Diversifying counsellor education: a case study of U.S. students of colour

Due to limited research on the experiences of students of colour in counselling programmes, counsellor educators are ill-equipped to implement strategies to promote the graduation of students of colour. In this case study, we explored the protective and risk factors for students of colour enrolled in a counselling programme in the Western U.S. Participants identified three protective factors (diversity, faculty support, and family and friends) and three risk factors (balancing family and school, disconnect from programme, and White dominance) that impacted their post-graduate experience. Implications for the retention of students of colour are discussed.     

Pilot Mindfulness Intervention for Children Born with Esophageal Atresia and Their Parents

A pilot mindfulness home intervention was conducted for 6 weeks among 8–12-year-old children born with esophageal atresia and their parents. Participants were randomly assigned to a waiting list control (WLC) group (n?=?8) and an experimental group (n?=?12). When all participants had completed the mindfulness-based program, data were pooled and treated for the entire sample (n?=?19). Self-assessment measures included the Mindful Attention Awareness Scale for Adolescents (MAAS-A), the Child and Adolescent Mindfulness Measure (CAMM), the modified Spielberger State-Trait Anxiety Inventory—Child (STAI-C), the Children’s Depression Inventory (CDI), and the Cognitive Emotion Regulation Questionnaire Kids version (CERQ-k). Parental assessment measures included the modified STAI-C. The results underlined the program’s feasibility and acceptability. Clinical effects of the mindfulness-based program were observed. Self-assessed data for children who had practiced mindfulness compared to the WLC group showed increased mindfulness and decreased depression. Reduced anxiety was found in all groups. Positive affect tended to improve from pre-test to post-test in children who had practiced mindfulness compared to the WLC group. Parental assessments showed significant improvement in positive affect and decreases in anxiety and negative affect in the intervention group compared to the WLC group. Cognitive emotion regulation strategies were also affected by the mindfulness-based program. Rumination scores significantly decreased from pre-test to post-test in the intervention group. This preliminary study suggests that regular mindfulness practice presents a promising approach to reducing the burden of this neonatal malformation.     

Saccadic eye movements: what do they tell us about aging cognition?

Although the relationship between age-related cognitive decline and saccadic eye movement (SEM) deficits has been outlined, specific cognitive alterations underlying age-related changes in saccadic performance remain unclear. This study attempted to better understand the nature of aging effects on SEMs. We compared SEMs in younger and older adults in prosaccade (PS) and antisaccade (AS) tasks under gap, step, and overlap conditions. We also examined relationships between these performances and several neuropsychological scores. Twenty-eight younger adults (YA), 24 older adults under 65 years (OA_<65) and 24 over 65 years (OA_>65) of age completed a neuropsychological evaluation, PS and AS tasks. Our results showed that latencies, AS cost, time to correct AS errors, and uncorrected AS, increased with aging. YA showed higher overlap effects than OA_>65 and OA_<65. Importantly, correlations and regressions revealed close relationships not only between latencies and processing speed measures but also between the AS cost and the inhibition process measures. Correct saccades and the time to correct AS errors were closely related to the inhibition process and cognitive flexibility measures. These findings suggest that the progressive age-related decline of processing speed and executive attention are associated with, and can be highlighted though SEMs in PS and AS tasks.     

A message in a bottle: Extrapharmacological effects of alcohol on aggression

The physiological and expectancy effects of alcohol consumption on the aggressive behavior of 116 general-population males aged 18-45 years were analyzed separately in a naturalistic field experiment using a 3 × 3 Balanced Placebo Design (BPD). Participants were given a non-alcoholic drink, a drink targeting a Blood Alcohol Concentration (BAC) of .05%, or a drink targeting a BAC of .1%. Within these three groups, three expectancy levels were induced regarding the quantity of alcohol ingested. Aggressive behavior was measured as retaliation against an aggressive confederate, in the form of amounts of hot sauce and salt administered in a taste test. Expectancies significantly increased aggressive behavior, whereas actual quantity of alcohol ingested was unrelated to aggression. Aggressive dispositions also predicted aggressive behavior.     

Are autistic traits autistic?

According to the extreme male brain theory of autism ( Baron‐Cohen, 2002 ), autistic traits would be extreme manifestations of typical male behaviours. The Auyeung et al. (2009) paper establishes a link between autistic traits and higher fetal testosterone (fT) levels in typically developing children. We argue that the construct behind this relationship needs further investigation. First, the link between fT levels and sexually dimorphic traits, that are for example, associated with empathizing and systemizing, is controversial. Likewise, describing autistic behaviours as being extreme male‐like is debatable. The cerebral hemisphere laterality pattern of individuals with autism also seems to differ from the pattern typically observed in males. Moreover, the parallel that should exist, according to the fT theory, between individuals with autism and individuals with congenital adrenal hyperplasia (CAH), because of their high fT levels, is unclear. The theory implying fT levels in autism fails to account for a big part of autism, and the link between fT and normal ‘autistic traits’ hardly demonstrates the causal link between fT and autism.     

Enhancing Justice?

This article focuses on the follow question: Are human enhancement technologies likely to be justice impairing or justice promoting? We argue that human enhancement technologies may not be inherently just or unjust, but when situated within obtaining social contexts they are likely to exacerbate rather than alleviate social injustices.     

Family-supportive organization perceptions, multiple dimensions of work-family conflict, and employee satisfaction: A test of model across five samples

Using samples of managers drawn from five Western countries, we tested a theoretical model linking employees’ perceptions of their work environment’s family-supportiveness to six different dimensions of work-family conflict (WFC), and to their job satisfaction, family satisfaction, and life satisfaction. Our results are consistent with a causal process whereby employees working in an environment viewed as more family-supportive experience lower levels of WFC. Reduced WFC then translates into greater job and family satisfaction, followed by greater overall life satisfaction. These findings were generalizable across the five samples.     

Distinct contributions of the basolateral amygdala and the medial prefrontal cortex to learning and relearning extinction of context conditioned fear

We studied the roles of the basolateral amygdala (BLA) and the medial prefrontal cortex (mPFC) in learning and relearning to inhibit context conditioned fear (freezing) in extinction. In Experiment 1, pre-extinction BLA infusion of the NMDA receptor (NMDAr) antagonist, ifenprodil, impaired the development and retention of inhibition but post-extinction infusion spared retention. Pre-extinction infusion of the GABA(A) agonist, muscimol, depressed freezing and impaired retention as did post-extinction infusion. In Experiment 2, pre-extinction mPFC infusion of ifenprodil spared the development of inhibition whereas muscimol depressed freezing. Both impaired retention when infused pre- or post-extinction. Thus, the development of inhibition involves NMDAr activation in the BLA, whereas its consolidation involves both NMDAr activation in the mPFC and NMDAr-independent mechanisms in the BLA. In Experiment 3, BLA infusion of ifenprodil impaired relearning and retention of inhibition when infused before but did not impair retention when infused after re-extinction. BLA infusion of muscimol depressed freezing but did not impair retention when infused before or after re-extinction. In Experiment 4, mPFC infusion of ifenprodil impaired relearning when infused before re-extinction, whereas muscimol depressed responses. Both drugs impaired retention when infused into the mPFC before or after re-extinction. Thus, relearning to inhibit fear responses involves NMDAr activation in both the BLA and mPFC and consolidation of the inhibitory memory involves NMDAr activation in the mPFC. However, relearning and consolidation occur in the absence of neuronal activity within the BLA. We propose that NMDAr in the mPFC supports relearning inhibition when the BLA is inactivated.     

Hippocampal low-frequency stimulation and chronic mild stress similarly disrupt fear extinction memory in rats

Disruptions of fear extinction-related potentiation of synaptic efficacy in the connection between the hippocampus (HPC) and the medial prefrontal cortex (mPFC) have been shown to impair the recall of extinction memory. This study was undertaken to examine if chronic mild stress (CMS), which is known to alter induction of HPC–mPFC long-term potentiation, would also interfere with both extinction-related HPC–mPFC potentiation and extinction memory. Following fear conditioning (5 tone-shock pairings), rats were submitted to fear extinction (20 tone-alone presentations), which produced an increase in the amplitude of HPC–mPFC field potentials. HPC low-frequency stimulation (LFS), applied immediately after training, suppressed these changes and induced fear return during the retention test (5 tone-alone presentations). CMS, delivered before fear conditioning, did not interfere with fear extinction but blocked the development of extinction-related potentiation in the HPC–mPFC pathway and impaired the recall of extinction. These findings suggest that HPC LFS may provoke metaplastic changes in HPC outputs that may mimic alterations associated with a history of chronic stress.