Victim Blaming in Young, Middle-Aged, and Older Adults: Variations on the Severity Effect

It has been long known that people blame victims for the bad things that happen to them, and that people blame victims more when the victims experience severe difficulties than when they experience minor difficulties, even if the victims were not particularly irresponsible. Little previous research has examined victim blaming in middle-age and older adults. One hundred and forty-five adults in 3 age groups (18–34, 35–59, and 60–84) read 4 scenarios (2 accidents, 1 crime, and 1 fire) imbedded in other scenarios. The scenarios were varied so that the victim is either very irresponsible or not very irresponsible, and the outcome is mild or severe. The oldest group of participants blamed the victims more than the other groups. However, in contrast to the typical severity effect, the oldest group blamed the very irresponsible victim more when the outcome was mild than they did when the outcome was severe.     

知觉或记忆条件下对连续变化刺激的相似判断

控制不同材料(图形和字母)的大小、位置和长宽比连续变化,在标准和待判断刺激同时(知觉任务)或相继呈现(记忆任务)两种条件下进行相似判断的实验研究。结论为(1)任务方式对图形的相似判断有非常显著的影响;(2)知觉任务中相似判断的反应时非常显著地长于记忆任务中的反应时;(3)对材料各种特征的相似判断和加工速度差异非常显著．某些特征在相似表征中有较重要的作用。     

干预条件性恐惧记忆表达的相关影响因素分析

创伤后应激障碍是个体经历严重应激后形成的一种焦虑障碍, 对其治疗的关键是熄灭由创伤应激导致的条件性恐惧记忆。条件性恐惧的动物模型研究发现恐惧记忆一旦获得后就难以熄灭, 容易复发, 而这一点也是PTSD的关键临床症状表现之一。因此, 如何更好更持久地熄灭恐惧记忆, 是一个具有重要理论和临床意义的研究热点。本文围绕促进恐惧记忆的长久消退和破坏恐惧记忆的再巩固两方面的行为或药理干预及机制进行综述。针对本文所述的几种基础实验处理, 临床上可以研究治疗创伤后应激障碍的相应疗法。     

论医患关系恶化的哲学根源:医学的异化

针对医患关系恶化的现状论述了其哲学根源--现代医学的异化.在界定医学异化的概念并对其成因进行探讨的基础上,深入分析医学异化对医学和医患关系的影响,最后提出了消解医学异化和改善医患关系的对策,即坚持医学研究"还原论"与"整体论"的辩证统一;预防医生人格的进一步异化;坚持"以人为本"的医疗原则;切实加强医患之间的交流和沟通.     

西北民族地区中学生心理地位与人际交往能力的关系研究

以西北民族地区甘南藏族自治州的1所多民族混合学校、1所单一少数民族学校以及兰州市1所普通中学的521名中学生为被试,采用心理地位量表和中学生人际交往能力问卷,探讨不同学校类型中学生心理地位和人际交往能力的关系。结果表明:(1)西北民族地区藏汉混合学校学生的心理地位显著高于单一民族中学的学生,但与普通中学的学生之间不存在显著差异;(2)西北民族地区藏汉混合学校学生人际交往能力得分显著高于单一民族中学的学生,但与普通中学学生之间差异不显著;(3)西北民族地区中学生的心理地位与人际交往能力之间存在显著相关;(4)西北民族地区中学生的心理地位特征能够较好地预测其人际交往能力的发展水平。     

从毕生发展的视角看健康的贫富差距

健康的贫富差距是所有个体从出生到死亡都需要面对的现实, 也是全球所有国家都存在的社会问题。目前已经有相当的理论和实证研究尝试从毕生发展的视角理解健康的贫富差距。在毕生发展研究中, 健康的贫富差距有其独特的概念和测量方式。该领域重要的理论包括累积优(劣)势假设、累积不公平模型和社会阶层文化周期模型。研究者就儿童期、青少年期、成年期和老年期的健康贫富差距开展了大量的实证研究。未来毕生发展研究和实践的可能方向包括早年和当前社会经济地位的交互作用、社会经济地位的稳定特征和情境效应、跨领域实践干预等。毕生发展视角的健康贫富差距研究有助于理解健康贫富差距的发展根源和形成过程, 是促进国民健康和开展健康公平实践工作的理论基础。     

专业社会工作者的角色与和谐医患关系的构建

当前医疗纠纷频繁发生,医患关系日趋紧张,问题的复杂性呼唤着多种力量的积极参与和共同努力。作为一种综合性的助人手段,社会工作者在严格秉承专业价值观的基础上,运用三大专业方法,协助医患双方有效预防、和平解决医疗纠纷问题,肩负着构建和谐医患关系的专业使命。     

网络成瘾与心理梳理

网络成瘾问题的形成往往涉及到自身、家庭和社会等多种复杂因素,因此单纯的药物治疗、心理辅导、说教等方式都很难取得根本的实效.尝试以心理学为主,综合运用社会学、教育学、医学等多门学科知识,选取一个典型的青少年个案进行介入和研究,结果发现,网络成瘾的矫治是一个复杂而又漫长的过程,需要多方面的努力,发人深思.     

Social suggestibility to central and peripheral misinformation

This study used a laboratory-based paradigm to investigate social influences on participants' susceptibility to misleading suggestions. Participants viewed a video clip of an action sequence with one or more peers, and then were required to discuss the event with the co-witness or with the group of co-witnesses. During the discussion a confederate, posing as a peer, presented misinformation about central and peripheral features of the co-witnessed event. Results indicated that participants were more susceptible to misleading suggestions during one-on-one discussions than during group discussions. In addition, participants were susceptible to misleading suggestions about central features of the witnessed event, although to a lesser extent than they were susceptible to misleading suggestions about peripheral features.     

The emerging fragile X premutation phenotype: evidence from the domain of social cognition

Fragile X syndrome is a neurodevelopmental disorder that is caused by large methylated expansions of a CGG repeat (>200) region upstream of the FMR1 gene that results in the lack of expression of the fragile X mental retardation protein (FMRP). Affected individuals display a neurobehavioral phenotype that includes a significant impairment in social cognition alongside deficits in attentional control, inhibition and working memory. In contrast, relatively little is known about the trajectory and specificity of any cognitive impairment associated with the fragile X premutation ("carrier-status") (approximately 55-200 repeats). Here, we focus on one aspect of cognition that has been well documented in the fragile X full mutation, namely social cognition. The results suggest that premutation males display a pattern of deficit similar in profile, albeit milder in presentation, to that of the full mutation. However, little evidence emerged for a correlation between CGG repeat length and severity of phenotypic outcomes. The findings are discussed in the context of functional neuroimaging and brain-behaviour-molecular correlates. We speculate that the deficiencies in social cognition are attributable to impairment of neural pathways modulated by the cerebellum.