually similar disorders. One category of particular interest has been distress or internalizing disorders, theorized to be related via an underlying construct of generalized distress or negative affect. The present study attempted to address the comorbidity of three distress disorders - posttraumatic stress disorder (PTSD), major depressive disorder (MDD), and generalized anxiety disorder (GAD) - using latent analyses by controlling for the variance attributable to negative affect. The sample consisted of 265 trauma-exposed individuals who completed self-report measures of PTSD, MDD, GAD, and negative affect. Confirmatory factor analysis was used to test initial model fit. Next, the model was re-computed, controlling for negative affect by regressing negative affectivity at the item-level. Results indicated that a significant amount of variance within and between these diagnostic categories is attributable to negative affect at both the item- and factor-level. The hypothesis that MDD’s non-somatic/affective factor and the GAD factor would have the highest attenuations in factor loadings after controlling for negative affect was supported. Therefore, negative affect significantly influences the co-occurrence of PTSD, MDD, and GAD clinically, emphasizing the need for transdiagnostic interventions for trauma victims.