The World as Representation: Schopenhauer's Arguments for Transcendental Idealism

In this paper, I discuss Spinoza's conception of the cosmological argument for the existence of God (hereafter CA), as it can be reconstructed taking departure from Letter 12. By a CA, I understand, quite broadly, an argument which infers a posteriori the existence of a necessary being, usually identified as God, from the experience that there exists some other being, often oneself, the existence of which is contingent upon the existence of this necessary being. The difference between various versions of the CA stems from differences in the way in which the relation of existential dependence is understood. The aim of this paper is to reconstruct a version of the CA which would be valid on Spinoza's principles. I argue that such a version must be understood in the context of Spinoza's causal rationalism. By such causal rationalism, I mean a doctrine committed to the idea that everything is rational and thus explicable and that causal explanations are somehow fundamental. Moreover, I hold that, in Spinoza's causal rationalism, there is one form of causation through which all other types of causation are ultimately understood. This ‘archetype’ of causation is self-causation.     

Do Tropes Resolve the Problem of Mental Causation?

David Robb has argued (in 'The Properties of Mental Causation', The Philosophical Quarterly , 47 (1997), pp. 178–94), that tropes are the properties of causation, i.e., the properties in virtue of which causal relations hold. He thinks that the welcome upshot of this is that there is no problem of mental causation. I argue that his reasons for thinking that tropes are the properties of causation are unsound. In addition, even if one takes the trope approach as a reasonable reform of our current practice, the problem of mental causation still arises. It is just been hidden in the question of what makes tropes identical.     

MENTAL CAUSATION FROM THE TOP-DOWN

Dual-attribute theories are alleged to face a problem with mental causation which commits them to either epiphenomenalism or overdetermination – neither of which is attractive. The problem, however, is predicated on assumptions about psychophysical relations that dual-attribute theorists are not obliged to accept. I explore one way they can solve the problem by rejecting those assumptions.     

Agent causation as the solution to all the compatibilist’s problems

In a recent paper I argued that agent causation theorists should be compatibilists. In this paper, I argue that compatibilists should be agent causation theorists. I consider six of the main problems facing compatibilism: (i) the powerful intuition that one can’t be responsible for actions that were somehow determined before one was born; (ii) Peter van Inwagen’s modal argument, involving the inference rule (β); (iii) the objection to compatibilism that is based on claiming that the ability to do otherwise is a necessary condition for freedom; (iv) “manipulation arguments,” involving cases in which an agent is manipulated by some powerful being into doing something that he or she would not normally do, but in such a way that the compatibilist’s favorite conditions for a free action are satisfied; (v) the problem of constitutive luck; and (vi) the claim that it is not fair to blame someone for an action if that person was determined by forces outside of his or her control to perform that action. And in the case of each of these problems, I argue that the compatibilist has a much more plausible response to that problem if she endorses the theory of agent causation than she does otherwise.     

How does downward causation exist?—A comment on Kim’s elimination of downward causation

The importance of downward causation lies in showing that it shows that functional properties such as mental properties are real, although they cannot be reduced to physical properties. Kim rejects nonreductive physicalism, which includes leading functionalism, by eliminating downward causation, and thereby returns to reductionism. In this paper, I make a distinction between two aspects of function—functional meaning and functional structure and argue that functional meaning cannot be reduced to the physical level whereas functional structure can. On this basis, I further distinguish between the integer of the function, which includes the functional meaning and the functional structure, and the whole of the functional realization, and also among the strong, medial, and weak supervenience relations. So-called downward causation is indeed the relationship between the whole of the functional realization and its physical realizer, which is a whole-part relation instead of a relation between levels. As a result of understanding downward causation in this way and abandoning the principle of the causal closure of the physical world, Kim’s argument becomes invalid and nonreductive functionalism, justified.     

No End in Sight: Causal Loops in Philosophy, Physics and Fiction

There have been many objections to the possibility oftime travel. But all the truly interesting ones concern the possibility of reversecausation. What is objectionable about reverse causation? I diagnose that the trulyinteresting objections are to a further possibility: that of causal loops. I raisedoubts about whether there must be causal loops if reverse causation obtains; but devote themajority of the paper to describing, and dispelling concerns about, various kinds ofcausal loop. In short, I argue that they are neither logically nor physically impossible.The only possibly objectionable feature that all causal loops share is that coincidenceis required to explain them. Just how coincidental a loop will be varies: some arereally quite ordinary, and some are incredibly unlikely. I end by speculating thatthe tendency amongst physicists to avoid discussion of causal loops involving intentionalaction may have been unfortunate, since intentional action is an excellent way tonon-mysteriously bring about what otherwise would have been an unlikely coincidence. Hencecausal loops may be more likely in a world with beings like us, than in one without.     

Agent causation and the alleged impossibility of rational free action

Galen Strawson has claimed that “the impossibility of free will and ultimate moral responsibility can be proved with complete certainty.” Strawson, I take it, thinks that this conclusion can be established by one argument which he has developed. In this argument, he claims that rational free actions would require an infinite regress of rational choices, which is, of course, impossible for human beings. In my paper, I argue that agent causation theorists need not be worried by Strawson’s argument. For agent causation theorists are able to deny a key principle which drives the regress. Oversimplifying things a bit, the principle states that if one is responsible for her rational actions, then she was antecedently responsible for the reasons on which she acted.     

Backwards causation and the permanence of the past

Can a present or future event bring about a past event? An answer to this question is demanded by many other interesting questions. Can anybody, even a god, do anything about what has already occurred? Should we plan for the past, as well as for the future? Can anybody precognise the future in a way quite different from normal prediction? Do the causal laws and the past jointly preclude free action? Does current physical theory entail a consistent version of backwards causation? Recent articles on the problem of backwards causation have drawn attention to the importance of the principle of the fixity of the past: that the past is now fixed. It can be shown that the standard argument against backwards causation (the bilking experiment) simply builds in the assumption of past fixity. A fixed past deprives future events of past efficacy. This has naturally led to the speculation that by abandoning past fixity real power over the past may be possible. In this paper I show that in order to have an interesting thesis of backwards causation it is not enough simply to drop past fixity. More must go. In particular, to ensure what could be called future-to-past efficacy we must abandon two entrenched principles of permanence: the principle of permanent fixity, and the principle of permanent truth. The only alternative for backwards causal theorists is to embrace real contradictions in nature.     

Action,movement, and neurophysiology

Action is to be distinguished from (mere) bodily movement not by reference to an agent's intentions, or his conscious control of his movements (Sect. I), but by reference to the agent as cause of those movements, though this needs to be understood in a way which destroys the alleged distinction between agent‐causation and event‐causation (Sect. II). It also raises the question of the relation between an agent and his neurophysiology (Sect. III), and eventually the question of the compatibility of purposive and mechanistic accounts of human behaviour (Sect. IV). For the two to be compatible it is necessary that, e.g., intentions and brain states be not merely co‐existent but also causal equivalents, in a way which allows for the mechanical explanation of teleological states — or vice versa.     

Negative causation in causal and mechanistic explanation

Instances of negative causation—preventions, omissions, and the like—have long created philosophical worries. In this paper, I argue that concerns about negative causation can be addressed in the context of causal explanation generally, and mechanistic explanation specifically. The gravest concern about negative causation is that it exacerbates the problem of causal promiscuity—that is, the problem that arises when a particular account of causation identifies too many causes for a particular effect. In the explanatory context, the problem of promiscuity can be solved by characterizing the phenomenon to be explained as a contrast between two or more events or non-events. This contrastive strategy also can solve other problems that negative causation presents for the leading accounts of mechanistic explanation. Along the way, I argue that to be effective, accounts of causal explanation must incorporate negative causation. I also develop a taxonomy of negative causation and incorporate each variety of negative causation into the leading accounts of mechanistic explanation.     

The Agency Theory of Causality,Anthropomorphism, and Simultaneity

The purpose of this article is to examine two important issues concerning the agency theory of causality: the charge of anthropomorphism and the relation of simultaneous causation. After a brief outline of the agency theory, sections 2–4 contain the refutation of the three main forms in which the charge of anthropomorphism is to be found in the literature. It will appear that it is necessary to distinguish between the subjective and the objective aspect of the concept of causation. This will lead, in section 5, to contrast two kinds of anthropomorphism, one which has been rightly rejected by modern science and one which is fully compatible with the objective reality of the causal processes. Finally, section 6 will apply the preceding considerations to simultaneous causation. On the one hand, in a basic sense, there can be no simultaneous causal relations. On the other hand, simultaneous causation arises when we consider the natural change by abstracting from the agent and from her/his projects of intervention in reality.     

Causal Reasoning and Clinical Practice: Challenges from Molecular Biology

Not only has the philosophical debate on causation been gaining ground in the last few decades, but it has also increasingly addressed the sciences. The biomedical sciences are among the most prominent fields that have been considered, with a number of works tackling the understanding of the notion of cause, the assessment of genuinely causal relations and the use of causal knowledge in applied contexts. Far from denying the merits of the debate on causation and the major theories it comprises, this paper is meant as a stimulus for theorists of causation in the philosophy of biomedicine, with a focus on clinical matters. Without aiming at putting forward an original theory of causation and starting from the narration of two actual but paradigmatic cases at the joints between biomedical research and clinical practice, we want to point out that some pathological situations addressed by molecular medicine actually prove resistant to (at least) some of our major epistemological accounts of causal explanation. Given this scenario, which is very frequent in our hospitals, our analysis aims to provide a stimulus for the debate among theorists of causation in biomedicine interested in real cases in science in practice. We believe that this might in turn encourage some more general rethinking of the complex intertwinement of science, philosophy of science and ethics, as well as of the role of philosophy of science for clinical medicine itself.

     

Absence Causation and a Liberal Theory of Causal Explanation

For the framework of event causation—i.e. the framework according to which causation is a relation between events—absences or omissions pose a problem. Absences, it is generally agreed, are not events; so, under the framework of event causation, they cannot be causally related. But, as a matter of fact, absences are often taken to be causes or effects. The problem of absence causation is thus how to make sense of causation that apparently involves absences as causes or effects. In an influential paper, Helen Beebee offers a partial solution to the problem by giving an account of causation by absence (i.e. causation in which absences are supposed to be causes). I argue that Beebee's account can be extended to cover causation of absence (i.e. causation in which absences are supposed to be effects) as well. More importantly, I argue that the extended Beebeeian account calls for a major modification to David Lewis's theory of causal explanation, usually taken as standard. Compared to the standard theory, the result of this modification, which I shall call ‘the liberal theory of causal explanation’, has, among other things, the advantage of being able to accommodate causal explanations in which the explananda are not given in terms of events.     

Causal Powers, Realization, and Mental Causation

Sydney Shoemaker has attempted to save mental causation by a new account of realization. As Brian McLaughlin argues convincingly, the account has to face two major problems. First, realization does not guarantee entailment. So even if mental properties are realized by physical properties, they need not be entailed by them. This is the first, rather general metaphysical problem. A second problem, which relates more directly to mental causation is that Shoemaker must appeal to some kind of proportionality as a constraint on causation in order to avoid redundant mental causation. I argue that, in addition, a “piling problem” arises, since causal powers seem to be bestowed twice. Then, I try to sketch an alternative view of the relation between causal powers and properties—a reductionist view—which fares better on some accounts. But it may have to face another and, perhaps, serious problem, the “problem of the natural unity of properties”. Finally, I will pose a question about the relation between causal powers and causation.

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Causes,Counterfactuals, and Non-Locality

In order to motivate the thesis that there is no single concept of causation that can do justice to all of our core intuitions concerning that concept, Ned Hall has argued that there is a conflict between a counterfactual criterion of causation and the condition of causal locality. In this paper I critically examine Hall's argument within the context of a more general discussion of the role of locality constraints in a causal conception of the world. I present two strategies that defenders of counterfactual accounts of causation can pursue to respond to Hall's challenge—including the adoption of a counterfactual condition that is sufficient for causal action-at-a-distance in place of Hall's ‘process’ condition—and conclude that Hall's argument against counterfactual accounts of causation is unsuccessful.     

Disproportional mental causation

In this paper I do three things. First, I argue that Stephen Yablo’s influential account of mental causation is susceptible to counterexamples involving what I call disproportional mental causation. Second, I argue that similar counterexamples can be generated for any alternative account of mental causation that is like Yablo’s in that it takes mental states and their physical realizers to causally compete. Third, I show that there are alternative nonreductive approaches to mental causation which reject the idea of causal competition, and which thus are able to allow for disproportional mental causation. This, I argue, is a significant advantage for such noncompetitive accounts.     

Mental causation in a physical world

It is generally accepted that the most serious threat to the possibility of mental causation is posed by the causal self-sufficiency of physical causal processes. I argue, however, that this feature of the world, which I articulate in principle I call Completeness, in fact poses no genuine threat to mental causation. Some find Completeness threatening to mental causation because they confuse it with a stronger principle, which I call Closure. Others do not simply conflate Completeness and Closure, but hold that Completeness, together with certain plausible assumptions, entails Closure. I refute the most fully worked-out version of such an argument. Finally, some find Completeness all by itself threatening to mental causation. I argue that one will only find Completeness threatening if one operates with a philosophically distorted conception of mental causation. I thereby defend what I call naïve realism about mental causation.     

Causal Inference from Descriptions of Experimental and Non-Experimental Research: Public Understanding of Correlation-Versus-Causation

The human tendency to conflate correlation with causation has been lamented by various scientists (Kida, 2006; Stanovich, 2009), and vivid examples of it can be found in both the media and peer-reviewed literature. However, there is little systematic data on the extent to which individuals conflate correlation with causation. In three experiments, we presented people with one of four research vignettes generated from the combination of two independent variables: whether the vignette described an experimental or non-experimental design, and whether it revealed a positive or negative association. Upon reading their vignette, participants selected inferences that could be drawn from the findings. Participants drew causal inferences from non-experimental vignettes as often as they did from experimental vignettes, and more frequently for causal statements and directions of association that fit with intuitive notions than for those that did not. We discuss our findings in relation to other biases in human thinking.     

Spinoza's Essentialist Model of Causation

Spinoza is most often seen as a stern advocate of mechanistic efficient causation, but examining his philosophy in relation to the Aristotelian tradition reveals this view to be misleading: some key passages of the Ethics resemble so much what Suárez writes about emanation that it is most natural to situate Spinoza's theory of causation not in the context of the mechanical sciences but in that of a late scholastic doctrine of the emanative causality of the formal cause; as taking a look at the seventeenth‐century philosophy of mathematics reveals, this is in consonance also with Spinoza's geometrical cast of mind. Against this background, I examine Spinoza's essentialist model of causation according to which each thing has a formal character determined by the thing's essence and what follows from that essence. In the case of real things this essential causal architecture results in efficacy, i.e. in bringing about real effects, the key idea being that without the essential, formally structured causal thrust there would be no efficacy in the first place. I also explain how this model accounts for efficient causation taking place between finite things.     

Two Kinds of Role Property

The realization relation is commonly explicated via, or identified with, the causal role playing relation. However, the realization relation does not formally match the causal role playing relation. While realization is a relation between a base realizer property and a single higher level realized property, I argue that the causal role playing relation as typically defined is a relation between a base property and two higher-level role properties. Advocates of using causal role playing to explicate realization must therefore decide which of the two kinds of role property they identify with realized properties; I argue that neither choice is good, and the fact that one must make a choice is worse. A search for a new way of defining realization is in order.