Tactics of manipulation

Manipulation is one means by which environments are altered to correspond to characteristics of individuals. We conducted two studies to identify the manipulation tactics that people use to elicit and terminate the actions of others. Factor analyses of four instruments revealed six types of tactics: charm, silent treatment, coercion, reason, regression, and debasement. Tactics of manipulation showed strong individual difference consistency across contexts. The charm tactic, however, was used more frequently for behavioral elicitation, whereas the coercion and silent treatment tactics were used more frequently for behavioral termination. Manipulation tactics covaried significantly across self-based and observer-based data sources with personality scales of Neuroticism, Extraversion, Ambitious-Lazy, Arrogant-Unassuming, Quarrelsome-Agreeable, and Calculating and with characteristics of subjects' social environments. We draw implications for an interactionist framework of person-environment correspondence, for an expansion of the taxonomic task that faces personality psychology, and for identifying links between personality and other scientific disciplines.     

On the relevant systemsP andP * and some related systems

In this paper we study the systemsP andP ^* (see Arruda and da Costa,O paradoxo de Curry-Moh Shaw-Kwei, Boletim da Sociedade Matemtica de São Paulo 18 (1966)) and some related systems. In the last section, we prove that certain set theories havingP andP ^* as their underlying logics are non-trivial.     

The spontaneous decay of compulsive urges

An experiment was carried out on eleven obsessional patients who complained of compulsive checking rituals. Exposure to a provoking situation produced marked anxiety/discomfort and strong urges to check. With minor exceptions, execution of the appropriate ritual was followed by marked decreases in anxiety/discomfort and urges. The results were interpreted as being consistent with an anxiety/discomfort reduction hypothesis.

The spontaneous decay of anxiety/discomfort and urges was observed during a 3-hr response-prevention period. The greatest part of the discomfort and compulsive urges dissipated within an hour. Discomfort scores and urges ran a closely parallel course. Few resurgences of discomfort or urges were observed during the 3-hr observation period, and there was little sign of displacement checking activity.     

Observation of behavior,inference of function,and the study of learning

Before the present century, the primary means of studying animals was observation of the form and effects of their behavior combined with presumption of their intent. In the present century, ethologists continued to emphasize observation of form and replaced presumption of intent with the study of proximate function and evolution. In contrast, most learning psychologists minimized both observation of form and presumption of intent by defining behavior in terms of simple environmental effects and establishing intent by deprivation operations. We discuss advantages of the use of observation in the study of learning, examine arguments that it is unnecessary, irrelevant, and unscientific, and consider some practical considerations in using observation. We conclude that observation of the form of behavior and concern with its ecological function should be an important part of the arsenal of techniques used to study learning.     

Medicine: The first step on the road to holiness?

The connections of Medicine and Religion are thoroughly documented in the primitive societies, and it comes as no surprise to see them together. Here we describe the true story of a Portuguese physician of the late 19th century whose image today is venerated in such a way as to compare him to a saint and to bestow upon him miraculous powers of healing. We propose that modern day physicians can learn from the example of this man that the role of doctor encompasses not only scientific and technical mastery but, most of all, piety, humanity and compassion.     

Reduced K+ Channel Inactivation, Spike Broadening, and After-Hyperpolarization in Kvβ1.1-Deficient Mice with Impaired Learning

A-type K⁺ channels are known to regulate neuronal firing, but their role in repetitive firing and learning in mammals is not well characterized. To determine the contribution of the auxiliary K⁺ channel subunit Kvβ1.1 to A-type K⁺ currents and to study the physiological role of A-type K⁺ channels in repetitive firing and learning, we deleted the Kvβ1.1 gene in mice. The loss of Kvβ1.1 resulted in a reduced K⁺ current inactivation in hippocampal CA1 pyramidal neurons. Furthermore, in the mutant neurons, frequency-dependent spike broadening and the slow afterhyperpolarization (sAHP) were reduced. This suggests that Kvβ1.1-dependent A-type K⁺ channels contribute to frequency-dependent spike broadening and may regulate the sAHP by controlling Ca²⁺ influx during action potentials. The Kvβ1.1-deficient mice showed normal synaptic plasticity but were impaired in the learning of a water maze test and in the social transmission of food preference task, indicating that the Kvβ1.1 subunit contributes to certain types of learning and memory.     

Personality correlates of dissociative experiences

This study investigated the prevalence of dissociative experiences in a normal British sample. The Dissociation Experiences Scale of Bernstein and Putnam was used to measure these experiences. The relationship between dissociative experiences and personality variables measured by the Eysenck Personality Questionnaire was a major focus of the study. It was found that the degree of dissociative experiences was related to neuroticism, but not to any other variable.     

Neurotransmitter receptors involved in post-training memory processing by the amygdala, medial septum, and hippocampus of the rat.

Rats were trained and tested in habituation to a novel environment and step-down inhibitory avoidance. Immediately after training in each task the animals received intra-amygdala, intraseptal, or intrahippocampal micro-injections of agonists and antagonists of various neurotransmitter receptors. In the habitation task, intrahippocampal, but not intra-amygdala or intraseptal administration of the NMDA receptor antagonist aminophosphornopentanoic acid (AP5, 5.0 micrograms) or of the muscarinic receptor antagonist, scopolamine (2.0 micrograms) caused amnesia and the indirect antagonist of GABA-A receptors, picrotoxin (0.08 microgram), caused retrograde facilitation. Intrahippocampal administration of the respective agonists, glutamate, oxotremorine, and muscimol, had effects of their own opposite to those of the blockers, and norepinephrine (0.3 microgram) caused memory facilitation. In the avoidance task, results obtained with drug infusions given into the three structures were very similar: in all cases, AP5, scopolamine, and muscimol were amnestic, and glutamate, oxotremorine, norepinephrine, and picrotoxin caused memory facilitation. In addition, also in the three structures, picrotoxin counteracted the amnestic effect of AP5 and/or scopolamine and the beta-adrenoceptor blocker, timolol (0.3 microgram), while ineffective on its own, attenuated all the effects of picrotoxin. The results suggest that similar synaptic mechanisms in the amygdala, medial septum, and hippocampus are involved in memory consolidation: NMDA, muscarinic, and beta-noradrenergic receptors stimulate and GABA-A receptors inhibit this process, and beta-noradrenergic receptors modulate the GABAergic synapses. In the avoidance task these mechanisms operate in the three structures: in habituation only those in the hippocampus are operative. Possibly in each structure these mechanisms regulate, if not actually consolidate, a different aspect, component, or form of memory.