Pretend play and development in early childhood (with implications for the oedipal phase)

Pretend play reflects both the unique mental organization and the developmental challenges of early to middle childhood, with a trajectory that parallels the monumental transformation of the toddler to the school-age child. Despite evidence for various forms of playing throughout the life cycle, the flowering of symbolic play in this phase is specific, essential, and typically transient in terms of its dominance in the life of the child. It reflects the simultaneous emergence, processing, and integration of the remarkable developmental advances occurring during this period, most especially the capacity to symbolize, in the service of the exponentially expanded psychosexual/social/emotional force field implied in the contemporary use of the term oedipus complex. Moreover, it constitutes a particular mental organization and ego state, with idiosyncratic mentation, affect regulation, and relationship to inner and outer experience, that accompanies the child's gradual orientation to consensual reality. Subsequent access to some form of this state varies widely among individuals but is rarely fully comparable.     

Constructive or cruel? Positive or patronizing? Reactions to expressions of positive and negative stereotypes of the mentally ill

Previous research has shown that people respond with greater sensitivity to negative stereotypical comments about a group that are made from someone outside the group in question than from someone who belongs to the group. In this paper, we investigated if the same effect occurs in response to comments made about stigmatized groups. Specifically, we examined how people react to comments made about the mentally ill. The conditions under which people accept or reject stereotypes of the mentally ill may shed light on the conditions necessary for effective anti-discrimination campaigns. In the current study, participants responded to positive or negative stereotypes of the mentally ill voiced by either someone who has, or has not, suffered from a mental illness. Participants were more sensitive, agreed less, and evaluated the speaker less favourably when comments came from the out-group rather than the in-group source. The effects were strongest for negative comments, however contrary to previous research participants also responded less favourably to positive comments from the out-group source. These reactions were mediated by the perceived constructiveness of the speaker's motives. Implications for the effectiveness of anti-discrimination campaigns are discussed.     

The effect of motion at encoding and retrieval for same- and other-race face recognition

In an experimental study, we assessed the role of motion when encoding and recognizing unfamiliar faces, using an old/new recognition memory paradigm. Our findings revealed a clear advantage for learning unfamiliar faces moving non-rigidly, compared with static faces. This advantage for motion was found with both same- and other-race faces. Furthermore, results indicate that it is more important that the face is learnt in motion than recognized from a moving clip. Interestingly, despite a reliable other-race effect being revealed, participants were able to utilize motion information exhibited by other-race faces in a manner akin to the motion advantage found for same-race faces. The implications of these findings are discussed in relation to the nature of the stored face representations, considering whether the facilitative role found here can be explained by factors other than motion per se.     

Trajectories of cognitive development among American Indian young children

Children who begin kindergarten with stronger skills learn faster than do those who enter with lower skills. Minority children tend to enter kindergarten already at a disadvantage, and the gap widens across time. However, little is known about cognitive development among American Indian young children. In this study, 110 American Indian infants from one Northern Plains reservation community were assessed four times between ages 6 months and 36 months, with the Mullen Scales of Early Learning. At 6 months of age, scores were near the national norms; a drop occurred between 6 months and 15 months. Scores then tended to level off below the norms through 36 months. In each domain, we observed a crucial decline over the 1st year of life and relatively little change in the 2nd and 3rd years of life, highlighting the importance of developing culturally syntonic interventions to facilitate cognitive development during the 1st year of life.     

Maternal influences on youth responses to peer stress

Understanding how youths develop particular styles of responding to stress is critical for promoting effective coping. This research examined the prospective, interactive contribution of maternal socialization of coping and peer stress to youth responses to peer stress. A sample of 144 early adolescents (mean age = 12.44 years, SD = 1.22) and their maternal caregivers completed questionnaires and semistructured interviews in 2 waves over a 1-year period. Results revealed that mothers' disengagement coping suggestions predicted maladaptive responses to stress, particularly for youths who received low levels of engagement suggestions, and engagement coping suggestions protected youths against maladaptive responses to stress. Importantly, these effects emerged only in the context of heightened peer stress. This research suggests that maternal socialization of coping has the potential to support or undermine youths' development of an effective repertoire of responses to stress.     

Consideration of the BDNF gene in relation to two phenotypes: hoarding and obesity

The gene coding for the brain derived neurotrophic factor (BDNF) has emerged as an interesting candidate for multiple brain and brain disorder-related phenomena. The primary aim of the present investigation was to consider the relationship between the BDNF Val66Met variant and two phenotypes: compulsive hoarding as a symptom dimension of obsessive-compulsive disorder (OCD), and body mass index (BMI). We examined the BDNF gene in a large (N=301) clinical sample of probands with OCD. Participants were classified as hoarding or nonhoarding using a strict, multimeasure grouping approach. Results revealed that the Val/Val genotype was linked with hoarding classification and more severe hoarding behaviors, as well as greater BMI levels. Hoarding status was also associated with greater BMI scores, with individuals in the hoarding group being far more likely to be classified as obese compared with the nonhoarding group. Our findings may provide a distinct avenue through which hoarding and BMI could be linked. These findings are suggestive of a complex gene, body weight, and psychopathology relationship wherein a primitive, survival "thrifty gene" strategy may be conserved and represented in a subgroup of humans manifesting severe hoarding symptoms.     

Challenges and resilience in the lives of urban, multiracial adults: an instrument development study

Multiracial Americans represent a rapidly growing population (Shih & Sanchez, 2009); however, very little is known about the types of challenges and resilience experienced by these individuals. To date, few psychological measures have been created specifically to investigate the experiences of multiracial people. This article describes 2 studies focused on the development and psychometric properties of the Multiracial Challenges and Resilience Scale (MCRS). The MCRS was developed using a nationwide Internet sample of urban, multiracial adults. Exploratory factor analyses revealed 4 Challenge factors (Others' Surprise and Disbelief Regarding Racial Heritage, Lack of Family Acceptance, Multiracial Discrimination, and Challenges With Racial Identity) and 2 Resilience factors (Appreciation of Human Differences and Multiracial Pride). A confirmatory factor analysis with data from a second sample provided support for the stability of this factor structure. The reliability and validity of the measure, implications of these findings, and suggestions for future research are discussed.     

Psychological stress in childhood and susceptibility to the chronic diseases of aging: moving toward a model of behavioral and biological mechanisms

Among people exposed to major psychological stressors in early life, there are elevated rates of morbidity and mortality from chronic diseases of aging. The most compelling data come from studies of children raised in poverty or maltreated by their parents, who show heightened vulnerability to vascular disease, autoimmune disorders, and premature mortality. These findings raise challenging theoretical questions. How does childhood stress get under the skin, at the molecular level, to affect risk for later diseases? And how does it incubate there, giving rise to diseases several decades later? Here we present a biological embedding model, which attempts to address these questions by synthesizing knowledge across several behavioral and biomedical literatures. This model maintains that childhood stress gets "programmed" into macrophages through epigenetic markings, posttranslational modifications, and tissue remodeling. As a consequence these cells are endowed with proinflammatory tendencies, manifest in exaggerated cytokine responses to challenge and decreased sensitivity to inhibitory hormonal signals. The model goes on to propose that over the life course, these proinflammatory tendencies are exacerbated by behavioral proclivities and hormonal dysregulation, themselves the products of exposure to early stress. Behaviorally, the model posits that childhood stress gives rise to excessive threat vigilance, mistrust of others, poor social relationships, impaired self-regulation, and unhealthy lifestyle choices. Hormonally, early stress confers altered patterns of endocrine and autonomic discharge. This milieu amplifies the proinflammatory environment already instantiated by macrophages. Acting in concert with other exposures and genetic liabilities, the resulting inflammation drives forward pathogenic mechanisms that ultimately foster chronic disease.