Book reviews

Horowitz, M. J. (Ed.) (1988). Psychodynamics and cognition. Chicago: University of Chicago press. Pp. 389. ISBN 0226-35368-0. £ 21.95

Ellis A. W. & Young, A. W. (1988). Human cognitive neuropsychology. Hove: Lawrencve erlbaum Associates Ltd. Pp. 358. ISBN 0-86377-033-9, £ 21.95 9 (hardback), 0-86377-034-7, £ 11.95 (paperback).

Humphreys, G. W., & Bruce, V.(1989). Visual cognition: Computational, experimental and neuropsychological perspectives. Hove & London: Lawrence Erlbaum Associates Ltd. Pp. 330. ISBN 0-86277-124-6, £ 19.95 (hardback), 0-86377-125-4, £ 9.95 (paperback)     

On the Control Over Stereotype Activation and Stereotype Inhibition

Stereotypes are known to influence how we think about (judge, feel, plan, attend to, remember), and act toward, others. Stereotyping is typically described as pervasive in society for a variety of reasons, one of which being that the cognitive processes that initiate stereotyping occur effortlessly, without conscious intent, and without awareness. That is, upon encountering members of stereotyped groups, we ‘uncontrollably’ have stereotypes of those groups retrieved and held accessible in a state of perceptual readiness. This occurs without our knowledge or awareness of the stereotype’s ‘primed’ status, and thus our lacking awareness of its potential to influence how we respond. The current article explores various ways this implicit process of stereotype activation is controlled. Despite the fact that stereotype activation happens with ease, and outside of awareness, control processes also operate with ease, and outside of awareness. The review illustrates a variety of ways in which a stereotype may not be triggered (and thus downstream stereotyping of a person controlled) upon encountering a person. The dominant response to a person, stereotypic or not, is determined by the goals the individual perceiver has upon entering the interpersonal encounter. Many goals relevant to interpersonal interaction promote stereotyping. However, stereotype promotion is a form of stereotype control, and if control can be exhibited in the form of promotion, it should also be demonstrable in the form of prevention. Indeed, goals that are incompatible with stereotyping yield dominant responses to having encountered a member of a stereotyped group that does not involve activation of, and may involve inhibition of, stereotypes.     

Transcendental idealism among the Jersey metaphysicians

Some questions are posed for van Fraassen, concerning the role and status of metaphysics in his Scientific Representation.     

Etiology of obsessions and compulsions: a behavioral-genetic analysis

It is unknown whether various types of obsessive-compulsive (OC) symptoms have a common genetic or environmental etiology. For example, it is unknown whether hoarding is etiologically associated with prototypic OC symptoms, such as washing, checking, and obsessing. Also unknown is whether particular OC-related symptoms are etiologically linked to the general tendency to experience emotional distress (negative emotionality). To investigate these and other issues, a community sample of 307 pairs of monozygotic and dizygotic adult twins provided scores on 6 OC-related symptoms (obsessing, neutralizing, checking, washing, ordering, and hoarding) and 2 markers of negative emotionality (trait anxiety and affective lability). Genetic factors accounted for 40%-56% of variance in the 8 phenotypic scores (M = 49% of variance for OC-related symptoms). Remaining variance was due to nonshared (person-specific) environment. More detailed analyses revealed a complex etiologic architecture, where OC-related symptoms arise from a mix of common and symptom-specific genetic and environmental factors. A general genetic factor was identified, which influenced all symptoms and negative emotionality. An environmental factor was identified that influenced all symptoms but did not influence negative emotionality. Each of the 6 types of symptoms was also shaped by its own set of symptom-specific genetic and environmental factors. The importance of genetic factors did not vary as a function of age or sex, and the architecture of general and specific etiologic factors was replicated for participants having relatively more severe OC symptoms. Gene-environment interactions were identified. Implications for an etiology-based classification system are discussed.     

Indirect peer aggression in adolescence and reproductive behavior

Evolutionary psychologists have suggested that indirect aggression during adolescence is a strategy to compete with same-sex peer rivals for resources, status, and mating opportunities. We collected survey data on 143 young adults to determine retrospectively, the amount of indirect aggressive behavior they perpetrated and the amount of indirect victimization they experienced in middle school and high school. We also collected information about reproductive opportunities such as age at first sexual intercourse and number of lifetime sex partners to determine whether aggression or victimization could be used to predict measures of reproductive opportunity. We performed a principal components analysis to develop factors from 16 aggression and victimization variables. Results indicate that females who reported indirect aggression toward peers had earlier ages at first sexual intercourse and females who were more victimized in adolescence experienced later ages at first sexual intercourse. We discuss these results in terms of intrasexual competition and evolutionary theory.     

Nonindependent and nonstationary response times in stopping and stepping saccade tasks

Saccade stop signal and target step tasks are used to investigate the mechanisms of cognitive control. Performance of these tasks can be explained as the outcome of a race between stochastic go and stop processes. The race model analyses assume that response times (RTs) measured throughout an experimental session are independent samples from stationary stochastic processes. This article demonstrates that RTs are neither independent nor stationary for humans and monkeys performing saccade stopping and target-step tasks. We investigate the consequences that this has on analyses of these data. Nonindependent and nonstationary RTs artificially flatten inhibition functions and account for some of the systematic differences in RTs following different types of trials. However, nonindependent and nonstationary RTs do not bias the estimation of the stop signal RT. These results demonstrate the robustness of the race model to some aspects of nonindependence and nonstationarity and point to useful extensions of the model.     

Virtual and drawing structures for the Müller-Lyer illusions

This research assessed the relative contribution of 3-D virtual structure that generated the stimulus drawings (scene-based and picture-based theories) and 2-D structure of the drawings (object-based theories). Virtual structures were right-angle convex and concave corners in front of and behind the picture plane, respectively. Virtual corner size was manipulated directly (Experiment 1) and indirectly by manipulating drawing station point distance (Experiments 2 and 3), corner depth (Experiment 4), and corner distance from the picture plane (Experiments 5 and 6). Experiments 2 and 4 held the size of the projected corner edge (interior target line) constant, causing virtual corner size to vary, whereas Experiments 3, 4, 5, and 6 held size of the virtual corners constant, causing size of the projected corner edge or interior target line to vary. Subjects reproduced the length of the projected corner edge (interior target line). The illusions (difference between reproduced size of the projected corner edge and T-junction control) were generally well fit by the weighted sum of virtual corner size and size of the projected corner edge, but the projected distance between boundary line terminations (intertip distance) appeared as an additional contributing factor in Experiments 5 and 6. The implications of this methodological approach are discussed for theories of the illusions.