A longitudinal comparison of couples with sex-typical and non-sex-typical orientations to intimacy

The present study was designed to investigate the relationship experience of males and females who do not conform to sex-typical orientations to intimacy. One hundred sixty-three couples' and 88 nondating singles' (total N=414) responses to the Relationship World Index—Version 2 (RWI-2) were analyzed via discriminant analysis to determine if subjects' sex could be predicted accurately upon the basis of their RWI-2 scores. One discriminant function was found which was significant by the Wilks' lambda test [x²(60)=140.57, p<.001]; the procedure correctly classified 76% of the respondents. Those who were classified correctly were considered sex-typical and the remaining 24% of the sample was considered non-sex-typical in their orientations to intimacy. Additional analyses using data from a 6-month longitudinal study located qualities which significantly distinguished sex-typical from non-sex-typical subjects and couples in which at least one member was non-sex-typical from couples in which both members were classified as sex-typical.     

Reasons black african adults use to explain their acceptance of sex-role reversals

A structured questionnaire based on Rest's Defining Issues Test format was adopted to investigate the reasons used by 50 Black African males and 50 Black African females to explain their degree of acceptance of sex-role reversals. Both males and females showed little acceptance of sex-role reversals. The organization of the reasons they used to explain their degree of acceptance of sex-role reversals was most parsimoniously explained by Rest's complex stage model rather than the model suggested by Ullian, which is based on Piagetian stage criteria.     

Aggressive behavior inhibition by serotonin and quipazine injected into the amygdala in the rat

The effects on aggressive behavior, open-field activity, and pain threshold of bilateral microinjections of serotonin (20 micrograms) and quipazine (20 micrograms), the direct serotonergic receptor agonist, into the cortico-medial amygdala were investigated in Wistar rats. Both drugs significantly prolonged the attack latency in isolated killer rats (predatory aggression model), and suppressed the incidence of aggressive postures/attacks in shock-induced fighting test (affective aggression). The only difference in the open-field behavior was the lower number of central square entries in drug-treated compared to saline-injected rats. None of the substances produced any significant change in jump threshold. It is concluded that stimulation of serotonin receptors within the amygdala produces inhibition of affective and muricidal behavior in isolated rats. The effect does not seem to be dependent on changes in general activity and pain sensitivity.     

Positive and negative motivational gradients during first use of the dissecting room

Immediately after their first use of the dissecting room, 45 preclinical medical students rated their enthusiasm and reluctance at seven points along a time sequence encompassing their experience from the night before to the completion of their first dissection. A second group of 42 preclinical medical students carried out the same ratings in real time. A naive group of 24 psychology students were asked to imagine a first visit to the dissecting room and rate their imagined feelings in the same way. Gradients for reluctance for the two experimental groups rose to maxima at the point of entering the dissecting room. The reluctance gradient for the naive group and the enthusiasm gradients for all three groups, prior to this point, were zero. These are discussed as negative and positive motivational gradients that meet the predictions of approach/avoidance theory (Miller, 1959).     

The inequivalence of self-reports of Type A behavior: Differential relationships of the Jenkins Activity Survey and the Framingham cale with affect,stress, and control

The Jenkins Activity Survey (JAS) and the Framingham Type A Scale (FTAS) have been found to have different psychological correlates. The JAS is typically unrelated to anxiety, while the FTAS is consistently correlated with trait anxiety. The present study of 200 male and female college students clarifies further the psychological heterogeneity of these measures. Enhanced motivation to control the environment has been suggested as an underlying psychological component of Type A behavior (Glass, 1977). Dysphoric emotions and the perception of more environmental stresses, on the other hand, are typical of neuroticism or negative affectivity (Watson & Clark, 1984). Although the results indicate that the JAS and FTAS are highly correlated, the JAS is more closely related to control motivation than is the FTAS. Further, compared to the JAS, the FTAS is more closely correlated with dysphoric emotions and perceived daily stresses. Thus, the results indicate that the JAS is closely correlated with core psychological components of the Type A pattern, while the FTAS has a relatively unique association with general emotional distress. Such differences may account for the fact that these measures are related to different disease end points.     

The cognitive model of depression and mood-induction procedures: A reply to Clark (1983)

In a recent article on mood-induction procedures, Clark (1983) concluded that the Velten Mood Induction Procedure (VMIP; Velten, 1968) produces a good analog state of mild, naturally occurring, retarded depression that is not explicable in terms of experimental demand characteristics. Clark concluded, however, that it is erroneous to view the relative success of the VMIP in simulating the effects of depression as support for cognitive theories of depression. While we agree with the first of Clark's conclusions, we disagree with his second conclusion that the VMIP is not a cognitive manipulation. The purpose of the present article is to argue that his ideas regarding the impact of the VMIP on mood and behavior are not supported by the existing data. Therefore, the VMIP results can be used to support Beck's (1976) cognitive theory of depression.