父母控制与儿童心理适应的关系：儿童自我控制的中介作用

以555名小学四到六年级儿童为被试,采用问卷调查法考察父母控制与儿童心理适应的关系,以及儿童自我控制在两者关系间的中介作用。结果发现：（1）六年级儿童的自我控制水平显著高于四年级,且情绪症状显著多于四、五年级;男生多动−注意缺陷水平显著高于女生;（2）父母行为控制对儿童积极心理适应具有显著正向预测作用,对儿童消极心理适应具有显著负向预测作用;而父母心理控制对儿童积极心理适应具有显著负向预测作用,对儿童消极心理适应具有显著正向预测作用;（3）儿童自我控制在父母控制（行为控制和心理控制）与儿童多动−注意缺陷和亲社会行为的关系间起显著中介作用,在父母控制与儿童情绪症状关系间中介作用不显著。     

道德外语效应的三种机制及与外语水平的关系

道德外语效应(Moral Foreign Language effect)是指人们的道德判断在外语条件下发生改变的现象,自该效应提出以来,其背后的机制一直都是研究的热点。道德外语效应的解释机制主要有审慎加工增强、情绪反应减少和道德规范联结的减弱等。本文对三种影响机制进行了探讨,发现情绪、道德规范和外语水平对道德判断可能会产生影响。同时,本文指出未来可以从自我控制、认知启动效应、消极情绪启动效应及道德规范与外语水平之间的关系进行研究和探讨。     

父母成人依恋、怜悯与亲子关系质量:主客体互倚分析

本研究考察了父母的成人依恋对亲子关系的影响以及怜悯的中介作用。采用方便取样法调查广东省某市小学2~5年级学生的父母。534对父母自我报告了各自的成人依恋、怜悯和亲子关系质量。主客体互倚模型分析发现：（1）父母各自的依恋焦虑和依恋回避显著负向预测自己与孩子的关系质量（主体效应）,但无法显著预测伴侣与孩子的关系质量（客体效应）;（2）父母各自的怜悯在上述主体效应中起到中介作用;（3）母亲的怜悯在自身依恋回避对父子关系的客体效应中起到“遮掩效应”;（4）母亲的怜悯在自身依恋焦虑对父子关系的客体效应中起到中介作用。研究结果有利于从成对关系视角理解父母成人依恋如何影响亲子关系,实际干预中可以通过提高父母的怜悯能力来改善亲子关系。     

散打运动员视觉信息加工的眼动研究*

散打比赛是一种对抗性很强的运动项目,要求运动员能够精确判断,及时地作出反应动作。这就需要运动员时刻保持高度的注意力,快速、准确地搜索相关信息,灵活地应对赛场上的情况。本研究以专家-新手范式对24名不同水平的散打运动员进行眼动实验研究,探讨高水平运动员与初学者之间的眼动差异,为初学者制定科学训练计划提供参考依据。结果表明：与新手组被试相比,专家组被试更加重视对比赛中得分部位的注视,从而为其准确预判提供了认知保证。     

不公平感及相关决策的认知神经机制

公平是人类社会生活的基本规范之一,不公平感及其相关决策则是研究者们关注的重要课题。长期以来,该领域的研究一般采用最后通牒博弈或其变式展开。大量脑成像研究探查了关于不公平感及相关决策的认知神经机制,尤其集中探讨了最后通牒博弈的回应者对不公平提议进行反应的脑区及其对应功能。经常得到关注的脑区包括了前脑岛、前扣带皮层、背外侧前额叶、内侧前额叶、杏仁核和颞顶交界等。对特殊人群的不公平感及相关决策进行研究可以帮助检验或澄清上述重要脑区及脑网络在不公平感及相关决策中扮演的角色,同时也阐释特殊人群的社会认知功能的特点。近年来,相当数量的研究关注了不同情境因素(包括分配方案相关因素和社会情境相关因素)调制不公平感及相关决策的过程,并讨论其背后的认知神经机制。未来的研究更应利用多模态数据分析方法,同时结合基因和激素层面的研究,以期深入对不公平感及相关决策的心理和生理机制的理解。     

书写对阅读的影响——来自传统书写与电脑打字的证据

书写和阅读是在文字的基础上发展起来的, 二者存在共享的神经网络。书写能影响阅读, 阅读不仅依靠视觉加工, 还依赖书写运动表征。不同书写方式对阅读的影响不同, 传统书写对阅读有促进作用, 电脑打字对阅读产生了一定的消极影响。对汉字来说, 传统书写能加强汉字的正字法表征, 促进汉字阅读; 电脑打字对汉字阅读的影响尚无一致结论。未来研究可以考察传统书写与电脑打字神经机制的差异, 以及电脑打字对汉字认知加工的影响及神经机制。     

The dimensionality of coping among Chinese health care workers

The transactional model defines coping as a process that changes on the basis of the context of an environmental encounter. An instrument used to investigate coping in diverse person-environment interactions is the Ways of Coping Questionnaire (WOC; S. Folkman & R. S. Lazarus, 1988). Although evidence exists to support the basic underlying structure of the WOC in Western societies, no research has been conducted on the instrument's dimensionality in non-Western societies. The authors identified 14 factors for the WOC administered to a sample of health care workers in Beijing, China. The 14 factors identified in the present study were similar to the 8 factors identified in the original validation study (S. Folkman, R. S. Lazarus, C. Dunkel-Schetter, A. DeLongis, & R. J. Gruen, 1986), but they were more content specific.     

Impairment of spatial learning and hippocampal synaptic potentiation in c-kit mutant rats

The c-kit receptor tyrosine kinase encoded by the white-spotting (W) gene is highly expressed in rat hippocampal CA1–CA4 regions. We found an impaired spatial learning and memory in homozygous c-kit (Ws/Ws) mutant rats that have a 12-base deletion in the tyrosine kinase domain of the c-kit gene and a very low kinase activity. Electrophysiological studies in hippocampal slices revealed that the long-term potentiation (LTP) induced by the tetanic stimulation (100 Hz, 1 sec) in the mossy fiber (MF)–CA3 pathway, but not in the Schaffer collaterals/commissural–CA1 pathway, was significantly reduced in c-kit mutants compared with wild-type (+/+) rats. The paired-pulse facilitation (PPF) was measured before the tetanus and after the establishment of the LTP in each slice. The initial PPF in the MF–CA3 pathway positively correlated with the amplitude of the LTP in the wild-type rats but not in the c-kit mutant rats. Furthermore, they failed to show the normal characteristics observed in the MF–CA3 pathway of +/+ rats; that is, the negative correlation between the initial PPF and the changes in PPF measured after the LTP. These findings suggest an involvement of SCF/c-kit signaling in hippocampal synaptic potentiation and spatial learning and memory.     

The differential effects of thalamus and basal ganglia on facial emotion recognition

This study examined if subcortical stroke was associated with impaired facial emotion recognition. Furthermore, the lateralization of the impairment and the differential profiles of facial emotion recognition deficits with localized thalamic or basal ganglia damage were also studied. Thirty-eight patients with subcortical strokes and 19 matched normal controls volunteered to participate. The participants were individually presented with morphed photographs of facial emotion expressions over multiple trials. They were requested to classify each of these morphed photographs according to Ekman's six basic emotion categories. The findings indicated that the clinical participants had impaired facial emotion recognition, though no clear lateralization pattern of impairment was observed. The patients with localized thalamic damage performed significantly worse in recognizing sadness than the controls. Longitudinal studies on patients with subcortical brain damage should be conducted to examine how cognitive reorganization post-stroke would affect emotion recognition.