Agency,Teleological Control and Robust Causation

I propose a compatibilist theory of agency and responsibility, according to which an agent is responsible for an effect, if and only if, she is the earliest source of robust causation over it, via an action she carried out in the service of her long term interests. This theory deploys a notion of teleological control, which is a type of guidance-control of the agent over the effect and it involves action plans and means-end reasoning. The theory makes room for degrees of responsibility, and accounts for the distinction between compulsion and determination. The teleological control view is informed by neuroscience and cognitive theory, and while it is indifferent to the distinction between determinism and indeterminism, it contends that the property of natural laws relevant to agency is the presence of successive stages of attractor and bifurcation dynamics. While the former grounds robust causation over effects of actions, the latter limits the temporal range of robustness, allowing us to characterize responsibility in terms of the earliest sources of robust causation.     

Causation in a New Old Key

I argue (1) that it is not philosophically significant whether causation is linguistically represented by a predicate or by a sentence connective; (2) that there is no philosophically significant distinction between event- and states-of-affairs-causation; (3) that there is indeed a philosophically significant distinction between agent- and event-causation, and that event-causation must be regarded as an analog of agent-causation. Developing this point, I argue that event-causation's being in the image of agent-causation requires, mainly, (a) that the cause is temporally prior to the effect, (b) that the cause necessitates (is sufficient with necessity) for the effect. Causal necessity is explained as a derivative of nomological necessity, and finally, via a definition of the causal sentence connective, the logic of event-causation is shown to be a part of temporal modal logic.     

Mental Causation and the Metaphysics of Causation

The paper argues for four claims: (1) The problem of mental causation and the argument for its solution in terms of the identity of mental with physical causes are independent of the theory of causation one favours. (2) If one considers our experience of agency as described by folk psychology to be veridical, one is committed to an anti-Humean metaphysics of causation in terms of powers that establish necessary connections. The same goes for functional properties in general. (3) A metaphysics of causation in terms of powers is compatible with physics. (4) If combined with the argument for mental causes being identical with physical causes, that metaphysics leads to a conservative reductionism.     

Causation as Property Acquisition

Persistence theories of causation – such as transference theory, conserved-quantity theory, and Douglas Ehring's theory – attempt to analyzecausation in terms of some persisting entityconnecting cause and effect. While mostpersistence accounts are intended as empiricaltheories, this article develops a persistenceanalysis of the concept of causation. The basic idea is that the central concept ofdirect causation can be analyzed in terms ofproperty acquisition. The analysis cohereswith our ordinary causal judgments andprovides a straightforward explanation of thedirection of causation. It also explains whybackwards causation is conceptually problematic.     

Causation Without Influence

David Lewis’s latest theory of causation defines the causal link in terms of the relation of influence between events. It turns out, however, that one event’s influencing another is neither a necessary nor sufficient condition for its being a cause of that event. In the article one particular case of causality without influence is presented and developed. This case not only serves as a counterexample to Lewis’s influence theory, but also threatens earlier counterfactual analyses of causation by admitting a particularly troublesome type of preemption. The conclusion of the article is that Lewis’s influence method of solving the preemption problem fails, and that we need a new and fresh approach to the cases of redundant causation if we want to hold on to the counterfactual analysis of causation.     

Counterfactual Causation and Mental Causation

Counterfactual conditionals have been appealed to in various ways to show how the mind can be causally efficacious. However, it has often been overestimated what the truth of certain counterfactuals actually indicates about causation. The paper first identifies four approaches that seem to commit precisely this mistake. The arguments discussed involve erroneous assumptions about the connection of counterfactual dependence and genuine causation, as well as a disregard of the requisite evaluation conditions of counterfactuals. In a second step, the paper uses the insights of the foregoing analyses to formulate a set of counterfactuals-based conditions that are characterized as sufficient to establish singular causal claims. The paper concludes that there are ample reasons to believe that some mental events satisfy all these conditions with respect to certain further events and, hence, that mental events sometimes are causes.     

Causation and Counterfactual Dependence

Recently Stephen Barker has raised stimulating objections to the thesis that, roughly speaking, if two events stand in a relation of counterfactual dependence, they stand in a causal relation. As Ned Hall says, however, this thesis constitutes the strongest part of the counterfactual analysis of causation. Therefore, if successful, Barker’s objections will undermine the cornerstone of the counterfactual analysis of causation, and hence give us compelling reasons to reject the counterfactual analysis of causation. I will argue, however, that they do not withstand scrutiny.     

Mental Causation versus Physical Causation: No Contest

Common sense supposes thoughts can cause bodily movements and thereby bring about changes in where the agent is or how his surroundings are. Many philosophers suppose that any such outcome is realized in a complex state of affairs involving only microparticles; that previous microphysical developments were sufficient to cause that state of affairs; hence that, barring overdetermination, causation by the mental is excluded. This paper argues that the microphysical swarm that realizes the outcome is an accident (Aristotle) or a coincidence (David Owens) and has no cause, though each component movement in it has one. Mental causation faces no competition "from below".     

Computation and Causation

The computer's effect on our understanding of causation has been enormous. By the mid-1980s, philosophical and social-scientific work on the topic had left us with (1) no reasonable reductive account of causation and (2) a class of statistical causal models tied to linear regression. At this time, computer scientists were attacking the problem of equipping robots with models of the external that included probabilistic portrayals of uncertainty. To solve the problem of efficiently storing such knowledge, they introduced Bayes Networks and directed graphs. By attaching a causal interpretation to Bayes Networks, the philosophy of causation changed dramatically. We are now able to be extremely general about how causal structure connects to data, and systematic about when causal structures are empirically indistinguishable. In this essay I try to motivate and describe this synthesis.     

II—Control Variables and Mental Causation

I introduce the notion of a 'control variable' which gives us a way of seeing how mental causation could be an unproblematic case of causation in general, rather than being some sui generis form of causation. Psychological variables may be the control variables for a system for which there are no physical control variables, even in a deterministic physical world. That explains how there can be psychological causation without physical causation, even in a deterministic physical world.     

Mental Causation is Not Just Downward Causation

According to a popular model of mental causation, an irreducible mental cause M1 brings about an irreducible mental effect M2 by bringing about M2's supervenience base (ground, realizer, etc.), P2. Call this ‘the Downward Causation View’. This paper raises doubts about the Downward Causation View on grounds that M1 does not cause M2 immediately and there is no causal chain from M1 to M2. Prima facie, then, M1 does not cause M2 on this view. But a theory of mental causation ought to account for how some mental phenomena cause other mental phenomena; so rival theories are to be preferred. After setting out the problem, I consider replies; all fail. 1     

A Nonreductivist Solution to Mental Causation

Nonreductive physicalism provides an appealing solution to the nature of mental properties. But its success as a theory of mental properties has been called into doubt by claims that it cannot adequately handle the problems of mental causation, as it leads either to epiphenomenalism or to thoroughgoing overdetermination. I argue that these apparent problems for the nonreductivist are based in fundamental confusion about causation and explanation. I distinguish two different types of explanation and two different relations to which they appeal: causation and determination. I argue that these types of explanation do not compete with one another, nor do these relations jointly result in overdetermination. In closing I develop a nonreductivist solution to mental causation which avoids both the hazards of epiphenomenalism and of overdetermination and so demonstrates a way to save nonreductive physicalism from the problems of mental causation.     

Cause,Effect, And Fake Causation

The possibility of apparently negative causation has been discussed in a number of recent works on causation, but the discussion has suffered from beingscattered. In this paper, the problem of apparently negative causation and its attemptedsolutions are examined in more detail. I discuss and discard three attempts that have beensuggested in the literature. My conclusion is negative: Negative causation shows that thetraditional cause & effect view is inadequate. A more unified causal perspective is needed.     

Degrees of Causation

The primary aim of this paper is to analyze the concept of degrees of causal contribution for actual events and examine the way in which it can be formally defined. This should go some way to filling out a gap in the legal and philosophical literature on causation. By adopting the conception of a cause as a necessary element of a sufficient set (the so-called NESS test) we show that the concept of degrees of causation can be given clear and even empirical meaning. We then apply a game theoretical framework to derive a measure of causal contribution. Our favoured measure turns out to be a generalised version of the normalized Penrose–Banzhaf index of voting power.     

Spinoza's Essentialist Model of Causation

Spinoza is most often seen as a stern advocate of mechanistic efficient causation, but examining his philosophy in relation to the Aristotelian tradition reveals this view to be misleading: some key passages of the Ethics resemble so much what Suárez writes about emanation that it is most natural to situate Spinoza's theory of causation not in the context of the mechanical sciences but in that of a late scholastic doctrine of the emanative causality of the formal cause; as taking a look at the seventeenth‐century philosophy of mathematics reveals, this is in consonance also with Spinoza's geometrical cast of mind. Against this background, I examine Spinoza's essentialist model of causation according to which each thing has a formal character determined by the thing's essence and what follows from that essence. In the case of real things this essential causal architecture results in efficacy, i.e. in bringing about real effects, the key idea being that without the essential, formally structured causal thrust there would be no efficacy in the first place. I also explain how this model accounts for efficient causation taking place between finite things.     

因果型连动结构的逻辑刻画

逻辑学以及形式语义学对汉语连动结构的研究一般都停留在一阶逻辑的层面上,即通过一阶逻辑中的形式化方法给出连动结构的形式化构造。但是在对不同类型连动结构进行更为深入的研究时,一阶逻辑这一工具就显的不够了。本文中,作者将以因果型连动结构为研究对象,利用一阶模态逻辑为其构建形式系统并尝试给出因果型连动结构和途径–目的型连动结构之间的区别和联系。     

Emergent Causation

Downward causation is commonly held to create problems for ontologically emergent properties. In this paper I describe two novel examples of ontologically emergent properties and show how they avoid two main problems of downward causation, the causal exclusion problem and the causal closure problem. One example involves an object whose colour does not logically supervene on the colours of its atomic parts. The other example is inspired by quantum entanglement cases but avoids controversies regarding quantum mechanics. These examples show that the causal exclusion problem can be avoided, in one case by showing how it is possible to interact with an object without interacting with its atomic parts. I accept that emergence cannot be reconciled with causal closure, but argue that violations of causal closure do not entail violations of the base-level laws. Only the latter would conflict with empirical science.     

Freedom,Causation, and the Consequence Argument

The problem of analyzing causation and the problem of incompatibilism versus compatibilism are largely distinct. Yet, this paper will show that there are some theories of causation that a compatibilist should not endorse: namely, counterfactual theories, specifically the one developed by David Lewis and a newer, amended version of his account. Endorsing either of those accounts of causation undercuts the main compatibilist reply to a powerful argument for incompatibilism. Conversely, the argument of this paper has the following message for incompatibilists: you have reason to consider defending a counterfactual theory of causation. This revised version was published online in June 2006 with corrections to the Cover Date.     

Interventionism and Higher-level Causation

Several authors have recently claimed that the notorious causal exclusion problem, according to which higher-level causes are threatened with causal pre-emption by lower-level causes, can be avoided if causal relevance is understood in terms of Woodward's interventionist account of causation. They argue that if causal relevance is defined in interventionist terms, there are cases where only higher-level properties, but not the lower-level properties underlying them, qualify as causes of a certain effect. In this article, I show that the line of reasoning supposed to establish this claim does not succeed and that interventionism is not better capable of dealing with higher-level causal claims than other accounts of causation. According to Woodward, higher-level causal claims are nonetheless more adequate than lower-level ones if they describe a realization-independent dependency relationship and, hence, meet the requirement that causes should be proportional to their effects. I argue, however, that combining interventionism with proportionality considerations raises difficulties and that, therefore, Woodward's account does not vindicate higher-level causation.