Olfactory dysfunction in man: anatomical and behavioral aspects

We reviewed studies examining the olfaction of patients with brain damage for the purpose of discerning correlations between disordered structure and function. Patient samples included those with neurological disorders and neurosurgical interventions and recording of spontaneous or elicited neuronal activity. Brain areas involved in olfaction include the olfactory bulbs, the orbitofrontal and medial temporal cortices, the thalamus, and the amygdala. Despite recent advances in olfactory anatomy, understanding of how these structures are related to olfactory detection, discrimination, and recognition continues to be limited. Inadequate localization of brain lesions and lack of comprehensive behavioral assessment have thus far prevented a detailed account of the organization of olfaction in the human brain.     

A longitudinal study of an adult with aphasia: Considerations for research and therapy

This paper described investigation and therapy carried out over a one year period with an adult aphasic patient characterized by severe impairment of auditory comprehension. A deblocking technique was used to reintegrate the mechanisms for correlating sound and meaning. The visual mode of presentation of linguistic material was used both to provide a stable representation of speech units and to allow reinforcement of auditory representations. The patient was given tasks of repetition, reading aloud, and sequencing, using progressively more complex material. The behavior of the patient gives support to the viability of the theoretical constructs of linguistics: this can be seen in the differential processing of function words vs. content words, and in the orderly progression of improvement through stages explicable as the recovery of well-defined and hierarchically dependent subcomponents of linguistic organization.     

Is the Association of ADHD with Socio-Economic Disadvantage Explained by Child Comorbid Externalizing Problems or Parent ADHD?

It has been unclear whether an associations of child ADHD with socio-economic disadvantage (SES) could be accounted for by (a) parental ADHD explaining both low SES and child ADHD, and/or (b) the joint overlap of ODD or CD with low SES and ADHD. Study 1 used a community-recruited case-control sample with detailed evaluation of SES indicators, child ADHD, child externalizing, and parent ADHD symptoms (n = 931 children, 521 ADHD, 577 boys, 354 girls) in a path modeling analysis with latent variables. Study 2 evaluated ADHD and externalizing behavior in a regression model using a poverty index for SES, in 70,927 children (48.2% female) aged 5–17 years from the US 2011–2012 National Survey of Children’s Health (NSCH). In Study 1, lower SES was related to the ADHD latent variable, β = ?.18, p < .001; 95%CI [?.25,-.12]. This effect held when parent ADHD and child ODD and CD were in the model, β = ?.11, p < .01, 95% CI [?.09,-.03], equivalent to OR = 1.50, 95% CI[1.12–2.04]). In Study 2, these results replicated. Adjusting only for age and sex, children from families who were below 200% of the federal poverty line were more likely to have moderate or severe ADHD than no ADHD, versus children above that line, OR = 2.13, 95% CI[1.79,2.54], p < .001. The effect held after adjusting for disruptive/externalizing problems, OR = 1.61, p < .01, 95%CI [1.32,1.96]. The effect size for comparable models was similar across both studies, lending higher confidence to the results. It is concluded that the SES association with child ADHD is not explained by artifact and requires a mechanistic explanation.     

“'Cause You Can't Play This by Yourself”: Athletes' Perceptions of Team Influence on Their Precompetitive Psychological States

Although correlational studies indicate that team attributes are related to athletes' precompetitive psychological states, it is unknown how team members experience these relationships. The present study's purpose was to investigate athletes' perceptions of precompetitive team influence. Consequently, we worked from a constructivist realist position and used qualitative methods (interviews with competitive team-sport athletes). An inductive content analysis revealed that athletes perceived established and novel team characteristics and processes to influence their precompetitive psychological states. Athletes perceived these influences as both adaptive and maladaptive, and it appeared that they were qualified by contextual factors as well as intuitive regulatory attempts.     

The Network Structure of Childhood Psychopathology in International Adoptees

International adoptees are at an increased risk of emotional and behavioral problems, especially those who are adopted at an older age. We took a new approach in our study of the network structure and predictability of emotional and behavioral problems in internationally adopted children in Finland. Our sample was from the on-going adoption study and comprised 778 internationally adopted children (387 boys and 391 girls, mean age 10.5 (SD 3.4) years). Networks were estimated using Gaussian graphical models and lasso regularization for all the children, and separately for those who were adopted at different ages. The results showed that anxiety/depressive symptoms, social problems, and aggressiveness were the most central symptom domains. Somatic symptoms were the least central and had the weakest effect on the other domains. Similarly, aggressiveness, social problems, and attention problems were high in terms of predictability (73–65%), whereas internalizing problems were relatively low (28–56%). There were clear but local age-group differences in network structure, symptom centrality, and predictability. According to our findings, network models provide important additional information about the centrality and predictability of specific symptom domains, and thus may facilitate targeted interventions among international adoptees.     

The Communities That Care (CTC) Family Attachment Scale: Measurement Invariance Across Family Structures

Familial protective factors are an integral part of prevention approaches aimed at problematic behaviour in adolescents. However, there is scarce evidence on the role of familial protective factors in families deviating from the two-parent family configuration. For evaluating targeted (preventive) interventions, a reliable and valid measurement of familial protective factors is crucial. We investigated the factor structure of the Communities That Care (CTC) Family Attachment Scale and tested its measurement invariance in different family structures. Adolescents (n?=?2.459, grades 6–11) from Lower Saxony, Germany filled in the German version of the CTC Youth Survey. Our analyses focused on the CTC Family Attachment Scale measuring the adolescent’s attachment to the mother and the father with six items. We evaluated the postulated unidimensional structure of the scale by confirmatory factor analysis (CFA) and tested the measurement invariance using multigroup factor analyses across different family structures (two-parent family/single-parent family). We used SPSS V.23 and the R packages lavaan and semTools. The two-factor solution for the CTC Family Attachment Scale with one factor representing attachment to the mother and one indicating attachment to the father had an adequate model fit in the total sample (χ²(5)?=?29.938; p?<?.001; CFI?=?.996; TLI?=?.988; RMSEA?=?.050, SRMR?=?.019). This two-factor solution of the CTC Family Attachment Scale showed strong measurement invariance regarding adolescents living in a two-parent family vs. those living with a single parent. The two-factor CTC Family Attachment Scale appears to be a suitable measure to assess family attachment in both two-parent and single-parent families with German adolescents.     

Well-being in major depression and social phobia with and without comorbidity

Background/Objective: Lower levels in well-being have been observed in individuals with Major Depression (MDD) and Social Phobia (SP), but well-planned direct comparisons with control individuals, not suffering from a mental disorder, are lacking. Furthermore, MDD is highly comorbid with anxiety disorders, and SP with depressive disorders. This study is among the first to examine differences in well-being in individuals with a clinical diagnosis of MDD or SP compared to individuals with no such diagnosis and to test differences in well-being within the combined diagnostic categories respective with and without anxiety-depressive comorbidity. Method: Participants were 119 individuals with a diagnosis of MDD, 47 SP and 118 controls. Results: Results revealed that overall well-being as well as emotional, psychological, and social well-being were lower in the MDD and SP group compared to the control group. Individuals with comorbidity reported lower well-being than individuals without comorbidity. Conclusions: These findings have clinical implications as presence of comorbidity may require a different therapeutic approach than with no comorbidity.     

Responsibility Without Blame: Empathy and the Effective Treatment of Personality Disorder

Effective treatment of personality disorder (PD) presents a clinical conundrum. Many of the behaviors constitutive of PD cause harm to self and others. Encouraging service users to take responsibility for this behavior is central to treatment. Blame, in contrast, is detrimental. How is it possible to hold service users responsible for harm to self and others without blaming them? A solution to this problem is part conceptual, part practical. I offer a conceptual framework that clearly distinguishes between ideas of responsibility, blameworthiness, and blame. Within this framework, I distinguish two sorts of blame, which I call 'detached' and 'affective.' Affective, not detached, blame is detrimental to effective treatment. I suggest that the practical demand to avoid affective blame is largely achieved through attention to PD service users' past history. Past history does not eliminate responsibility and blameworthiness. Instead, it directly evokes compassion and empathy, which compete with affective blame.