Levels of Selection Are Artefacts of Different Fitness Temporal Measures

In this paper I argue against the claim, recently put forward by some philosophers of biology and evolutionary biologists, that there can be two or more ontologically distinct levels of selection. I show by comparing the fitness of individuals with that of collectives of individuals in the same environment and over the same period of time – as required to decide if one or more levels of selection is acting in a population – that the selection of collectives is a by‐product of selection at the individual level; thus, talking about two or more levels of selection represents merely a different perspective on one and the same process. 1     

DEVIANCE AND CAUSALISM

Drawing on the problem of deviance, I present a novel line of argumentation against causal theories of action. The causalist faces a dilemma: either she adopts a simple account of the causal route between intention and outcome, at the cost of failing to rule out deviance cases, or she adopts a more sophisticated account, at the cost of ruling out cases of intentional action in which the causal route is merely unusual. Underlying this dilemma, I argue, is that the agent's perspective plays an ineliminable role in determining which causal pathways are deviant and which are not.     

Driftability

In this paper, I argue (contra some recent philosophical work) that an objective distinction between natural selection and drift can be drawn. I draw this distinction by conceiving of drift, in the most fundamental sense, as an individual-level phenomenon. This goes against some other attempts to distinguish selection from drift, which have argued either that drift is a population-level process or that it is a population-level product. Instead of identifying drift with population-level features, the account introduced here can explain these population-level features based on a property that I label driftability. Additionally, this account shows that biology’s “first law”—the Principle of Drift (Brandon, J Phil 102(7):319–335 2006)—is not a foundational law, but is a consequence of driftability.     

REASONS, CAUSES, AND CONTRASTS

Abstract:   The standard argument for the causal theory of action is "Davidson's Challenge": explain the connection between reasons and actions without appealing to the idea that reasons cause actions. I argue that this is an argument to the best contrastive explanation. After examining the nature of contrastive explanation in detail, I show that the causalist does not yet have the best explanation. The best explanation would appeal further to the motivational strength of reasons. Finally, I show how this undermines the argument for causalism, since noncausalists, too, can meet Davidson's Challenge by appealing to motivational strength to explain the cases at issue.     

Natural Selection: A Case for the Counterfactual Approach

This paper investigates the conception of causation required in order to make sense of natural selection as a causal explanation of changes in traits or allele frequencies. It claims that under a counterfactual account of causation, natural selection is constituted by the causal relevance of traits and alleles to the variation in traits and alleles frequencies. The “statisticalist” view of selection (Walsh, Matthen, Ariew, Lewens) has shown that natural selection is not a cause superadded to the causal interactions between individual organisms. It also claimed that the only causation at work is those aggregated individual interactions, natural selection being only predictive and explanatory, but it is implicitly committed to a process-view of causation. I formulate a counterfactual construal of the causal statements underlying selectionist explanations, and show that they hold because of the reference they make to ecological reliable factors. Considering case studies, I argue that this counterfactual view of causal relevance proper to natural selection captures more salient features of evolutionary explanations than the statisticalist view, and especially makes sense of the difference between selection and drift. I eventually establish equivalence between causal relevance of traits and natural selection itself as a cause.     

Aristotle's Four Causes of Action

Aristotle's typical procedure is to identify four causes of natural changes. Intentional action, a natural change, has standardly been treated as an exception: most think that Aristotle has the standard causalist account, according to which an intentional action is a bodily movement efficiently caused by an attitude of the appropriate sort. I show that action is not an exception to Aristotle's typical procedure: he has the resources to specify four causes of action, and thus to articulate a powerful theory of action unlike any other on offer.     

When do adaptive developmental mechanisms yield maladaptive outcomes?

This article discusses 3 ways in which adaptive developmental mechanisms may produce maladaptive outcomes. First, natural selection may favor risky strategies that enhance fitness on average but which have detrimental consequences for a subset of individuals. Second, mismatch may result when organisms experience environmental change during ontogeny, for instance, because they move from one environment to another. Third, organisms may learn about their environment in order to develop an appropriate phenotype; when cues indicate the environmental state probabilistically, as opposed to deterministically, sampling processes may produce mismatch. For each source of maladaptation, we present a selection of the relevant empirical research and illustrate how models from evolutionary biology can be used to make predictions about maladaptation. We also discuss what data can be collected to test these models in humans. Our goal is to show that evolutionary approaches not only yield insights into adaptive outcomes but can also illuminate the conditions leading to maladaptation. This perspective provides additional nuance to the dialectic between the developmental psychopathology model and evolutionary developmental psychology.     

Sufficiency and Necessity Assumptions in Causal Structure Induction

Research on human causal induction has shown that people have general prior assumptions about causal strength and about how causes interact with the background. We propose that these prior assumptions about the parameters of causal systems do not only manifest themselves in estimations of causal strength or the selection of causes but also when deciding between alternative causal structures. In three experiments, we requested subjects to choose which of two observable variables was the cause and which the effect. We found strong evidence that learners have interindividually variable but intraindividually stable priors about causal parameters that express a preference for causal determinism (sufficiency or necessity; Experiment 1). These priors predict which structure subjects preferentially select. The priors can be manipulated experimentally (Experiment 2) and appear to be domain‐general (Experiment 3). Heuristic strategies of structure induction are suggested that can be viewed as simplified implementations of the priors.     

Adaptive Memory: Remembering With a Stone-Age Brain

ABSTRACT— If memory evolved, sculpted by the processes of natural selection, then its operating characteristics likely bear the "footprints" of ancestral selection pressures. Psychologists rarely consider this possibility and generally ignore functional questions in their attempt to understand how human memory works. We propose that memory evolved to enhance reproductive fitness and, accordingly, its systems are tuned to retain information that is fitness-relevant. We present evidence consistent with this proposal, namely that processing information for its survival relevance leads to superior long-term retention—better, in fact, than most known memory-enhancement techniques. Even if one remains skeptical about evolutionary analyses, adopting a functional perspective can lead to the generation of new research ideas.     

Negative causation in causal and mechanistic explanation

Instances of negative causation—preventions, omissions, and the like—have long created philosophical worries. In this paper, I argue that concerns about negative causation can be addressed in the context of causal explanation generally, and mechanistic explanation specifically. The gravest concern about negative causation is that it exacerbates the problem of causal promiscuity—that is, the problem that arises when a particular account of causation identifies too many causes for a particular effect. In the explanatory context, the problem of promiscuity can be solved by characterizing the phenomenon to be explained as a contrast between two or more events or non-events. This contrastive strategy also can solve other problems that negative causation presents for the leading accounts of mechanistic explanation. Along the way, I argue that to be effective, accounts of causal explanation must incorporate negative causation. I also develop a taxonomy of negative causation and incorporate each variety of negative causation into the leading accounts of mechanistic explanation.     

An Inferential Model of Scientific Understanding

In this article I argue that two current accounts of scientific understanding are incorrect and I propose an alternative theory. My new account draws on recent research in cognitive psychology which reveals the importance of making causal and logical inferences on the basis of incoming information. To understand a phenomenon we need to make particular kinds of inferences concerning the explanations we are given. Specifically, we come to understand a phenomenon scientifically by developing mental models that incorporate the correct causal and logical properties responsible for the causes or logical properties of the phenomenon being explained.     

How two causes are different from one: The use of (un)conditional information in Simpson's paradox

In a causally complex world, two (or more) factors may simultaneously be potential causes of an effect. To evaluate the causal efficacy of a factor, the alternative factors must be controlled for (or conditionalized on). Subjects judged the causal strength of two potential causes of an effect that covaried with each other, thereby setting up a Simpson's paradox--a situation in which causal judgments should vary widely depending on whether or not they are conditionalized on the alternative potential cause. In Experiments 1 (table format) and 2 (trial-by-trial format), the subjects did conditionalize their judgments for one causal factor on a known alternative cause. The subjects also demonstrated that they knew what information was needed to properly make causal judgments when two potential causes are available. In Experiment 3 (trial-by-trial), those subjects who were not told about the causal mechanism by which the alternative cause operated were less likely to conditionalize on it. However, the more a subject recognized the covariation between the alternative cause and the effect, the more the subject conditionalized on it. Such behavior may arise from the interaction between bottom-up and top-down processing.     

Causal Necessity in Aristotle

Like many realists about causation and causal powers, Aristotle uses the language of necessity when discussing causation, and he appears to think that by invoking necessity, he is clarifying the manner in which causes bring about or determine their effects. In so doing, he would appear to run afoul of Humean criticisms of the notion of a necessary connection between cause and effect. The claim that causes necessitate their effects may be understood— or attacked— in several ways, however, and so whether the view or its criticism is tenable depends on how we understand the necessitation claim. In fact, Aristotelian efficient causation may be said to involve two distinct necessary connections: one is a relation between causes considered as potential, while the other relates them considered as active. That is, the claims that (1) what has the power to heat necessarily heats what has the power to be heated, and that (2) a particular flame which is actually under a pot necessarily heats it, both of which appear to be true for Aristotle, involve distinct notions of necessity. The latter kind of necessity is based on the facts, as Aristotle sees them, about change, whereas the former is based in the nature of properties. Though different, both kinds of necessity are instances of what contemporary philosophers would call metaphysical necessity, and together they also amount to a theory of causal determination.     

Causal uncertainty as a chronically accessible construct

Causal uncertainty refers to feelings that one may not understand the causes of events. A number of studies have shown that causal uncertainty has significant effects on people's processing of information and on important life outcomes. Weary and Edwards have postulated that causal uncertainty is, in part, a cognitive construct that can vary in its accessibility. This assumption has allowed for a variety of predictions to be made about the causes and consequences of causal uncertainty. However, this accessibility assumption has never been directly tested. To do this, in Study 1 an emotional Stroop procedure was used. Higher causal uncertainty was associated with longer latencies to name the color in which causal uncertainty‐related words were written compared to uncertainty‐irrelevant words. In Study 2, both manipulated and chronic causal uncertainty led to faster times to respond to causal uncertainty‐related stimuli in an attitude accessibility task. Both studies are consistent with the theoretically predicted chronic accessibility of causal uncertainty beliefs. Copyright © 2008 John Wiley & Sons, Ltd.     

The World as Representation: Schopenhauer's Arguments for Transcendental Idealism

In this paper, I discuss Spinoza's conception of the cosmological argument for the existence of God (hereafter CA), as it can be reconstructed taking departure from Letter 12. By a CA, I understand, quite broadly, an argument which infers a posteriori the existence of a necessary being, usually identified as God, from the experience that there exists some other being, often oneself, the existence of which is contingent upon the existence of this necessary being. The difference between various versions of the CA stems from differences in the way in which the relation of existential dependence is understood. The aim of this paper is to reconstruct a version of the CA which would be valid on Spinoza's principles. I argue that such a version must be understood in the context of Spinoza's causal rationalism. By such causal rationalism, I mean a doctrine committed to the idea that everything is rational and thus explicable and that causal explanations are somehow fundamental. Moreover, I hold that, in Spinoza's causal rationalism, there is one form of causation through which all other types of causation are ultimately understood. This ‘archetype’ of causation is self-causation.     

经验信息与共变信息对判断两原因共同作用的影响

本研究考察了被试在使用经验信息和共变信息进行两原因共同作用因果判断时所具有的特点。研究结果表明:(1)两原因的性质对于被试的选择有着显著影响。当两原因的可信度相等时,更多被试认为两原因都可以引起结果。(2)两原因都不出现时结果出现的概率P(e/~i~j)对于被试的选择没有显著影响。(3)两原因单独出现时结果出现的概率对于被试的选择有着显著的影响,但是这一变量是与两原因的可信程度共同产生影响的。     

QUANTUM ASPECTS OF LIFE: RELATING EVOLUTIONARY BIOLOGY WITH THEOLOGY VIA MODERN PHYSICS

Abstract In the present paper, I shall argue that quantum theory can contribute to reconciling evolutionary biology with the creation hypothesis. After giving a careful definition of the theological problem, I will, in a first step, formulate necessary conditions for the compatibility of evolutionary theory and the creation hypothesis. In a second step, I will show how quantum theory can contribute to fulfilling these conditions. More precisely, I claim that (1) quantum probabilities are best understood in terms of ontological indeterminism, but (2) reflect nevertheless causal openness rather than divine indifference or arbitrariness, and (3) such a genuinely creative universe can be considered as the work of a loving Creator. I ask subsequently whether these necessary conditions are also sufficient for the compatibility of evolutionary theory and the creation hypothesis. Finally, I will show that relating evolutionary biology with theology via quantum theory could also shed some light on the nature of life.     

Frankfurt versus Frankfurt: a new anti-causalist dawn

There is an important anomaly to the causalist/compatibilist paradigm in the philosophy of action and free will. This anomaly, which to my knowledge has gone unnoticed so far, can be found in the philosophy of Harry Frankfurt. Two of his most important contributions to the field – his influential counterexample to the Principle of Alternate Possibilities and his ‘guidance’ view of action – are incompatible. Frankfurt's counterexample to the Principle works only if we do not understand action as Frankfurt does in his guidance account. If, on the other hand, we understand agency in terms of the agent's guidance, then his counterexample to the Principle fails because, then, counterfactual scenarios of Frankfurt-type counterexamples are such that what happens does not count as the relevant agent's action. So Frankfurt-type counterexamples do not show that the agent could not have avoided acting as she did: so they fail to offer a scenario in which the agent is intuitively responsible even though she could not have avoided acting as she did. Therefore, Frankfurt-type counterexamples do not challenge the Principle, according to which ‘a person is morally responsible for what he has done only if he could have done otherwise’. The importance of this inconsistency goes far beyond the issue of coherence within Frankfurt's philosophy. I shall argue that this inconsistency represents an important anomaly within the causalist/compatibilist framework; so that we should start to seriously consider having to move on from the established paradigm.