Cross-modality translations improve recognition by reducing false alarms

Conway and Gathercole [(1990). Writing and long-term memory: Evidence for a “translation” hypothesis. The Quarterly Journal of Experimental Psychology, 42, 513–527] proposed a translation account to explain why certain types of encoding produce benefits in memory: Switching modalities from what is presented to what is encoded enhances item distinctiveness. We investigated this hypothesis in a recognition experiment in which the presentation modality of a study list (visual vs. auditory) and the encoding activity (speaking vs. typing vs. passive encoding) were manipulated between-subjects. Manipulating encoding activity between-subjects ruled out any potential influence of the relationally distinct processing that can occur in a within-subject manipulation (in which all previous translation effects have been demonstrated). We found no overall difference in memory for words presented auditorily vs. visually nor for visual vs. auditory encoding, but critically presentation modality and encoding activity did interact. Translating from one modality to another – particularly from auditory presentation to visual encoding (typing) – led to the best memory discrimination. This was largely because of reduced false alarms, not increased hits, consistent with the distinctiveness heuristic.     

Etiology of strokes in children with sickle cell anemia

The most devastating complication of sickle cell anemia is cerebral infarction, affecting approximately 30% of all individuals with sickle cell anemia. Despite being one of the most common causes of stroke in infants and children, the mechanism of cerebral infarction in this population has not been extensively studied and is poorly understood. Multiple, synergistic factors are important in the pathogenesis of stroke including the hemodynamic effects of cerebral arterial occlusive disease, viscosity, chronic and acute anemia and acute medical events. This review focuses on the relationship between these factors in order to provide a foundation for further study of the etiology of strokes in this high-risk population.     

Facilitated cognitive disengagement in depression

The present study was conducted to test predictions derived from the hypothesis that depression may serve the purpose of adaptively facilitating disengagement from obsolete cognitive plans. Groups of students with either low or high depression scores were contrasted using a procedure that featured an initial learning phase, within which participants learned to perform a task, followed by a relearning phase within which they needed to disengage from initial learning in order to relearn to perform an altered task. It was assumed that performance in the relearning phase would be affected by the degree to which participants could readily disengage from their initial learning. As predicted, participants in the high depression group, relative to those in the low depression group, demonstrated superior performance in the relearning phase alone. The theoretical and applied implications of this finding are discussed.     

Does group efficacy increase group identification? Resolving their paradoxical relationship

Although group identification and group efficacy are both important predictors of collective action against collective disadvantage, there is mixed evidence for their (causal) relationship. Meta-analytic and correlational evidence suggests an overall positive relationship that has been interpreted as consistent with the idea that group identification leads to group efficacy. However, experimental evidence has not supported this causal relationship. To resolve this paradox, we show in an experiment that it is group efficacy that leads to increased group identification because group efficacy puts individuals' identity into action. We discuss the theoretical and practical implications of these findings.     

Morphemes in their place: Evidence for position-specific identification of suffixes

Previous research strongly suggests that morphologically complex words are recognized in terms of their constituent morphemes. A question thus arises as to how the recognition system codes for morpheme position within words, given that it needs to distinguish morphological anagrams like overhang and hangover. The present study focused specifically on whether the recognition of suffixes occurs in a position-specific fashion. Experiments 1 and 2 revealed that morphologically complex nonwords (gasful ) are rejected more slowly than orthographic controls (gasfil ) but that the same interference effect is not present when the morphemic constituents are reversed ( fulgas vs. filgas). Experiment 3 went further in demonstrating that reversing the morphemes within words (e.g., nesskind) does not yield morpheme interference effects against orthographic controls (e.g., nusskind). These results strongly suggest that suffix identification is position specific, which imposes important constraints on the further development of models of morphological processing.     

Remembered study mode: Support for the distinctiveness account of the production effect

The production effect is the finding that words spoken aloud at study are subsequently remembered better than are words read silently at study. According to the distinctiveness account, aloud words are remembered better because the act of speaking those words aloud is encoded and later recovery of this information can be used to infer that those words were studied. An alternative account (the strength-based account) is that memory strength is simply greater for words read aloud. To discriminate these two accounts, we investigated study mode judgements (i.e., “aloud”/”silent”/”new” ratings): The strength-based account predicts that “aloud” responses should positively correlate with memory strength, whereas the distinctiveness account predicts that accuracy of study mode judgements will be independent of memory strength. Across three experiments, where the strength of some silent words was increased by repetition, study mode was discriminable regardless of strength—even when the strength of aloud and repeated silent items was equivalent. Consistent with the distinctiveness account, we conclude that memory for “aloudness” is independent of memory strength and a likely candidate to explain the production effect.     

A Cybernetic Theory of Psychopathology

Abstract

Cybernetics, the study of principles governing goal-directed, self-regulating systems, offers a useful approach to understanding psychopathology or psychological dysfunction, overcoming limitations of other naturalistic approaches. Whereas influential theories of psychopathology have relied on definitions of dysfunction rooted in evolution and fitness, we define psychopathology in terms of cybernetic dysfunction, failure to make progress toward important goals. Cybernetic function in organisms is not identical to evolutionary function, despite their causal phylogenetic relationship. We define psychopathology as persistent failure to move toward one’s goals, due to failure to generate effective new goals, interpretations, or strategies when existing ones prove unsuccessful. This definition allows a thorough integration of dimensional approaches to psychopathology and personality and provides a new perspective on the nosology of mental disorder. We review evidence that the major dimensions of psychopathology correspond to major trait dimensions of personality, but we assert that extremity on these dimensions is neither necessary nor sufficient for psychopathology, which requires cybernetic dysfunction. Drawing from psychological and neurobiological research on personality and psychopathology, we present a theory of the mechanisms underlying the five major dimensions of psychopathology, some of their subdimensions, and the general risk factor for psychopathology. We conclude by discussing implications of our theory for research, diagnosis, and mental health interventions.     

The Effectiveness of Group Metacognitive Therapy for Generalised Anxiety Disorder: A Pilot Study

Generalised anxiety disorder (GAD) responds well to individually delivered metacognitive therapy (MCT). The current study investigated the effectiveness of MCT administered to groups of patients with GAD in a general outpatient treatment centre. Thirty-three consecutively referred adult patients with GAD were assessed before treatment (pretreatment), after the last treatment session (posttreatment), and six months after treatment had ended (follow-up). Analyses of treatment effects were conducted for all patients entering the study (i.e., intent-to-treat analysis, using the last-observation-carried-forward procedure), and repeated for the patients who completed treatment (completers analysis), using paired samples t-tests. Further, effect sizes (ESs) and indices of clinical significance were calculated, and compared with those reported in studies into the efficacy and effectiveness of individually delivered MCT for GAD. Among both the intent-to-treat and the completers sample, large pre- to posttreatment reductions in symptoms of worry, trait-anxiety, and general psychopathology. The magnitude of change and the degree of clinical significance were smaller than those reported in studies into the effectiveness of individually delivered MCT for GAD, whereas the attrition rate (27 %) was higher. It is tentatively concluded that MCT for GAD can be effectively delivered in a group format in a heterogenous clinical practice setting. Further evaluation is clearly indicated.