Family Therapy in Trouble: Psychoeducation as Solution and as Problem

The authors cite clinical literature attesting to the importance of recognizing the family, rather than the individual, as the proper locus of conceptualizing, diagnosing, and treating mental illnesses. Specifically with regard to severe psychiatric illnesses, in particular schizophrenia, family dysfunction contributes to the emergence of the illness, significantly affects its course, and strongly influences the achievement and maintenance of treatment gains. Currently, a movement is afoot to limit sharply the amount and kind of treatment offered to schizophrenic patients and their families. Rooted in a “medical model” or “biogenic” view of the etiology of schizophrenia, this school of thought prescribes psychoeducation as the family treatment of choice. The present article looks at some misconceptions regarding treatment that prompted a widespread turning away from psychoanalytically oriented family psychotherapy for schizophrenic patients and their families, examines the reductionism (biological and behavioral) inhering in the exclusive use of psychoeducation, and looks at the clinical dangers of such reductionism. Finally, it proposes that family psycho-therapists should not abandon a concern with the inner lives of severely ill patients and their families in the face of spuriously generalized claims made by reductionist researchers.     

Phonological priming in the lexical decision task: A failure to replicate

Some models of the lexicon predict that recognition of words should produce activation spreading to phonologically related words. Consistent with this prediction, Hillinger (1980) demonstrated priming in a visual lexical decision task for word targets preceded by graphemically similar or graphemically dissimilar primes that rhymed with the target. In Experiment 1, we investigated whether this phonological priming effect occurred automatically or because of subject strategies. Although semantically associated primes produced significant facilitation in Experiment 1, no evidence of phonological priming was obtained. Experiments 2 to 5 were conducted in an attempt to obtain the phonological priming effect. Experiment 5 was a replication of one of Hillinger’s experiments. In none of these experiments was phonological priming observed. These results indicate either that the lexicon is not organized such that spreading activation occurs on the basis of phonological similarity, or that visual lexical decisions are made without phonological mediation.     

Frontal amnesia and the dysexecutive syndrome

This study analyzes the memory deficits shown by an amnesic patient with bilateral frontal damage and a dysexecutive syndrome. He resembles a classic amnesic patient in showing grossly impaired episodic memory for both verbal and nonverbal material, together with normal digit span, and on occasion normal recency in free recall. He differs from the classic amnesic pattern however in showing an impairment in both the speed and accuracy of performance on tests of semantic memory, and in clear evidence for impaired performance on some though not all procedural learning tasks. Finally, his autobiographical memory was poor and subject to substantial confabulation. It is suggested that the pattern of deficits is consistent with a combination of a classic amnesic syndrome with the additional problems associated with the frontal dysexecutive syndrome, rather than exhibiting a qualitatively different form of amnesia.