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601.
The goal of this study was to examine the effect of guided affective imagery therapy in an outpatient setting.At first, 140 patients were recruited.Unfortunately, there were many drop-outs at the beginning of the study as well as at later assessments.The data of 66 patients could be evaluated.The patients were assessed at the beginning of the therapy, after 25 sessions, at the end of the therapy and 18 month after the end of the therapy. Because of too many drop-outs, the originally planned waiting-control-design was realized only to some extent.The diagnoses of the patients consisted mainly of neurotic disorders,many of them with a dysthymia. Data were gathered by standardized questionnaires. There were marked effects of the therapies at the end of the treatments and also at the follow-up evaluation.Effect sizes were larger than 0.8 in somatic symptoms, mood, depression and life satisfaction.On the one hand, therefore, these therapies had been very effective. On the other hand, it became obvious during the course of the study, how difficult it is to conduct such a design with many therapists in a private practice and without financial support. 相似文献
602.
The authors investigated relationships among spirituality, social support, childhood maltreatment, and symptoms of distress. One hundred and fifty‐eight upper‐level university students completed questionnaires related to childhood maltreatment, distress symptomatology, spirituality, and social support. Significant associations were found for (a) maltreatment with high symptoms of distress, low existential spiritual well‐being, and low social support from family; (b) symptoms of distress with low existential spiritual well‐being and low social support from family; and (c) receiving counseling for maltreatment with high symptoms of distress and low support from family. 相似文献
603.
604.
We present a psychometric scale that assesses risk taking in five content domains: financial decisions (separately for investing versus gambling), health/safety, recreational, ethical, and social decisions. Respondents rate the likelihood that they would engage in domain‐specific risky activities (Part I). An optional Part II assesses respondents' perceptions of the magnitude of the risks and expected benefits of the activities judged in Part I. The scale's construct validity and consistency is evaluated for a sample of American undergraduate students. As expected, respondents' degree of risk taking was highly domain‐specific, i.e. not consistently risk‐averse or consistently risk‐seeking across all content domains. Women appeared to be more risk‐averse in all domains except social risk. A regression of risk taking (likelihood of engaging in the risky activity) on expected benefits and perceived risks suggests that gender and content domain differences in apparent risk taking are associated with differences in the perception of the activities' benefits and risk, rather than with differences in attitude towards perceived risk. Copyright © 2002 John Wiley & Sons, Ltd. 相似文献
605.
606.
Jacoby's white noise task and an explicit recognition task were used to investigate whether individuals with eating disorders demonstrate implicit memory bias and explicit memory bias, respectively, for information related to eating and body appearance. Included were 33 women with eating disorders (seven with anorexia nervosa and 26 with bulimia nervosa), 29 with nonclinical eating disorder-related concerns, and 36 healthy controls. Results showed partial support for implicit memory bias but no support for explicit memory bias. These findings suggest that eating disorders may be characterized by relative initial automatic bias for eating disorder-relevant information but not by bias at later stages of information processing. However, previous studies have demonstrated explicit memory bias in eating disorders, which is inconsistent with this interpretation. Future research is required to clarify the precise cognitive biases associated with eating disorders. 相似文献
607.
Weber BJ 《Memory & cognition》2008,36(5):1013-1023
The Allais paradox decision bias was first offered as a challenge to the expected utility theory over 60 years ago. Although the Allais paradox is a standard challenge for normative theories of risky choice, its causes are not well understood. The present experiment uses two manipulations of the Allais paradox to investigate the commonly proposed probability-weighting explanation of the paradox. Reducing the magnitude of the outcomes did not affect the size of the Allais paradox, contradicting previous literature and supporting the probability weighting hypothesis. Reducing the probability of the nonzero outcomes to eliminate certainty reduced, but did not eliminate, the Allais paradox, a result inconsistent with probability weighting and other theories of the Allais paradox. The results suggest that the certainty effect alone cannot explain the Allais paradox. 相似文献
608.
This experiment was designed to investigate the relationship between the fundamental visual, spatial properties of existing complex streetscapes and aesthetic judgments. Participants performed aesthetic judgments over 35 photographs of typical spatial situations taken along the Meissner Strasse in Radebeul, Germany. In a modified Q-Sort procedure, the participants assigned the pictures to five categories, including 1 (beautiful) and 5 (not beautiful). Vegetation, Stylistic Uniformity, Homogeneity of Scale, and Symmetry were identified as primary components of aesthetic judgment by using principal component analysis. Stimuli, photographically edited according to these factors, were then tested using the same Q-Sort procedure, which confirmed these determinants. These results are intended to help the development of the theoretical understanding of the link between the influence of selected spatial properties and the aesthetic judgment of the visual quality of urban spaces. 相似文献
609.
Fichtner G 《The International journal of psycho-analysis》2008,89(4):827-843
Freud's early paper Psychical (or mental) treatment, first published in a family reference book for educated lay persons, was reproduced in the Gesammelte Werke with a stated publication date of 1905. This date was subsequently called into question owing to certain parts of the subject-matter (the use of hypnosis and suggestion in 'mental treatment'), and the contribution was erroneously assigned, for instance by James Strachey, to the year 1890. This error is corrected in the present paper. Furthermore, the existence of a second edition of this reference book, which contains an addition to Freud's text and appeared in 1918-19, has previously gone unnoticed. The first edition had been published in 1905-6. Freud's contribution must, however, have been written at an appreciably earlier date. The probable time of its genesis is discussed. Freud's new text is reproduced (in English translation) for the first time in an appendix to this paper. 相似文献
610.
Azad SC Kurz J Marsicano G Lutz B Zieglgänsberger W Rammes G 《Learning & memory (Cold Spring Harbor, N.Y.)》2008,15(3):143-152
Previously, we found that in the lateral amygdala (LA) of the mouse, WIN55,212-2 decreases both glutamatergic and GABAergic synaptic transmission via activation of the cannabinoid receptor type 1 (CB1), yet produces an overall reduction of neuronal excitability. This suggests that the effects on excitatory transmission override those on inhibitory transmission. Here we show that CB1 activation by WIN55,212-2 and Delta(9)-THC inhibits long-term depression (LTD) of basal synaptic transmission in the LA, induced by low-frequency stimulation (LFS; 900 pulses/1 Hz). The CB1 agonist WIN55,212-2 blocked LTD via G(i/o) proteins, activation of inwardly rectifying K+ channels (K(ir)s), inhibition of the adenylate cyclase-protein kinase A (PKA) pathway, and PKA-dependent inhibition of voltage-gated N-type Ca2+ channels (N-type VGCCs). Interestingly, WIN55,212-2 effects on LTD were abolished in CB1 knock-out mice (CB1-KO), and in conditional mutants lacking CB1 expression only in GABAergic interneurons, but were still present in mutants lacking CB1 in principal forebrain neurons. LTD induction per se was unaffected by the CB1 antagonist SR141716A and was normally expressed in CB1-KO as well as in both conditional CB1 mutants. Our data demonstrate that activation of CB1 specifically located on GABAergic interneurons inhibits LTD in the LA. These findings suggest that CB1 expressed on either glutamatergic or GABAergic neurons play a differential role in the control of synaptic transmission and plasticity. 相似文献