Lateralization of narrow-band noise by blind and sighted listeners

The effects of varying interaural time delay (ITD) and interaural intensity difference (IID) were measured in normal-hearing sighted and congenitally blind subjects as a function of eleven frequencies and at sound pressure levels of 70 and 90 dB, and at a sensation level of 25 dB (sensation level refers to the pressure level of the sound above its threshold for the individual subject). Using an 'acoustic' pointing paradigm, the subject varied the IID of a 500 Hz narrow-band (100 Hz) noise (the 'pointer') to coincide with the apparent lateral position of a 'target' ITD stimulus. ITDs of 0, +/-200, and +/-400 micros were obtained through total waveform delays of narrow-band noise, including envelope and fine structure. For both groups, the results of this experiment confirm the traditional view of binaural hearing for like stimuli: non-zero ITDs produce little perceived lateral displacement away from 0 IID at frequencies above 1250 Hz. To the extent that greater magnitude of lateralization for a given ITD, presentation level, and center frequency can be equated with superior localization abilities, blind listeners appear at least comparable and even somewhat better than sighted subjects, especially when attending to signals in the periphery. The present findings suggest that blind listeners are fully able to utilize the cues for spatial hearing, and that vision is not a mandatory prerequisite for the calibration of human spatial hearing.     

Selective influence of second target exposure duration and Task1 load effects in the attentional blink phenomenon

The purpose of the experiments was to constrain the locus of interference in the attentional blink (AB) paradigm. Two visual stimuli, T₁ and T₂, were shown 300 msec apart, and each was followed by a mask. T₁ was an “H,” an “S,” an “ &, ” or a blank field; T₂ consisted of five letters. In Task₁, blank fields and & characters could be ignored, whereas Hs and Ss had to be identified and reported. Task₂ was always to report as many letters as possible from T₂. Task₂ performance was lower when T₁ had to be reported, as expected from the attentional blink phenomenon (AB). The exposure duration of T₂ was also manipulated. More letters could be reported as exposure duration was increased. However, this effect was additive with manipulations of Task₁ processing load that produced the AB effect. Log-linear analyses assuming that effects of T₂ exposure duration and Task₁ load effects occur at functionally distinct stages of processing provided satisfactory fits to the results, suggesting that none of the AB effect occurs as early as those of T₂ exposure duration. The results suggest that the locus of the AB effect is later than the stage(s) of processing affected by exposure duration.     

Testing the predictions of the central capacity sharing model

The divergent predictions of 2 models of dual-task performance are investigated. The central bottleneck and central capacity sharing models argue that a central stage of information processing is capacity limited, whereas stages before and after are capacity free. The models disagree about the nature of this central capacity limitation. The central bottleneck model claims that central processing acts on only 1 task at a time and, therefore, constitutes a bottleneck that processes tasks serially. The central capacity sharing model postulates that the central stage is a limited-capacity parallel processor that divides resources among to-be-performed tasks. As a result of this difference, in the psychological refractory period paradigm, the central capacity sharing model predicts that lengthening Task 2 precentral processing will improve Task 1 performance at short stimulus onset asynchronies, whereas the central bottleneck model does not. Results of 2 experiments confirm the prediction of the central capacity sharing model.     

Changes in the neuropsychological correlates of clinical dimensions between the acute and stable phase of schizophrenia

This study was designed to investigate whether the neuropsychological correlates of the symptom dimensions of schizophrenia vary with the clinical state in patients followed from the acute to stable the phase of the illness. Fifteen patients were assessed for symptoms (SAPS-SANS) and undergone a complete neuropsychological assessment at two sessions. The first session (S1) was carried out within six days after admission, i.e., acute phase and the second (S2) at least two month after hospitalization, i.e., stable phase. The data were analyzed using stepwise regression models in which neuropsychological scores were in entered to predict each dimensional score. This analysis was applied on the S1 and S2 data as well as on the S2-S1 difference to assess the neuropsychological predictors of clinical changes. Generally, the results replicate the previous associations between neuropsychological and dimensional measures found in stable patients. In addition, this study shows if each dimension appears to rely on a key structure, symptom variations seems to involve changes in the spread of the dysfunction and/or changes in the connectivity between the key and other regions. The results also suggest that functional changes related to some symptoms dimensions occur to compensate for the dysfunction associated with other symptoms.     

Development and validation of a brief version of the dyadic adjustment scale with a nonparametric item analysis model

The main purpose of the current research was to develop an abbreviated form of the Dyadic Adjustment Scale (DAS) with nonparametric item response theory. The authors conducted 5 studies, with a total participation of 8,256 married or cohabiting individuals. Results showed that the item characteristic curves behaved in a monotonically increasing function for all items of the brief 4-item version of the DAS (DAS-4). The DAS-4 proved to be informative at all levels of couple satisfaction. Compared with the 32-item version of the DAS (DAS-32), it was as effective in predicting couple dissolution and was significantly less contaminated by socially desirable responding. In addition, structural equation modeling demonstrated that the underlying latent construct measured by the DAS-4 was very stable over a 2-year period. This brief version has the advantage of being less time consuming and constitutes a promising alternative to the original DAS-32 for clinicians and researchers.     

Cytokine production by spleen cells after social defeat in mice: activation of T cells and reduced inhibition by glucocorticoids

Social disruption (SDR) is an effective model of social stress associated with an enhanced inflammatory reactivity of the immune system. The aim of the present study was to further describe SDR effects on cytokine production by spleen cells, testing selectively monocyte and T cell functions as a result of this stressor. For this purpose, splenocytes from control mice (C) and mice socially stressed for 7 days (SDR) were cultured in the presence of lipopolysaccharide (LPS) or concanavalin A (Con A). Splenocyte proliferation, cytokine production and sensitivity of spleen cells to corticosterone were assessed in vitro. The humoral response to keyhole limpet hemocyanin (KLH) immunization was assessed. SDR induced splenomegaly and enhanced splenocyte basal proliferation. The pro-inflammatory influence of SDR was confirmed by an increased release of interleukin-6 (IL-6) by LPS-stimulated cultures and by a reduced sensitivity of spleen cells to the anti-inflammatory effect of corticosterone. The mechanism increasing cytokine production in response to LPS was cytokine specific, since among inflammatory cytokines, IL-6 but not interferon-gamma (IFN-gamma) was enhanced by stress. In stressed mice, the increase in IL-6 and IFN-gamma and the decrease in IL-10 release in Con A-stimulated cultures indicate that SDR did not modify the Th1/Th2 cytokine balance but globally activated T cells. Plasma anti-KLH antibody levels were similar in both groups. Wounded and non-wounded mice presented similar responses to stress. This study shows that social disruption stress enhances the reactivity of cells from both the acquired and innate immune systems.     

Changing features do not guide attention in change detection: Evidence from a spatial cuing paradigm

Smilek, Eastwood, and Merikle (2000) demonstrated that the detection of change was facilitated when the target character changed in many rather than few of its features. Specifically, the function relating search response time to display set size was shallower when more features changed than when fewer features changed. The researchers interpreted these results as indicating that large feature changes provide preattentive guidance of focal attention to the location of the change. We tested this preattentive guidance hypothesis by examining change detection performance in the context of a spatial cuing paradigm. The hypothesis predicts that (1) the cost on invalidly cued trials should be less when more features change than when fewer features change, and (2) the features manipulation should have no effect on validly cued trials. In contrast to these predictions, our results show that cuing effects are equivalent across all levels of feature change and that a robust effect of the features manipulation is observable for both validly and invalidly cued trials. We argue that large feature changes do not provide preattentive guidance and in fact can be detected more readilyafter attention is already in place at the target location.     

Virtually no evidence for virtually perfect time-sharing

An examination of previous claims for virtually perfect time-sharing in dual-task situations reveals confounding effects that may have obscured dual-task interference. Two experiments are conducted in which these confounding effects are minimized, revealing statistically significant dual-task interference. These results support the hypothesis that human information processing is dominated by a structural central capacity limitation and call into question the hypothesis that dual-task interference can be eliminated by meeting the 5 conditions outlined by D. Meyer and D. Kieras (1999).     

Arguments for adjuncts

It is commonly assumed across the language sciences that some semantic participant information is lexically encoded in the representation of verbs and some is not. In this paper, we propose that semantic obligatoriness and verb class specificity are criteria which influence whether semantic information is lexically encoded. We present a comprehensive survey of the English verbal lexicon, a sentence continuation study, and an on-line sentence processing study which confirm that both factors play a role in the lexical encoding of participant information.     

A central capacity sharing model of dual-task performance

The authors present the central capacity sharing (CCS) model and derive equations describing its behaviors to explain results from dual-task situations. The predictions of the CCS model are contrasted with those of the central bottleneck model. The CCS model predicts all of the hallmark effects of the psychological refractory period (PRP) pardigm: -1 slope of the PRP effect at short stimulus onset asynchronies (SOAs), underadditivity of precentral Task 2 manipulations, additivity of central or postcentral Task 2 manipulations with SOA, and carry forward to Task 2 of Task 1 precentral or central manipulations at short SOAs. The CCS model also predicts that Task 1 response times increase with decreasing SOA. The model is a viable alternative to the central bottleneck model.