Persistence of learned helplessness in humans

The duration of the effects of a common learned helplessness induction procedure, exposure to insoluble concept-formation problems, was assessed by varying the interval between the induction procedure and subsequent exposure to soluble anagrams. Participants tested immediately or 30 min after the induction procedure exhibited reliable helplessness deficits on all dependent measures. These effects, relative to the performance of a nonhelpless control group, were absent in subjects who experienced delays of 2 or 6 hr before anagram testing. The implications of these results for the development of more enduring helplessness effects and for the conducting of research into analogue intervention are discussed.     

The debilitating effect of exposure to noncontingent escape: A test of the learned helplessness model

Two studies were conducted to test the hypothesis that exposure to non-contingent escape leads to performance deficits similar to those observed when subjects are exposed to noncontingent aversive outcomes from which there is no escape, and that causal attributions mediate these deficits. Previous attempts to produce “appetitive helplessness” (deficits resulting from exposure to noncontingent positive events) have been plagued by subjects' tendency to believe that they are responsible for positive events. In Experiment 1, 40 subjects were exposed to contingent or noncontingent noise escape trials. As predicted by the learned helplessness model, subjects who received inescapable noise performed less well on a subsequent anagram task than subjects exposed to escapable noise. Similarly, subjects who escaped from the noise owing to the benevolence of a powerful other rather than because of their own efforts, showed performance deficits paralleling those of the inescapable noise subjects. In Experiment 2, subjects who escaped an aversive tone through no effort of their own showed subsequent performance deficits, but globality of their self-reported attributions did not predict subsequent anagram performance. The results of these studies provide support for the hypothesis that uncontrollability, independent of the valence of a particular outcome, is responsible for helplessness deficits, but do not support the mediational role of attributions, at least in the laboratory.     

Effects of amount of helplessness training and internal-external locus of control on mood and performance.

Based on Wortman and Brehm's integration of reactance theory with Seligman's model of learned helplessness, an investigation was conducted to examine the effects of amount of helplessness training and internal--external locus of control on subsequent task performance and on self-ratings of mood. Subjects were divided into "internal" and "external" groups and were then given either high, low, or no helplessness training on a series of concept-formation problems. After completing a mood checklist, all subjects worked on an anagram task presented as a second experiment by a second experimenter. The results revealed that internals exhibited greater performance decrements and reported greater depression under high helplessness than did externals. In the low helplessness conditions, internals tended to perform better than control subjects, while externals tended to perform worse than control subjects; low helplessness subjects also reported the highest levels of hostility. The results are discussed within the context of Wortman and Brehm's integration of reactance and learned helplessness theories.     

Action-oriented versus state-oriented reactions to experimenter-induced failures

The present study assessed different effects of action-oriented versus state-oriented styles of coping with failure on achievement-related performance and cognition. In a learned helplessness experiment, students were exposed to an academic failure situation and were then tested on a series of problem-solving tasks, either immediately after the pretreatment or after a delay of 24 hours. Performance and cognitive concomitants were measured during both experimental periods. Results demonstrated that action orientation was associated with self-immunizing cognitions during helplessness training. Action-oriented participants improved their performance level even after repeated failure feedbacks. Moreover, action-oriented students assigned to the delayed test condition responded with increased striving for success and showed performance increments, even in comparison with control subjects. In contrast, state-oriented participants developed symptoms of helplessness and showed impaired performance during failure inductions. In later tests on problem-solving tasks, state-oriented groups responded with increased fear of failure. Independent of immediate or delayed test conditions, they soon lapsed into new performance decrements.     

Learned helplessness, depression, and the attribution of failure.

Depressed and nondepressed college students received experience with solvable, unsolvable, or no discrimination problems. When later tested on a series of patterned anagrams, depressed groups performed worse than nondepressed groups, and unsolvable groups performed worse than solvable and control groups. As predicted by the learned helplessness model of depression, nondepressed subjects given unsolvable problems showed anagram deficits parallel to those found in naturally occurring depression. When depressed subjects attributed their failure to the difficulty of the problems rather than to their own incompetence, performance improved strikingly. So, failure in itself is apparently not sufficient to produce helplessness deficits in man, but failure that leads to a decreased belief in personal competence is sufficient.     

The effect of debriefing on laboratory induced helplessness: An attributional analysis1

Debriefing was assessed as means of reversing helplessness deficits through reattribution. Fifty-five subjects listened to escapable or inescapable tones. One inescapable group, prior to anagrams, was debriefed regarding noise uncontrollability. A second inescapable group was administered anagrams by a different experimenter. While exposure to inescapable noise led to performance deficits, switching experimenters obviated these deficits. Debriefing actually facilitated anagram performance. All inescapable subjects—debriefed or not—attributed their lack of control on the noise task to experimenter interference, casting some doubt on reattribution as an explanation of debriefing's effects. Results were discussed in terms of the reformulated learned helplessness model and the ethical implications of debriefing in learned helplessness research.     

Alcohol consumption by college women following exposure to unsolvable problems: Learned helplessness or stress induced drinking?

Three experiments were intended to examine the relationship between alcohol, depression and learned helplessness. In Study I, more female undergraduate heavy drinkers than light drinkers were found to have critically elevated scores on a depression index. A sex difference in the relationship between drinking and depression is suggested since male depressives were equally distributed among drinking categories. In Studies II and III, female undergraduates were given unsolvable problems in a learned helplessness paradigm. Relative to controls these subjects reported increased depressive and hostile affect and drank more beer in a taste rating task. However, we failed to find deficits in anagram solution with those subjects given the learned helplessness manipulation. Specific questions are raised regarding the boundary conditions of learned helplessness while implications bearing on stress-related alcohol consumption are discussed.     

Learned helplessness versus reactance: The effects of sex-role stereotypy

Both learned helplessness and reactance theories hypothesize that the effects of noncontingent reinforcement on later performance are related to the amount of experience with noncontingent reinforcement and to the subjects' expectations of control. In addition, learned helplessness theorists have suggested that performance may depend upon the causes to which subjects ascribe failure. The present study investigated these hypotheses by defining expectation of control as the degree of sex-role stereotypy and by assessing causal attributions. Forty men and 40 women were given either zero, three, four, five, or six discrimination problems for which they received noncontingent reinforcement; they were subsequently tested on anagrams and math problems. Causal attributions were rated after each set of tasks. The data suggested the following. (1) In general, under conditions of noncontingency, high masculinity subjects performed better on anagrams and low masculinity subjects performed worse on anagrams than subjects in the control conditions; stereotypic femininity was not related to performance. (2) Ratings of attributions for failing the discrimination problems were generally unrelated to performance, although there was weak support for the facilitating effects of effort attributions. (3) Subsequent to anagram and math performance, women rated external attributions higher following success and internal attributions higher following failure than did men. The implications for learned helplessness and reactance theories are discussed.The authors would like to thank Stephen Haynes and Jack McKillip for their helpful comments on earlier versions of this paper.     

Effects of learned helplessness training on instrumental heart rate control in humans

In Experiment 1, subjects who received feedback contingent on short interbeat intervals (relative to a baseline period) learned to accelerate their heart rates, but subjects who received noncontingent feedback did not. In Experiment 2, subjects who were exposed to noncontingent aversive noises later showed significant performance deficits on both an instrumental and a cognitive task. Attributional style predicted helplessness deficits on the cognitive but not the instrumental task. Experiment 3 demonstrated that experimentally induced helplessness interferes with biofeedback learning. Attributional style did not predict the occurrence of helplessness deficits in this context. Results are discussed in terms of the nature of biofeedback training and the range of behaviors that learned helplessness training affects.     

Sensitive maintenance: a cognitive process underlying individual differences in memory for threatening information

Dispositional styles of coping with threat influence memory for threatening information. In particular, sensitizers excel over repressors in their memory for threatening information after long retention intervals, but not after short ones. We therefore suggested that sensitizers, but not repressors, employ active maintenance processes during the retention interval to selectively retain threatening material. Sensitive maintenance was studied in 2 experiments in which participants were briefly exposed to threatening and nonthreatening pictures (Experiment 1, N = 128) or words (Experiment 2, N = 145). Following, we administered unannounced recognition tests before and after an intervening task that generated either high or low cognitive load, assuming that high cognitive load would impede sensitizers' memory maintenance of threatening material. Supporting our hypotheses, the same pattern of results was obtained in both experiments: Under low cognitive load, sensitizers forgot less threat material than repressors did; no such differences were observed under high cognitive load.     

Learned helplessness and the detection of contingency: A direct test

Two studies tested a basic hypothesis of the learned helplessness model: That performance deficits associated with exposure to uncontrollable outcomes are directly mediated by an individual's perception of response-outcome independence. In the first experiment 48 subjects were exposed to noise bursts. For one experimental group, the termination of the noise was response-contingent. For five other groups, noise-burst termination was independent of subjects' responses. These five groups varied in the number of trials on which they received positive feedback: As predicted, subjects overestimated the amount of control they had over noise termination as a positive linear function of the amount of noncontingent positive feedback they received. Although subjects exposed to either noncontingent positive or negative feedback showed subsequent performance deficits on an anagrams task, the expected relation between perceived control and subsequent performance failed to emerge. These findings were replicated in a second experiment. In addition, subjects' locus, stability, and globality attributions failed to predict subsequent performance. These results call into question the central premises of helplessness theory: That perceived uncontrollability and causal attributions mediate learned helplessness.     

Vicariously and directly learned helplessness and effectiveness1

Experiment 1 demonstrated the typical helplessness effect of inescapability on directly participating female subjects. Predictions drawn from social learning theory and comparison level theory on vicariously learned helplessness and effectiveness were then tested in a second study employing the prototypical learned helplessness induction design. In Experiment 2, 180 women served as either observers or direct participants and were pretreated as dyads with escapable, inescapable, or irrelevant noise. Consistent with social learning theory, subsequent instrumental task performance demonstrated facilitation effects of escapability on observers, and the same effect on direct participants also was found. No debilitation effects of vicariously or directly experienced inescapability were obtained. The combined results suggest that the induction of helplessness and effectiveness may depend on the social context in which relative controllability is operative, and are discussed primarily in terms of possible coaction effects.     

Learned helplessness and learned resourcefulness: effects of noncontingent success and failure on individuals differing in self-control skills

We designed two experiments to investigate the role of self-control processes in learned-helplessness studies by assessing the differential reactions to uncontrollability of subjects who presumably had either a rich (high resourceful, or HR) or poor (low resourceful, or LR) repertoire of self-control skills. HR and LR subjects received noncontingent success feedback, failure feedback, or no feedback on a task that ostensibly assessed "therapeutic abilities." Subjects were subsequently tested on insolvable puzzles (Experiment 1) or on solvable anagrams (Experiment 2). According to Kanfer and Hagerman's (1981) self-regulation model, self-regulatory activities are evoked primarily in situations in which subjects are faced with repeated failure. Hence we predicted that individual differences in self-control would influence performance on the insolvable puzzles and not anagram performance after exposure to noncontingent failure. This prediction was confirmed: Only the performance of LR subjects on the insolvable puzzles was debilitated by the helplessness induction, whereas HR and LR subjects showed equal helplessness-induced deficits on the anagrams. The latter finding was interpreted in terms of the learned-helplessness model without the mediating effects of self-regulatory processes. As predicted from the self-control model, HR subjects more frequently checked statements indicating positive self-evaluations and task-oriented thoughts and less frequently checked negative self-evaluations than did LR subjects during exposure to uncontrollability in both experiments. We concluded that the self-control model accounts best for subjects' self-reactions during exposure to uncontrollability or failure, whereas the learned-helplessness model accounts for the generalization of helplessness from uncontrollable situations to controllable ones.     

Learned helplessness and judgments of control

One of the central hypotheses of learned helplessness theory is that exposure to noncontingency produces a reduced ability to perceive response-outcome relations (the postulated "cognitive deficit"). To test this hypothesis, subjects were exposed to a typical helplessness induction task and then asked to make judgments of the amount of control their responses exerted over a designated outcome (the onset of a light). Support for the postulated cognitive deficit would be found if subjects who experienced the induction underestimated the relation between their responses and outcomes. The results, however, demonstrated that induction subjects (n = 30) made higher and more accurate judgments of control than subjects in a no-treatment control group (n = 30). This finding clearly fails to support the postulated cognitive deficit and highlights the need for other direct tests of the basic hypotheses of helplessness theory.     

Male-Female Performance in Learned Helplessness

The impact of uncontrollability and failure on adult males and females in a learned helplessness situation was investigated. It was hypothesized that females would show more helplessness effects. Twenty-four males and 24 females enrolled in a university psychology course were studied. Helplessness was induced through a series of anagram tasks used in earlier research by one of the authors. As expected, females showed more helplessness effects in the helplessness-induction manipulations of the anagram tasks. Discussion is offered in terms of possible sex differences in childhood training with regard to handling frustration and failure.     

Predictable long-delay matching-to-sample trials result in long-latency sample responding by pigeons

When a delayed matching-to-sample task does not involve short-delay trials interspersed among the long-delay trials, there is evidence that pigeons show long latencies in responding to the sample (sample observing response). They may even stop responding altogether. Two experiments were conducted to confirm the phenomenon and determine what aspect of the procedure is responsible for it. In Experiment 1 pigeons were trained on a delayed matching-to-sample task in which the delay remined constant throughout each session and was increased as each bird reached a performance criterion. The results indicated that sampleresponse latency increased in a regular fashion as delay increased. When the delay increased to 8.0 s, responding to the sample often stopped for long periods of time. In Experiment 2 pigeons were exposed to constant delay sessions (LONG), or mixed delay sessions with the delay length signaled (COR) or unsignaled (UNC). Sample-response latencies increased with increasing delay on long-delay trials for the LONG and the COR groups but not for the UNC group. Results supported the hypothesis that predictability of the long delay and not the loss of association between sample responses and reinforcement was responsible for the increased sample-response latency.     

Repeated failure: a source of helplessness or a factor irrelevant to its emergence?

We tested the validity of the egotism model of human helplessness. In contrast to the original theoretical approach of Seligman and his associates, which points to response-outcome noncontingency as the main source of helplessness, the egotism alternative proposes that repeated failure itself is the critical determinant of helplessness symptoms. Repeated failure threatens the self-esteem of the subject, who supposedly engages in a least-effort strategy during the test phase of a typical learned helplessness study, which results in performance impairment. To examine the egotism explanation, we gave subjects noncontingent-feedback training with or without repeated failure on five consecutive discrimination problems. In two experiments, noncontingent-feedback preexposure produced helplessness deficits in performance on avoidance learning, whereas repeated failure appeared irrelevant to helplessness. This and our other findings from research are inconsistent with the egotism explanation and support instead Seligman's original proposal, in which helplessness is attributed to prolonged experience with noncontingency.     

A case study of three theories of learned helplessness: The role of test importance

The current study tests three alternative explanations (learned helplessness, cognitive interference, and egotism) for poor performance following unsolvable problems. In Experiment 1, subjects were exposed to no feedback or to failure in unsolvable problems and were further divided according to the importance of a test task (unstipulated, low, and high importance). In Experiment 2, during the training phase subjects were exposed to either no feedback, failure, or failure plus explicit hypothesis instructions. Then, subjects in each group received either low or high test-importance instructions. Results bring support to the cognitive interference explanation of performance deficits. Exposure to unsolvable problems was found to impair performance in a high importance task, but not in a low importance task. Such a deleterious effect of prior failure and high importance instructions was reversed by discouraging people from engaging in state-oriented actions. The theoretical implications of the findings were discussed.     

The conceptualization of helplessness: II. Laboratory correlates of the phenomenological definition of helplessness

The present research was designed to investigate the correspondence between the phenomenological definition of helplessness and its laboratory analogue—the learned helplessness paradigm. In experiment 1, subjects were exposed to unsolvable, solvable, or no problems. It was found that exposure to unsolvable problems increased the report of helplessness feelings and impaired subsequent performance. In addition, experiment 1 demonstrated a negative and significant correlation between performance and the report of helplessness feelings. Experiment 2 isolated the cognitive component of helplessness by measuring the amount of expectancy changes following success and failure. The amount of expectancy changes was negatively correlated with the belief in an outcome's uncontrollability. Finally, experiment 3 showed that exposure to only one unsolvable problem was associated with the reports of coping and anger feelings, whereas exposure to four unsolvable problems was related to surrender feelings. These results demonstrated that laboratory-induced helpless situations elicit the same feelings as real-life helplessness situations.The study was conducted by the first author in partial fulfillment of the requirements for the M.A. degree at Bar Ilan University under the supervision of Prof. R. E. Lubow.     

Effects of stress and hippocampal NMDA receptor antagonism on recognition memory in rats

Exposures to uncontrollable stress have been shown to alter ensuing synaptic plasticity in the hippocampus and interfere with hippocampal-dependent spatial memory in rats. The present study examined whether stress, which impairs hippocampal long-term potentiation (LTP), also affects (nonspatial) hippocampal-dependent object-recognition memory, as tested on the visual paired comparison task (VPC) in rats. After undergoing an inescapable restraint–tailshock stress experience, rats exhibited markedly impaired recognition memory at the 3-h (long) familiarization-to-test phase delay but not at the 5-min (short) delay. In contrast, unstressed control animals showed robust recognition memory (i.e., they exhibited reliable preferences for novel over familiar objects) at both short- and long-delay periods. The impairing effect of stress on long-delay recognition memory was transient because 48 h after undergoing stress experience, animals performed normally at the long delay. Similar to stress, microinfusions of DL-2-amino-5-phosphonovaleric acid (APV), a competitive N-methyl-D-aspartate receptor (NMDAR) antagonist that blocks LTP, into the dorsal hippocampus selectively impaired object-recognition memory at the long-delay period. Together, these results suggest that stress and intrahippocampal administration of APV affect recognition memory by influencing synaptic plasticity in the hippocampus.

[The following individuals kindly provided reagents, samples, or unpublished information as indicated in the paper H. Blair.]