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Conclusion Here then is my theoretical model of the person who emerges from therapy—a person functioning freely in all the fullness of his organismic potentialities; a person who is dependable in being realistic, self-enhancing, socialized and appropriate in his behavior; a creative person, whose specific formings of behavior are not easily predictable; a person who is ever changing, ever developing, always discovering himself and the newness in himself in each succeeding moment of time. This is the person who in an imperfect way actually emerges from the experience of safety and freedom in a therapeutic experience, and this is the person whom I have tried to describe for you in pure form.My purpose has not been to convince you of the correctness of this view. Indeed I would have to confess that I have written this paper primarily for my own satisfaction, to clarify the thoughts which have been stirring in me. But if this presentation causes you to formulate your view of the person who emerges from therapy, or enables you to point out flaws in my own thinking which I have not yet seen, or arouses in you the desire to put to objective test either this picture or one which you paint for yourself, then it will have fully served both its primary and its secondary purpose.Reprinted by permission from Psychotherapy: Theory, Research and Practice, Vol. 1, No. 1, August, 1963. 相似文献
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11beta-hydroxysteroid dehydrogenase type 1 (11beta-HSD-1) catalyses the interconversion of active cortisol and corticosterone with inert cortisone and 11-dehydrocorticosterone, thus regulating glucocorticoid access to intracellular receptors. In rats, chronic glucocorticoid excess or stress increases 11beta-HSD-1 in the hippocampus, producing suggestions that it may attenuate the deleterious effects of chronic glucocorticoid excess. However, 11beta-HSD-1 predominantly catalyses 11beta-reduction in the intact liver and hippocampal cells, thus regenerating active glucocorticoids from inert substrate. We studied 11beta-HSD activity in the tissues of male tree shrews following 28 days of sustained psychosocial stress or exogenous administration of cortisol. In the hippocampus, chronic psychosocial stress attenuated 11-HSD-1 activity (69 +/- 9% of control), whereas cortisol alone had no effect. In the liver, both chronic stress and cortisol administration decreased 11beta-HSD-1 activity (47 +/- 11% and 49 +/- 4% fall, resp.). Attenuation of 11beta-HSD-1 within tissues may reflect a homeostatic mechanism designed to minimise the adverse effects of prolonged stress and/or glucocorticoid excess. 相似文献
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AN EMPIRICAL INVESTIGATION OF THE PREDICTORS OF EXECUTIVE CAREER SUCCESS 总被引:12,自引:0,他引:12
TIMOTHY A. JUDGE DANIEL M. CABLE JOHN W. BOUDREAU ROBERT D. BRETZ JR. 《Personnel Psychology》1995,48(3):485-519
This study examined the degree to which demographic, human capital, motivational, organizational, and industrylregion variables predicted executive career success. Career success was assumed to comprise objective (pay, ascendancy) and subjective (job satisfaction, career satisfaction) elements. Results obtained from a sample of 1,388 U.S. executives suggested that demographic, human capital, motivational, and organizational variables explained significant variance in objective career success and in career satisfaction. Particularly interesting were findings that educational level, quality, prestige, and degree type all predicted financial success. In contrast, only the motivational and organizational variables explained significant amounts of variance in job satisfaction. These findings suggest that the variables that lead to objective career success often are quite different from those that lead to subjectively defined success. 相似文献