高职生因果定向与学业表现的关系

为了考察高职生因果定向的特点及因果定向与学生学业表现(学业成绩和专业技能)的关系。该研究以修订的一般因果定向量表为工具,在济南和淄博的三所高职抽取505名学生作为被试进行调查。结果发现:1)修订后的一般因果定向量表具有良好的信效度;2)高职生在因果定向的三个维度上得分差异显著,由高到低依次为自主定向、控制定向和非个人定向;3)自主定向显著正向预测学业表现,控制定向显著负向预测学业表现,而非个人定向对学业表现的预测作用不显著。我国高职生与国外被试的因果定向特点基本一致,说明因果定向具有一定的文化普适性。同时,不同因果定向对学业表现具有不同的预测作用,教育者有必要对学生进行因果定向的教育和引导。     

上行比较影响行为表现—不同类型高自尊的作用

已有研究表明,上行比较对高自尊个体产生积极效应。然而鉴于高自尊包括安全性高自尊和防御性高自尊,有必要在上行比较影响不同任务领域内的行为表现这一观点的基础上探讨高自尊的不同类型对上行比较与行为表现之间关系的影响。两个实验均采用高自尊类型、上行比较和任务领域一致性的2×2×2混合因素设计,不同之处在于其分别采用创造性测验和非创造性的语词测验作为不同领域任务。结果表明,防御性高自尊个体没有像安全性高自尊个体那样采取领域转换策略来修复自我关注,由上行比较所引起的领域转换只对安全性高自尊个体产生积极影响。     

幼儿提问类型及策略对其问题解决的影响

该研究以69名5岁幼儿为被试,通过创设猜测物体的问题情境,以考察幼儿提问对其解决猜测物体的问题的影响.结果表明:(1)面对问题情境,幼儿运用提问解决问题的正确率显著高于不提问直接猜测的正确率.(2)幼儿运用两阶段整体策略和逐一排除策略提问来帮助其问题解决的效果好于直接猜测的效果.(3)幼儿提出混合类问题和外在感知类问题来帮助其解决猜测物体问题的效果好于提出内在属性类问题.     

不确定性决策违背“确定事件原则”的心理机制再探

不确定性决策违背＂确定事件原则＂的心理机制再探从决策理由切入,在现实情境中考察了违背确定事件原则（sure-thing principle）的心理机制,检验了基于理由（reason-based）的假设。研究结果表明,违背确定事件原则的被试在不确定条件下知觉到的理由冲突程度显著高于遵循确定事件原则的被试在不确定条件下知觉到的理由冲突程度;违背确定事件原则的被试在不确定条件下知觉到的理由清晰程度显著低于其在两种确定条件下知觉到的理由清晰程度,而遵循确定事件原则的被试在不确定条件下知觉到的理由清晰程度则介于其在两种确定条件下知觉到的理由清晰程度之间。研究结果支持了基于理由的假设。     

左右半脸及其位置对面孔吸引力的影响

本研究将拼嵌技术（chimera technique）和融合技术（Morph tech—nique）结合起来,制作新形式的面孔刺激,将“刺激”和“位置”分离并分别操纵,检验了左右半脸及其位置对面孔吸引力的影响。实验结果显示：（1）“刺激”和“位置”都会影响面孔吸引力;（2）“刺激”的影响是直接的,具体表现为右半脸不论呈现在面孔的左侧还是右侧,其吸引力总是高于左半脸的吸引力;（3）“位置”的影响模式较为复杂,具体表现为左右半脸在原始位置呈现时（右丰脸呈现在右侧,左半脸呈现在左侧）,其吸引力总是高于镜像位置。     

Influence of brain-derived neurotrophic factor and catechol O-methyl transferase polymorphisms on effects of meditation on plasma catecholamines and stress

Meditation may show differential effects on stress and plasma catecholamines based on genetic polymorphisms in brain-derived neurotrophic factor (BDNF) and catechol O-methyl transferase (COMT). Eighty adults (40 men, 40 women; mean age 26 years) who practiced meditation regularly and 57 healthy control adults (35 men, 22 women; mean age 26 years) participated. Plasma catecholamines (norepinephrine (NE), epinephrine (E), and dopamine (DA)) concentrations were measured, and a modified form of the Stress Response Inventory was administered. The results were analyzed using two-way analysis of covariance (ANCOVA) with control and meditation subjects, gene polymorphism as factors, and meditation duration as the covariate. Two-way ANCOVA showed a significant interaction between control and meditation subjects, and BDNF Val66Met polymorphism on DA/NE+DA/E (p = 0.042) and NE/E+NE/DA (p = 0.046) ratios. A significant interaction was found for control and meditation subjects with COMT Val158Met polymorphism and plasma NE concentrations (p = 0.009). Post hoc ANCOVA in the meditation group, adjusted for meditation duration, showed significantly higher plasma NE concentrations for COMT Met carriers than COMT Val/Val subjects (p = 0.025). Significant differences of stress levels were found between the control and meditation subjects in BDNF Val/Met (p < 0.001) and BDNF Met/Met (p = 0.003), whereas stress levels in the BDNF Val/Val genotype did not differ between the control and meditation groups. This is the first evidence that meditation produces different effects on plasma catecholamines according to BDNF or COMT polymorphisms.     

Stress induces glucocorticoid-mediated apoptosis of rat Leydig cells in vivo

Stress can disrupt endocrine signalling in the male reproductive axis through high concentrations of glucocorticoids, the hallmark of stress. Our previous work revealed that a stress level of exogenous glucocorticoids could induce apoptosis of rat Leydig cells, which are the primary source of testosterone. The aim of this study was to investigate whether stress can induce apoptosis in rat Leydig cells in vivo and, if so, whether the process is the result of a direct effect of glucocorticoids. In a chronically stressed rat model, serum corticosterone concentration was increased significantly whereas serum testosterone was decreased. The frequency of apoptotic Leydig cells in stressed rats was also increased. Adrenalectomised rats subjected to chronic stress showed an elevated serum testosterone, while the apoptotic frequency of Leydig cells was not increased. It was established that glucocorticoid-induced Leydig cell apoptosis is mediated by glucocorticoid receptors (GRs), which translocate from cytoplasm to nucleus. Adenovirus microRNA-induced downregulation of GR expression in vitro alleviated the corticosterone-induced increase in apoptosis of Leydig cells. These results indicate that the stress-induced increase in corticosterone secretion resulted in apoptosis in rat Leydig cells in vivo, and thereby decreased testosterone synthesis.     

Dopamine D1 receptors are responsible for stress-induced emotional memory deficit in mice

It is established that stress impairs spatial learning and memory via the hypothalamus-pituitary-adrenal axis response. Dopamine D1 receptors were also shown to be responsible for a stress-induced deficit of working memory. However, whether stress affects the subsequent emotional learning and memory is not elucidated yet. Here, we employed the well-established one-trial step-through task to study the effect of an acute psychological stress (induced by tail hanging for 5, 10, or 20?min) on emotional learning and memory, and the possible mechanisms as well. We demonstrated that tail hanging induced an obvious stress response. Either an acute tail-hanging stress or a single dose of intraperitoneally injected dopamine D1 receptor antagonist (SCH23390) significantly decreased the step-through latency in the one-trial step-through task. However, SCH23390 prevented the acute tail-hanging stress-induced decrease in the step-through latency. In addition, the effects of tail-hanging stress and/or SCH23390 on the changes in step-through latency were not through non-memory factors such as nociceptive perception and motor function. Our data indicate that the hyperactivation of dopamine D1 receptors mediated the stress-induced deficit of emotional learning and memory. This study may have clinical significance given that psychological stress is considered to play a role in susceptibility to some mental diseases such as depression and post-traumatic stress disorder.     

Theories of spatial representations and reference frames: What can configuration errors tell us?

The issue of reference frame is central to theories of spatial representations. Various classifications have been made for different types of reference frames, along with prototypical research paradigms to distinguish between them. This article focuses on the configuration error paradigm proposed by Wang and Spelke (Cognition 77:215-250, 2000) that has been used to examine the nature of the spatial representations underlying object localization during self-movement. Three basic models of spatial memory and spatial updating are discussed, as well as the assumptions behind the configuration error paradigm, to distinguish between static representations, such as the traditional allocentric cognitive map and the egocentric snapshots, and dynamic representations, such as the egocentric updating system. Recent experimental findings are reexamined and shown to be consistent with multiple models, among which the egocentric-updating-and-reload model with an enduring egocentric component provides the simplest interpretations.