The Sources and Exercise of Authority and the Limits of Sustainable Diversity

This article seeks to address the following two questions: a) What are the sources of authority within your church, and how is authority exercised? b) In your church's understanding, what are the bases for maintaining union and what are the limits of sustainable diversity?     

Logical Connectives for Constructive Modal Logic

Model-theoretic proofs of functional completenes along the lines of [McCullough 1971, Journal of Symbolic Logic 36, 15–20] are given for various constructive modal propositional logics with strong negation.     

《结构存在论》引论

罗姆巴赫的结构现象学思想与东方哲学有着绝妙的契合。在其核心著作《结构存在论》一开篇,罗姆巴赫就明确地通过对老子哲学的阐释来说明他的“结构”概念。无论在艺术,伦理学和宗教信仰还是在科学活动中,结构的构造状态都被视为普全的基本形式;而这种向分支领域扩展的基础在于对现象学视域内存在论的结构分析。同时,他也强调结构只能在活的历史关联中被描述,不存在最终有效的静态结构范畴。     

现象学之道

罗姆巴赫完全赞同海德格尔将现象学方法带入存在论层面的尝试,但同时他力图克服他所理解的海氏缘在分析的狭隘性。在他的结构现象学中,缘在的结构分析必须被置于世界整体的结构生成中才可把握;而这种作为本己真实性的大结构的发生,不仅超越人的缘在也超越自然形式———相应地,对大结构的认识就只有通过密释学才有可能。由此现象学在罗姆巴赫那里“脱离了那种基于人的存在之上的固定”。经此深入,他提出了“深层结构的现象学”,深层现象乃是发生的境遇实践,从这里他迈出了东西方关于缘在体验的共同哲学的第一步。     

Working memory in wayfinding-a dual task experiment in a virtual city

This study examines the working memory systems involved in human wayfinding. In the learning phase, 24 participants learned two routes in a novel photorealistic virtual environment displayed on a 220° screen while they were disrupted by a visual, a spatial, a verbal, or—in a control group—no secondary task. In the following wayfinding phase, the participants had to find and to "virtually walk" the two routes again. During this wayfinding phase, a number of dependent measures were recorded. This research shows that encoding wayfinding knowledge interfered with the verbal and with the spatial secondary task. These interferences were even stronger than the interference of wayfinding knowledge with the visual secondary task. These findings are consistent with a dual-coding approach of wayfinding knowledge.     

Severity of dysfluency correlates with basal ganglia activity in persistent developmental stuttering

Previous studies suggest that anatomical anomalies [Foundas, A. L., Bollich, A. M., Corey, D. M., Hurley, M., & Heilman, K. M. (2001). Anomalous anatomy of speech-language areas in adults with persistent developmental stuttering. Neurology, 57, 207-215; Foundas, A. L., Corey, D. M., Angeles, V., Bollich, A. M., Crabtree-Hartman, E., & Heilman, K. M. (2003). Atypical cerebral laterality in adults with persistent developmental stuttering. Neurology, 61, 1378-1385; Foundas, A. L., Bollich, A. M., Feldman, J., Corey, D. M., Hurley, M., & Lemen, L. C. et al., (2004). Aberrant auditory processing and atypical planum temporale in developmental stuttering. Neurology, 63, 1640-1646; Jancke, L., Hanggi, J., & Steinmetz, H. (2004). Morphological brain differences between adult stutterers and non-stutterers. BMC Neurology, 4, 23], in particular a reduction of the white matter anisotropy underlying the left sensorimotor cortex [Sommer, M., Koch, M. A., Paulus, W., Weiller, C., & Buchel, C. (2002). Disconnection of speech-relevant brain areas in persistent developmental stuttering. Lancet, 360, 380-383] could be at the origin of persistent developmental stuttering (PDS). Because neural connections between the motor cortex and basal ganglia are implicated in speech motor functions, PDS could also be associated with a dysfunction in basal ganglia activity [Alm, P. (2004). Stuttering and the basal ganglia circuits: a critical review of possible relations. Journal of Communication Disorders, 37, 325-369]. This fMRI study reports a correlation between severity of stuttering and activity in the basal ganglia and shows that this activity is modified by fluency shaping therapy through long-term therapy effects that reflect speech production improvement. A model of dysfunction in stuttering and possible repair modes is proposed that accommodates the data presented here and observations previously made by us and by others.     

Transcranial magnetic stimulation in child psychiatry: disturbed motor system excitability in hypermotoric syndromes

Normal development and dysfunctions of motor system excitability can be investigated in vivo by means of single‐ and paired‐pulse transcranial magnetic stimulation (TMS). While different TMS‐parameters show different developmental time courses between 8 and 16 years of age, distinct dysfunctional patterns of motor system excitability can be demonstrated in child psychiatric disorders with hypermotoric behavior: in tic disorder, a shortened cortical silent period can be stated providing evidence for deficient inhibitory mechanisms within the sensorimotor loop, probably primarily at the level of the basal ganglia. In attention deficit hyperactivity disorder (ADHD), a decreased intracortical inhibition indicates deficient inhibitory mechanisms within the motor cortex (but enhancement of intracortical inhibition after oral intake of 10 mg methylphenidate). In children with comorbid ADHD and tic disorder, the findings of a reduced intracortical inhibition as well as a shortened cortical silent period provide evidence for additive effects at the level of motor system excitability. Thus, TMS allows us to obtain substantial insight into both the normal development and the neurobiological basis of hypermotoric syndromes in child psychiatry.