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The present experiments examined the role of the central cholinergic system in the memory impairment induced by post-training administration of a nitric oxide synthase (NOS) inhibitor in mice. Male Swiss mice received a one-trial inhibitory avoidance training (0.8 mA, 50 Hz, 1-s footshock) followed immediately by an ip injection of the NOS inhibitor
-NG-nitroarginine methyl ester (
-NAME; 100 mg/kg). Retention (cut-off time, 300 s) was tested 48 h after training. The administration of
-NAME results in memory impairment for the inhibitory avoidance task. The effects of
-NAME (100 mg/kg, ip) on retention were reversed in a dose-related manner by the centrally acting anticholinesterase physostigmine (35, 70, or 150 μg/kg, sc) administered 30 min after the NOS inhibitor. Further,
-NAME (100 mg/kg, ip)-induced memory impairment was completely antagonized by the centrally acting muscarinic cholinergic agonist oxotremorine (OTM; 25, 50, or 100 μg/kg, sc) when given 30 min after
-NAME. The peripherally acting anticholinesterase neostigmine (150 μg/kg, sc) did not modify the memory-impairing effects of
-NAME. These findings suggest that the memory impairment following post-training administration of a NOS inhibitor is mediated, at least in part, by a reduction of the activity of central muscarinic cholinergic mechanisms and are consistent with our previous view that nitric oxide may be involved in post-training neural processes underlying the storage of newly acquired information. 相似文献
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Male Swiss mice were allowed to explore a novel environment, provided by an open-field activity chamber, for 10 min. The procedure was repeated twice with a 24-h interval. The difference in the exploratory activity between the first (training) and the second (testing) exposures to the chamber was taken as an index of retention of this habituation task. Posttraining intraperitoneal administration of glucose (10–300 mg/kg) enhanced retention in a dose-related manner, although only the dose of 30 mg/kg of glucose produced significant effects. Thus, the dose–response curve adopted an inverted U-shaped form. Glucose (30 mg/kg) given to untrained mice did not modify their exploratory performance when recorded 24 h later. The effects of glucose on retention were time-dependent, suggesting an action on memory storage. The memory-improving actions of glucose were prevented by the simultaneous administration of both the central acting muscarinic cholinergic antagonist atropine (0.5 mg/kg) and by the central acting nicotinic cholinergic antagonist mecamylamine (5 mg/kg). In contrast, neither methylatropine (0.5 mg/kg), a peripherally acting muscarinic receptor blocker, nor hexamethonium (5 mg/kg), a peripherally acting nicotinic receptor blocker, prevented the effects of glucose on retention. Low subeffective doses of glucose (10 mg/kg) and the central anticholinesterase physostigmine (35 μg/kg), but not neostigmine (35 μg/kg), given together, act synergistically and facilitated retention. We suggest that glucose modulates memory storage of one form of learning elicited by stimuli repeatedly presented without reinforcement, probably through an enhancement of brain acetylcholine synthesis and/or its release. 相似文献
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Philosophia - It is argued that George Berkeley’s term ‘common sense’ does not indicate shared conviction, but the shared capacity of reasonable judgement, and is therefore to be... 相似文献
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Sellers Christina M. Díaz-Valdés Antonia Porter Andrew C. Glenn Catherine R. Miller Adam Bryant Battalen Adeline Wyman O’Brien Kimberly H. McManama 《Journal of abnormal child psychology》2021,49(11):1503-1511
Research on Child and Adolescent Psychopathology - The purpose of this study was to understand the trajectories of nonsuicidal self-injury (NSSI) and suicide plans (SP) in the 90 days... 相似文献
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Vera Margarida Cachapa Macedo Isabel Correia Marília Prada 《Journal of applied social psychology》2021,51(11):1109-1115
Overweight and obesity are risk factors for a range of chronic diseases, many of them caused by excessive consumption of unhealthy foods and insufficient consumption of healthy foods. The present study aims to experimentally test the effect of being exposed to injustice on the intention to consume healthy and unhealthy foods, through self-regulation. We predicted that injustice decreases self-regulation which in turn increases the intention to consume unhealthy foods and decreases the intention to consume healthy foods. Undergraduate students (N = 175; 89% women; Mage = 24.28, SD = 7.36) were randomly exposed to a scenario describing an unjust or a just academic situation. In a subsequent allegedly unrelated task, participants were asked to indicate their intention to consume each of 26 food items (half depicting unhealthy foods and half healthy foods, random order) during the subsequent week. As expected, injustice decreased self-regulation which in turn increased the intention to consume unhealthy foods and decreased the intention to consume healthy foods. These results highlight the central importance of justice perceptions in the study of consumption and contribute to frame it in the study of the consequences of economic inequalities for nutrition. 相似文献