The doctrine of distribution

Peter Geach describes the ‘doctrine of distribution’ as the view that a term is distributed if it refers to everything that it denotes, and undistributed if it refers to only some of the things that it denotes. He argues that the notion, so explained, is incoherent. He claims that the doctrine of distribution originates from a degenerate use of the notion of ‘distributive supposition’ in medieval supposition theory sometime in the 16th century. This paper proposes instead that the doctrine of distribution occurs at least as early as the 12th century, and that it originates from a study of Aristotle's notion of a term's being ‘taken universally’, and not from the much later theory of distributive supposition. A detailed version of the doctrine found in the Port Royal Logic is articulated, and compared with a slightly different modern version. Finally, Geach's arguments for the incoherence of the doctrine are discussed and rejected.     

Developing arithmetic in set theory without infinity: some historical remarks

In this paper some of the history of the development of arithmetic in set theory is traced, particularly with reference to the problem of avoiding the assumption of an infinite set. Although the standard method of singling out a sequence of sets to be the natural numbers goes back to Zermelo, its development was more tortuous than is generally believed. We consider the development in the light of three desiderata for a solution and argue that they can probably not all be satisfied simultaneously.     

Reflections on <Emphasis Type="Italic">Mourning Religion</Emphasis>: A Response to the Respondents

This article is a response to the Mourning Religion review essays published in this forum. Specifically, it addresses the respondents’ concerns as they impact three related areas: the relationship between mourning and religion, mourning and melancholia, and mourning and the religious intellectual.     

Temporary inactivation of dorsal hippocampus attenuates explicitly nonspatial, unimodal, contextual fear conditioning

The current study examined the effects of temporary inactivation of the DH on freezing, rearing, ambulating, grooming, and whisking behavior in an explicitly nonspatial contextual fear conditioning paradigm in which olfactory stimuli served as temporally and spatially diffuse contexts. Prior either to training, testing, or both, male Sprague–Dawley rats received bilateral microinfusions of saline or the GABA_A agonist muscimol into the DH. Results indicate that temporary inactivation of DH produced both anterograde and retrograde deficits in contextually conditioned freezing, while sparing the acquisition and expression of freezing to a discrete auditory or olfactory CS. These data suggest that there is a decidedly nonspatial component to the role of DH in contextual conditioning, and that olfactory contextual conditioning is a fruitful means of further exploring this function.     

Why the handicapped child case is hard

This paper discusses the handicapped child case and some other variants of Derek Parfit's non-identityproblem (Parfit, 1984) The case is widely held to show that there is harmless wrongdoing, and that amoral system which tries to reduce wrongdoing directly to harm (``person-affecting morality')is inadequate.I show that the argument for this does not depend (as some have implied it does) on Kripkean necessity of origin. I distinguish the case from other variants (``wrongful life cases') of the non-identityproblem which do not bear directly on person-affecting morality as I understand it. And finally, I describe a respect in which the handicapped child case is puzzling and counter-intuitive, even on the supposition that it is a case of harmless wrongdoing. I conclude that the case is ``hard': it will take more than the rejection of person-affecting morality to remove its puzzling character.     

Social Exclusion and the Transition from School to Work: The Case of Young People Not in Education, Employment, or Training (NEET)

In the modern labor market what Côté (1996) describes as “identity capital”—comprising educational, social, and psychological resources—is at a premium in entering and maintaining employment. One consequence is the extension of education and training while young people acquire the qualifications and skills that will enhance their employability. In accordance with the perspective of life span developmental psychology, this places particular pressure on those young people growing up in disadvantaged circumstances and lacking support, especially when attempting to negotiate the transition from school to work. A particular policy concern in Britain has been directed at those young people who leave full-time education at the minimum age of 16 and then spend a substantial period not in education, employment, or training (NEET). This article reports the result of analyzing longitudinal data, collected for a subsample of the 1970 British Birth Cohort Study surveyed at age 21, to model the relationship of NEET status to earlier educational achievement and circumstances and to assess the added difficulties NEET poses in relation to the building of adult identity capital. It is concluded that although poor educational achievement is the major factor in entering NEET, inner city living for boys and lack of parental interest in their education for girls are also important. For young men the consequences of NEET lie mainly in subsequent poor labor market experience. For young women, the majority of whom are teenage mothers, the damaging effects of NEET extend to the psychological domain as well. It is concluded that effective counseling targeted at high risk groups, along the lines of the new UK “ConneXions” service, are needed to help young people avoid the damaging effects of NEET and make a successful transition to adult life.     

Scopolamine-induced deficits in spontaneous alternation performance: attenuation with lateral ventricle injections of glucose.

This experiment determined whether centrally administered glucose can attenuate scopolamine-induced deficits in spontaneous alternation performance. All rats were surgically prepared with indwelling cannulae directed at the lateral ventricle. Thirty min prior to alternation tests, rats received systemic (ip) injections of saline or scopolamine (3 mg/kg). Ten or thirty min prior to training, the rats also received a direct injection into the lateral ventricle of either artificial cerebrospinal fluid (CSF) or glucose (3 micrograms in 1 microliter). Scopolamine significantly impaired spontaneous alternation performance relative to controls. Additional treatment with ICV glucose 30 min, but not 10 min prior to testing, significantly attenuated the scopolamine-induced deficit. These results add support to the view that glucose acts directly on brain systems to attenuate behavioral effects of cholinergic antagonists.