The performance of attention-deficit-disordered and normal children on conservation tasks

The present study was designed to investigate whether a specific subgroup of learning-disabled children (attention-deficit-disordered, ADD) differed from their normal counterparts on Piagetian tasks of conservation. The subjects were 34 third-and fourth-grade children. Seventeen children had been diagnosed as ADD; the remaining half were designated as normal. The two groups were equivalent in chronological age, mental age, and intelligence. The results indicated significant differences between the two groups on tasks tapping conservation of substance and number. These results may support the theory of a lag in the development of conservation in children with specific learning disabilities (ADD). Implications of the present research for therapeutic and remedial programs that serve ADD children are also discussed.     

Dispersion in models of categorical perception

A signal detection theory model of auditory discrimination with a nonlinear mapping from stimulus continuum to perceptual continuum can account for the enhanced discrimination at the category boundary found in categorical perception. Properties of this transformation are specified by a unimodal “dispersion function”. Furthermore, it is shown that a system consisting of two acoustic feature detectors with an associated decision function is also a dispersive system, which models categorical perception of a stimulus continuum as well as boundary shifts under adaptation. The effect of detector adaptation on discrimination is discussed in view of three different types of decision variable and different types of detector noise.     

Measurement and structure of pathological gambling behavior

The serious effects of pathological gambling on both gamblers and their families warrants research on the disorder. This study aimed at identifying components of pathological gambling with the goal of developing methods for early detection and preventive strategies. A factor analysis of behaviors hypothesized to relate to pathological gambling resulted in a multidimensional model of the disorder. Eight factors were identified, including distinct neurotic, psychopathic, and impulsive components, and used in a comparative analysis of 83 admitted compulsive gamblers and 61 nongamblers. Five of the factors differentiated the two groups even when sex, age, and education were controlled. Implications of these findings are discussed.     

On the self-serving function of social anxiety: shyness as a self-handicapping strategy

We tested the hypothesis that socially anxious or shy individuals use their anxiety symptoms as a strategy to control attributions made about their performances in social-evaluative settings (i.e., self-handicapping strategies). Specifically, we predicted that trait-socially anxious or shy persons would report more symptoms of social anxiety in an evaluative setting in which anxiety or shyness could serve as an excuse for poor performance than would individuals in (a) an evaluative setting in which shyness was precluded as an excuse or (b) a nonevaluative setting. Furthermore, we predicted that this self-protective pattern of symptom reporting would not occur for individuals who were not trait-socially anxious because these persons would not commonly use such symptoms as a self-handicapping strategy. Results supported these predictions for male subjects, but not for female subjects. Sex differences in the strategic use of shyness are discussed in relation to other research on sex differences in the etiology and correlates of social anxiety.     

Flicker fusion frequency and organic syndrome in alcoholics

Flicker fusion frequencies of 48 healthy controls and of 35 alcoholics were measured to detect possible associations between flicker fusion and signs of nonspecific brain damage, the aim of the study. FFF was statistically significantly lower in patients addicted to alcohol than in healthy subjects. Lower flicker fusion frequencies were associated with the severeness of organic psychosis as rated psychopathologically. The hypothesis that addiction to alcohol is a sign of diffuse, nonspecific, organic brain damage was discussed.