Can Ziskin withstand his own criticisms? problems with his model of cross-examination

Ziskin has mounted a concerted attack on the scientific basis of psychology and psychiatry and their ability to provide expertise to the courts. He has assailed both the clinical methods and conclusions rendered by these mental health professionals. To rebut expert testimony, Ziskin has proposed general principles of cross-examination. We discuss the limitations of this model and provide an initial study of its effectiveness.     

Creating false memories of infancy with hypnotic and non‐hypnotic procedures

This study was designed to test the conditions under while false memory reports are likely to be elicited from hypnotic and non‐hypnotic participants. Low, medium and high hypnotizables were administered either a hypnotic or non‐hypnotic suggestion for regression to the day after birth. False memories of infancy were generated in 68 of 78 age‐regressed participants, nearly half of whom reported strong beliefs in the reality of their recovered ‘memories’. Non‐hypnotic participants were slightly but significantly more likely to report infancy experiences than were hypnotic participants. However, hypnotizability did not have a significant effect on the classification of these experiences as memories or as fantasies. Implications for the experimental and clinical contexts are discussed. Copyright © 1999 John Wiley & Sons, Ltd.     

Stereotype activation and control of race bias: cognitive control of inhibition and its impairment by alcohol

Two experiments tested the hypothesis that alcohol increases race-biased responding via impairment of self-regulatory cognitive control. Participants consumed either a placebo or alcohol and then made speeded responses to stereotypic trait words presented after White and Black face primes while behavioral and event-related brain potential (ERP) data were recorded. Alcohol did not affect stereotype activation in either experiment. Experiment 2 showed that alcohol significantly impaired the ability to inhibit race-biased responses but did not reliably influence control of counterstereotypic responses. This disinhibition appears driven by impairment of regulative cognitive control, as indexed by amplitude of the negative slow wave ERP component. These findings suggest that controlling racial bias can be a function of effective implementation of basic self-regulatory processes in addition to the motivational processes identified in other research.     

Introduction to the special section on aging, cognition, and neuroimaging

In response to the recent increase in the number of laboratories engaged in the use of functional and structural neuroimaging to study cognitive aging, this special section has been compiled to serve as an entry into this area for the readers of Psychology and Aging. These articles are representative of the field and cover many of the issues faced by researchers in this area. This introduction presents some background into the techniques that are used and provides an overview of the articles.     

The disposables: our treatment of violent adolescents

This paper discusses the clinical perceptions of levels of attachment disorder as theorized by John Bowlby. The findings are based on interview data of juvenile delinquent offenders who have committed criminal acts that are serious enough to warrant their being tried as adults. The data are based on clinical interviews, and the interviewer encourages empirical research of the clinical findings. It is suggested that attachment levels may be a viable means of determining appropriate types of rehabilitative interventions for juvenile offenders.     

Protein kinase a in major depression: the link between hypothalamic-pituitary-adrenal axis hyperactivity and neurogenesis

The latest and most generative biological theories of major depression center on two major hypotheses. The first focuses on the concept that hyperactivity of the hypothalamic-pituitary-adrenal (HPA) axis leads to many of the pathological changes in the brain that accompany major depression. The second posits that neurogenesis leads to the repair of depression-related injuries. These two hypotheses are complementary: the former alludes to the etiology or consequences of depression, while the latter suggests mechanisms of antidepressant action. Significant crosstalk occurs between these two systems at many levels. Protein kinase A (PKA) may play an important role in this crosstalk at the intracellular level of signaling cascades. PKA is involved in the formation of long-term potentiation and fear conditioning in response to stress. Chronic stress leads to the suppression of hippocampal activity, which may cause the hyperactivity of the HPA axis during melancholic depression. PKA is also involved in the stimulation of hippocampal neurogenesis after antidepressant treatment. In theory, neurogenesis may lead to the restoration of hippocampal function, and this may be the mechanism that leads to antidepressant-mediated normalization of HPA hyperactivity. Thus, PKA is active during processes that potentially lead to depression and other processes that lead to the resolution of the illness. These opposing processes may be mediated by separate PKA isozymes that activate two distinct pathways. This review highlights the dual role of this enzyme in two biological hypotheses pertaining to depression and its treatment.     

Neuropsychological Issues in the Assessment of Refugees and Victims of Mass Violence

Brain injury, stressor severity, depression, premorbid vulnerabilities, and PTSD are frequently intertwined in trauma populations. This interaction is further complicated when the neuropsychologist evaluates refugees from other cultures. In addition, the observed psychiatric symptoms reported in refugees and victims of mass violence may in fact not be the primary features of PTSD and depression but psychiatric symptoms secondary to the effects of traumatic brain injury. This paper reviews the occurrence of starvation, torture, beatings, imprisonment, and other head injury experiences in refugee and POW populations to alert treators to the presence of chronic and persistent neuropsychiatric morbidity, with implications for psychosocial adjustment. The concept of fixed neural loss may also interact with environmental and emotional stresses, and a model of neuropsychological abnormalities triggered by traumatic events and influenced by subsequent stress will also be considered. Neuropsychologists working with refugees play an important role in assessing the possibility of traumatic brain injury with tools that are relatively culture-fair.