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1.
The etiology of the observed relationship between general cognitive ability and reading performance was investigated by analyzing data from samples of twin pairs tested in the Colorado Learning Disabilities Research Center. Bivariate phenotypic and genetic structural equation models were fitted to data from 486 twin pairs selected for reading deficits (276 identical and 210 same-sex fraternal) and 314 control pairs (195 identical and 119 same-sex fraternal). Subtests of the Peabody Individual Achievement test (Reading Recognition, Reading Comprehension, and Spelling) were used as measures of reading performance, and verbal and performance IQ scores from the Wechsler Intelligence Scale for Children-Revised (WISC-R) or Wechsler Adult Intelligence Scale-Revised (WAIS-R) were used as indices of general cognitive ability. The results of these confirmatory factor analyses indicate that shared environmental influences do not contribute significantly to covariation between general cognitive ability and reading performance. In contrast, genetic influences contribute substantially to the relationship between the two latent factors. Moreover, the genetic and phenotypic variances of reading performance in the proband group are larger than those in controls, whereas both the genetic and phenotypic correlations between reading performance and general cognitive ability are lower in probands. These results are consistent with recent reports that reading disability may be caused by one or more genes with major effects.  相似文献   

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3.
T S Parish 《Adolescence》1990,25(98):353-356
In the present study, 334 youths evaluated their families by responding to the Personal Attribute Inventory for Children. An analysis of variance revealed no significant main effects due to respondents' birth order or gender, but did find a significant main effect due to family structure and a significant two-way interaction effect between respondents' family structure and gender. Specifically, males from divorced remarried families and females from divorced nonremarried families were found to evaluate their respective families significantly more negatively than did their counterparts from other familial configurations. Implications of these findings are discussed.  相似文献   

4.
中文读写能力及其相关因素的结构模型   总被引:3,自引:0,他引:3       下载免费PDF全文
运用问卷调查法考查了中文儿童读写能力及其相关因素之间的关系.通过验证性因素分析对量表的信度和效度指标进行了分析,发现量表具有较好的项目信度和结构效度.结构模型分析发现阅读听写能力影响书面语意义理解和书写技能,而书面语意义理解和书写技能对书面表达有显著影响.家庭阅读背景影响儿童的口语能力和阅读听写能力,口语能力影响意义理解和阅读听写能力,基本知觉加工影响口语能力和阅读听写,动作技能影响书写技能.上述结果表明中文字词识别、书写技能和意义理解水平都影响书面表达,即段落产生能力.而口语能力、家庭阅读背景、基本知觉加工和动作技能分别对读写能力的不同侧面产生影响.本研究还分析了模型的年龄和性别特点.  相似文献   

5.
IQ tests were administered to all available members over 4 years old in 101 transracial adoptive families when the adopted children were an average of 7 years old and again when they averaged 17 years old. At both times, 426 members of 93 families were studied; 398 were seen in person and administered the WAIS-R or WISC-R. IQ correlations were calculated for adopted and biological parent-child pairs, and for genetically related and unrelated siblings. Educational levels of birth parents were correlated with the IQ scores of their adopted-away children. Results show that biologically related family members tended to resemble each other intellectually more than did adoptive family members at both time points. IQ correlations for biological parent-child pairs exceeded those for adoptive parent-child pairs, and correlations were greater for genetically related than unrelated siblings. In late adolescence, the IQ scores of unrelated siblings in the transracial adoptive families were more similar than those of unrelated adolescent siblings pairs reported in other studies. The pattern of IQ correlations for unrelated siblings suggested that familial environmental influences on IQ decline from childhood to late adolescence, but this conclusion was not supported by parent-child IQ correlations. The effects of selective placement on familial IQ correlations were small. Estimates of genetic and familial environmental influences on IQ were very similar to those of other studies. This suggests that the influences on intellectual development in this sample of black/interracial adoptees reared in white families are similar to those for children in the majority populations of the United States and Western Europe.  相似文献   

6.
There is an ongoing debate whether phonological deficits in dyslexics should be attributed to (a) less specified representations of speech sounds, like suggested by studies in young children with a familial risk for dyslexia, or (b) to an impaired access to these phonemic representations, as suggested by studies in adults with dyslexia. These conflicting findings are rooted in between study differences in sample characteristics and/or testing techniques. The current study uses the same multivariate functional MRI (fMRI) approach as previously used in adults with dyslexia to investigate phonemic representations in 30 beginning readers with a familial risk and 24 beginning readers without a familial risk of dyslexia, of whom 20 were later retrospectively classified as dyslexic. Based on fMRI response patterns evoked by listening to different utterances of /bA/ and /dA/ sounds, multivoxel analyses indicate that the underlying activation patterns of the two phonemes were distinct in children with a low family risk but not in children with high family risk. However, no group differences were observed between children that were later classified as typical versus dyslexic readers, regardless of their family risk status, indicating that poor phonemic representations constitute a risk for dyslexia but are not sufficient to result in reading problems. We hypothesize that poor phonemic representations are trait (family risk) and not state (dyslexia) dependent, and that representational deficits only lead to reading difficulties when they are present in conjunction with other neuroanatomical or—functional deficits.  相似文献   

7.
Harvey M  Byrd M 《Adolescence》2000,35(138):345-356
This study examined the relationship between university students' perceptions of their familial attachment and the manner in which their families cope with life's difficulties. It was hypothesized that individuals with high levels of secure attachment would perceive their families as using more active coping strategies (e.g., mobilizing the family to deal with a problem and making efforts to acquire social support). The results supported this hypothesis. Further, individuals with high levels of anxious/ambivalent attachment perceived their families as using a passive appraisal coping strategy, possibly because of a desire to avoid confrontation for fear of disturbing family accord. The findings are discussed in terms of individuals' dispositional coping styles formed as a function of type of familial attachment, as well as situational factors that might serve to mitigate the use of their basic coping strategies.  相似文献   

8.
This project investigated the predictors of Addiction-Prone Personality (APP) scores in youth and young adults from biological (N = 328, 53% female) and adoptive (N = 77, 53% female) families. The development of offspring’s APP traits was examined from three different angles: (1) patterns in biological and adoptive families, (2) offspring’s vs. parent’s perceptions of familial environment, and (3) different points in the life span. The offspring’s APP scores were found to be significantly predicted by parents’ APP scores in both biological and adoptive families. Parents’ APP scores and offspring’s gender consistently showed significant direct influences on offspring’s APP scores in biological families. The familial care factor (maternal and paternal care, family cohesion, and family adaptability) was found to be the consistent significant predictor of offspring’s APP scores in adoptive families even when offspring became older. These results are consistent in showing that the social environment plays an important role in the development of Addiction-Prone Personality traits.  相似文献   

9.
The phenotypic structure of personality traits has been well described, but it has not yet been explained causally. Behavior genetic covariance analyses can identify the underlying causes of phenotypic structure; previous behavior genetic research has suggested that the effects from both genetic and nonshared environmental influences mirror the phenotype. However, nonshared environmental effects are usually estimated as a residualterm that may also include systematic bias, such as that introduced by implicit personality theory. To reduce that bias, we supplemented data from Canadian and German twin studies with cross-observer correlations on the Revised NEO Personality Inventory. The hypothesized five-factor structure was found in both the phenotypic and genetic/familial covariances. When the residual covariance was decomposed into true nonshared environmental influences and method bias, only the latter showed the five-factor structure. True nonshared environmental influences are not structured as genetic influences are, although there was some suggestion that they do affect two personality dimensions, Conscientiousness and Love. These data reaffirm the value of behavior genetic analyses for research on the underlying causes of personality traits.  相似文献   

10.

Elements of military life can create challenges for all family members, including military-connected adolescents, and can have detrimental consequences for their adjustment. Although research with samples of military-connected adolescents has examined the influences of military stressors for adolescent adjustment (e.g., depressive symptoms, anxiety), less research has identified possible mechanisms responsible for these effects, particularly the role of specific familial factors. Drawing from social ecological theory and attachment theory, we examined the associations between military stressors (e.g., parental rank, combat deployments, permanent change of station moves) and self-reported adolescent adjustment (e.g., depressive symptoms, self-efficacy) along with examining adolescents’ perceptions of parent-adolescent relationship quality with both the active duty and civilian parent as a linking mechanism. Using a path analysis, data from 265 Army families were examined to identify the direct and indirect associations between military stressors and adolescent adjustment through parent-adolescent relationship quality. Most military stressors were not significantly related to relationship quality of either parent or indicators of adolescent adjustment. However, parent-adolescent relationship quality with each parent (active duty and civilian parent) was uniquely related to adolescents’ adjustment. Discussion is provided regarding how military stressors and familial factors are conceptualized within the context of military families and implications for future research, family therapy, and policies are suggested.

  相似文献   

11.
This paper presents a multiple-group multivariate hierarchical specification of family problem behaviors across ethnicities using structural equation modeling techniques which explicitly model the individual-level and family-level covariance matrices in familial problem behavior. Analyses were conducted across White and African American ethnic groups. The sample (N = 1,168; 647 White and 521 African Americans) comprised children and their parents from 392 families. In addition to relations between family conflict and deviant behaviors, covariates were included at each level of analysis: neighborhood desirability at the family (between) level, and age and gender at the individual (within) level. At the between level, neighborhood desirability influenced family conflict and family conflict influenced family levels of deviance. At the within level, conflict was significantly related to individual levels of deviance. Discussion focuses on the substantive results as well as the application of multilevel analyses to contextual influences of family problem behavior.  相似文献   

12.
Behavioral and molecular genetic research has established that child cognitive ability and academic performance are substantially heritable, but genetic variation does not account for all of the stratification of cognitive and academic outcomes across families. Which specific contexts and experiences contribute to these shared environmental influences on cognitive ability and academic achievement? Using an ethnically and socioeconomically diverse sample of N = 1728 twins ages 7–20 from the Texas Twin Project, we identified specific measured family, school, and neighborhood socioecological contexts that statistically accounted for latent shared environmental variance in cognitive abilities and academic skills. Composite measures of parent socioeconomic status (SES), school demographic composition, and neighborhood SES accounted for moderate proportions of variation in IQ and achievement. Total variance explained by the multilevel contexts ranged from 15% to 22%. The influence of family SES on IQ and achievement overlapped substantially with the influence of school and neighborhood predictors. Together with race, the measured socioecological contexts explained 100% of shared environmental influences on IQ and approximately 79% of shared environmental influences on both verbal comprehension and reading ability. In contrast, nontrivial proportions of shared environmental variation in math performance were left unexplained. We highlight the potential utility of constructing “polyenvironmental risk scores” in an effort to better predict developmental outcomes and to quantify children's and adolescents’ interrelated networks of experiences. A video abstract of this article can be viewed at: https://youtu.be/77E_DctFsr0  相似文献   

13.
Previous studies have documented the primarily genetic aetiology for the stronger phenotypic covariance between reading disability and ADHD inattention symptoms, compared to hyperactivity-impulsivity symptoms. In this study, we examined to what extent this covariation could be attributed to “generalist genes” shared with general cognitive ability or to “specialist” genes which may specifically underlie processes linking inattention symptoms and reading difficulties. We used multivariate structural equation modeling on IQ, parent and teacher ADHD ratings and parent ratings on reading difficulties from a general population sample of 1312 twins aged 7.9–10.9 years. The covariance between reading difficulties and ADHD inattention symptoms was largely driven by genetic (45%) and child-specific environment (21%) factors not shared with IQ and hyperactivity-impulsivity; only 11% of the covariance was due to genetic effects common with IQ. Aetiological influences shared among all phenotypes explained 47% of the variance in reading difficulties. The current study, using a general population sample, extends previous findings by showing, first, that the shared genetic variability between reading difficulties and ADHD inattention symptoms is largely independent from genes contributing to general cognitive ability and, second, that child-specific environment factors, independent from IQ, also contribute to the covariation between reading difficulties and inattention symptoms.  相似文献   

14.
采用学生阅读能力测试卷和学生阅读状况调查问卷,对随机选取的559名中学生进行调查,并运用结构方程模型探讨了家庭环境影响阅读能力的内在机制。结果发现,家庭环境影响阅读投入的路径系数为0.18,阅读投入影响阅读兴趣的路径系数为0.51,阅读兴趣影响阅读投入的路径系数为0.80,阅读投入影响阅读能力的路径系数为0.46,各路径系数均达到显著水平(p0.05)。结果表明,(1)家庭环境通过阅读投入的完全中介作用影响阅读兴趣;阅读兴趣通过阅读投入的完全中介作用影响阅读成绩;(2)家庭环境对阅读能力的影响是通过阅读投入和阅读兴趣的循环互动作用实现的。  相似文献   

15.
Among factors favoring the addictive behaviors, some specific family processes are often identified in eating disorders and dependences with psychoactive substances. Different models of systemic orientation described the familial disturbances involved in the addictive pathologies emphasizing either the emotional interaction and the processes of differentiation family, either the structure and the level of family cohesion. This article proposes a critical review of the systemic literature on the familial disturbances involved in the families of adolescents with eating disorders or with a substance-dependence. The systemic models (Bowen, Minuchin) are reference values in the understanding of family processes and therapy with families. The observations of these family processes have been confronted to empirical researches integrating different epistemological levels and different methods. The empirical results are heterogenous concerning the different forms of family functioning associated with these addictive pathologies. The empirical studies support the hypothesis of a family dysfunctioning in the different addictive behaviors and demonstrate the relationship between the severity of familial disorders and the severity of the addictive disorders. However, the family configuration described by the typological models is rarely found complete or with a similar intensity between the families. The studies support the hypothesis of an emotional distance in the family interactions and a lack of parental care which could correspond to factors of chronicity or vulnerability. Comparing the family characteristics of these two types of addictive behaviors, the literature supports the existence of (1) some specific family characteristics according to the type of addictive behavior (in particular the conflict avoidance which appears specifically in anorexic families) (2) some similar family patterns of interactions between the members of addictive families in particular the representation of a low family cohesion, an intergenerational enmeschment and a high level of control. These family patterns suggest rather to a dimensional approach of the family characterized by different protective or risk factors. We suggest that these family patterns could constitute, according to their importance (intensity and frequence), a family risk factor of development or aggravation of the addictive behaviors.  相似文献   

16.
Familial clustering of a disease is defined as the occurrence of the disease within some families in excess of what would be expected from the occurrence in the population. It has been demonstrated for several cancer types, ranging from rare cancers as the adenomatosis-coli-associated colon cancer or the Li-Fraumeni syndrome to more common cancers as breast cancer and colon cancer. Familial clustering, however, is merely an epidemiological pattern, and it does not tell whether genetic or environmental causes or both in combination are responsible for the familial clustering. Familial clustering may be due to genetic predisposition to the disease, but exposure to environmental factors — shared by members of some families, but not by members of other families — may also cause familial clustering and hence mimic genetic inheritance in the study of nuclear families. Based on assumptions regarding the individual steps in the biological process starting with exposure to carcinogens and ending with death from disseminated cancer we suggest that genetic and environmental factors may both be involved in most of these steps. The present paper focuses on research methodologies necessary to discriminate between the effect of genes and family environment in the development of cancer.  相似文献   

17.
Severe traumatic brain injury: some effects on family caregivers   总被引:1,自引:0,他引:1  
This study assesses the effects of severe traumatic brain injuries on family members and functioning-a topic of interest for those working with survivors and their families. This issue is receiving increased attention as recent findings suggest that family adjustment influences outcome for brain-injured persons. The Family Environment Scale and the Profile of Mood States were completed by 25 individuals who had a family member with a severe traumatic brain injury. These scales were also completed by a comparison group of 32 individuals who had no brain-injured family member. In terms of family functioning, the findings suggest that, when a family member suffers a severe traumatic brain injury, depression may be elevated, along with a decreased ability to express feelings, decreased time and energy for social and recreational activities, and increased control in comparison to families without a brain-injured member. While this might contribute to family isolation which could last for many years, the overall finding of the present study was that caregiver families were coping adequately.  相似文献   

18.
During the last three years, cognitive data have been reported for 4,639 pairings using the family design, 2,540 pairings using the adoption design, and 2,164 pairs of twins. Together, these extensive data point to less heritable influence on IQ than is indicated by the widely cited older data. The difference between the newer and older data could be due to environmental or genetic secular changes in the population or to methodological differences. Contrary to usual assumptions, the newer data also suggest that more of the environmental influences for parents and offspring and for older siblings operate within families (making family members different from one another) than between families (making family members similar to one another).  相似文献   

19.
In China, acute stigma accompanying an HIV diagnosis can lead to self-isolation. In a cultural setting where family relationships are highly valued and contribute critically to well-being, such self-isolation can thwart HIV self-management and engagement in medical care, and so heighten risk for health disparities. To understand this phenomenon, we conducted individual in-depth interviews with 34 persons living with HIV/AIDS (PLwHA) in Shanghai and Beijing. Inductive content analysis revealed a range of forms of self-isolation motivation, beliefs, and behaviors influenced by: 1) internalized stigma and desire to avoid discrimination; 2) HIV-related factors such as HIV knowledge and disease progression; and 3) familial factors such as a sense of responsibility and family members’ reactions. Based on a proposed framework centering on dialectical family influences (whereby PLwHA are pushed away from, yet pulled toward the family fold), implications for provision of multidisciplinary care in medical settings are considered, including culturally appropriate strategies to decrease health disparities.  相似文献   

20.
This study describes the family aggregation of gyral morphology in the posterior perisylvian region in families that contain one or more children with a developmental language disorder. The probands in these families were 8 male and 2 female children referred through therapy programs and schools for children with language and reading problems. Family members included both biological parents (10 m, 10 f) and all available siblings (6 m, 4 f). Gyral morphology in the members of these families was compared with control subjects (10 m, 10 f) who were without a personal or family history of developmental language disorders. Gyral morphology was evaluated using T1-weighted sagittal scans from a GE Signa 1.5T magnet, 5 mm consecutive slices through the full brain volume. A less common type of Sylvian fissure morphology was more frequently found in the hemispheres of language-disordered subjects and their first-degree relatives than in control subjects. In addition, the pattern of Sylvian fissure morphology across generations within the families suggests that this feature might be inherited from either parent. The elevated rate of extra gyri in the posterior perisylvian region in families affected by language disorder links an anomaly within a language-related brain region with familial risk for this disorder.  相似文献   

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