Effect of stride frequency on the energy cost of walking in obese teenagers

The aim of this study was to compare the energy cost of obese and non-obese teenagers while walking at their preferred speed and different stride frequencies. Twelve obese and twelve non-obese teenagers walked continuously on the treadmill at their most comfortable speed for 6 periods of 4 min each. Each period corresponded to a specific stride frequency: preferred (PSF), force-driven harmonic oscillator (FDHO), PSF + 10%, PSF + 20%, PSF − 10% and PSF − 20%. Cardiorespiratory parameters were collected between the 3rd and 4th minute of each stage, and used to calculate the energy cost of walking (EC). The main results showed a significantly higher cost of walking expressed relative to lean body mass. In addition, a U-shaped relationship between EC and stride frequency was shown in both groups, with PSF and FDHO leading to a significantly lower value compared to all other frequencies. This showed first, that FDHO is a good predictor of PSF and minimal energy cost of walking in both groups, and second, that excess body fat does not affect the relationship between energy expenditure and stride frequency. Walking at lower or higher than preferred frequencies could be used as an exercise mode to promote weight loss in obese teenagers.     

Associations of Mother’s and Father’s Parenting Practices with Children’s Observed Social Reticence in a Competitive Situation: A Monozygotic Twin Difference Study

This study used the monozygotic (MZ) twin difference method to examine whether the unique environmental effects of maternal and paternal overprotection and hostility at the age of 30 months predict twins’ observed social reticence in a competitive situation in kindergarten, while controlling for the effect of family-wide influences, including genetic and shared environmental factors, family socio-economical status and twin’s birth weight. It was also examined whether these associations are moderated by parental depressive symptoms. Participants were 137 MZ twin pairs who were part of an ongoing longitudinal study. Hierarchical linear regressions revealed that differences in maternal and paternal overprotection predicted differences in twins’ social reticence, albeit only in boys. Differences in paternal hostile parenting predicted differences in girls’ reticent behavior, but only when fathers showed high levels of depressive symptoms. Hence, overprotected boys, as well as girls confronted with father’s hostility and depressive symptoms, may tend to withdraw rather than face the challenge when experiencing difficult social situations such as competition. The results from the present study suggest that targeting maladaptive maternal as well as paternal child-rearing practices and psychopathology early on may be useful for reducing later internalizing behavior in the offspring.     

Examining genetic and environmental effects on reactive versus proactive aggression

This study compared the contribution of genes and environment to teacher-rated reactive and proactive aggression in 6-year-old twin pairs (172 pairs: 55 monozygotic girls, 48 monozygotic boys, 33 dizygotic girls, 36 dizygotic boys). Genetic effects accounted for 39% of the variance of reactive aggression and for 41% of the variance of proactive aggression. The remainder of the variance was explained by unique environmental effects. Genetic as well as unique environmental effects were significantly correlated across reactive and proactive aggression (genetic correlation = .87, environmental correlation = .34), but this overlap was largely due to a common underlying form of aggression (i.e., teacher-rated physical aggression). Once common etiological factors due to physical aggression were accounted for, reactive and proactive aggression shared no other genes and only a few environmental influences, although additional specific genetic and environmental effects were observed for both reactive and proactive aggression. These specific effects indicate that both reactive and proactive aggression may be influenced mostly by socialization experiences that are specific to each type of aggression and only to a very small degree by specific genes.