The power of causal beliefs and conflicting evidence on causal judgments and decision making

In two experiments, we investigated the relative impact of causal beliefs and empirical evidence on both decision making and causal judgments, and whether this relative impact could be altered by previous experience. Participants had to decide which of two alternatives would attain a higher outcome on the basis of four cues. After completing the decision task, they were asked to estimate to what extent each cue was a reliable cause of the outcome. Participants were provided with instructions that causally related two of the cues to the outcome, whereas they received neutral information about the other two cues. Two of the four cues—a causal and a neutral cue—had high validity and were both generative. The remaining two cues had low validity, and were generative in Experiment 1, but almost not related to the outcome in Experiment 2. Selected groups of participants in both experiments received pre-training with either causal or neutral cues, or no pre-training was provided. Results revealed that the impact of causal beliefs and empirical evidence depends on both the experienced pre-training and cue validity. When all cues were generative and participants received pre-training with causal cues, they mostly relied on their causal beliefs, whereas they relied on empirical evidence when they received pre-training with neutral cues. In contrast, when some of the cues were almost not related to the outcome, participants’ responses were primarily influenced by validity and—to a lesser extent—by causal beliefs. In either case, however, the influence of causal beliefs was higher in causal judgments than in decision making. While current theoretical approaches in causal learning focus either on the effect of causal beliefs or empirical evidence, the present research shows that both factors are required to explain the flexibility involved in human inferences.     

Summarizing information by means of causal sentences through causal graphs

The objective of this work is to propose a complete system able to extract causal sentences from a set of text documents, select the causal sentences contained, create a causal graph in base to a given concept using as source these causal sentences, and finally produce a text summary gathering all the information connected by means of this causal graph. This procedure has three main steps. The first one is focused in the extraction, filtering and selection of those causal sentences that could have relevant information for the system. The second one is focused on the composition of a suitable causal graph, removing redundant information and solving ambiguity problems. The third step is a procedure able to read the causal graph to compose a suitable answer to a proposed causal question by summarizing the information contained in it.     

Two metamodels of causal effects

Two metamodels, termed Model S and Model V, are proposed for definition, measurement, and generalization of quantitative causal effects. The effect is defined as a part change in score in Model S and as a part change in variance in Model V. Two additional changes, total and remainder change, are defined. The latter is due to all other factors or variables than the cause, while total change is the sum of remainder and effect change. Furthermore, it is shown how contrafactual concepts, which imply that some parts of the study situation are supposed to be otherwise, enter into the metamodels. Casual effects are defined and measured in terms of non-contrafactual concepts, except that statistical induction includes contrafactual as well as non-contrafactual inferences. Non-statistical generalization involves both kinds of inferences. Contrafactual definitions are considered inadequate, and a contrafactual interpretation of statistical adjustment is unnecessary and should be replaced by a non-contrafactual one.     

Non-bayesian inference: causal structure trumps correlation

The study tests the hypothesis that conditional probability judgments can be influenced by causal links between the target event and the evidence even when the statistical relations among variables are held constant. Three experiments varied the causal structure relating three variables and found that (a) the target event was perceived as more probable when it was linked to evidence by a causal chain than when both variables shared a common cause; (b) predictive chains in which evidence is a cause of the hypothesis gave rise to higher judgments than diagnostic chains in which evidence is an effect of the hypothesis; and (c) direct chains gave rise to higher judgments than indirect chains. A Bayesian learning model was applied to our data but failed to explain them. An explanation-based hypothesis stating that statistical information will affect judgments only to the extent that it changes beliefs about causal structure is consistent with the results.     

The development of causal categorization

Two experiments examined the impact of causal relations between features on categorization in 5- to 6-year-old children and adults. Participants learned artificial categories containing instances with causally related features and noncausal features. They then selected the most likely category member from a series of novel test pairs. Classification patterns and logistic regression were used to diagnose the presence of independent effects of causal coherence, causal status, and relational centrality. Adult classification was driven primarily by coherence when causal links were deterministic (Experiment 1) but showed additional influences of causal status when links were probabilistic (Experiment 2). Children's classification was based primarily on causal coherence in both cases. There was no effect of relational centrality in either age group. These results suggest that the generative model (Rehder, 2003a) provides a good account of causal categorization in children as well as adults.     

The mental representation of causal conditional reasoning: mental models or causal models

In this paper, two experiments are reported investigating the nature of the cognitive representations underlying causal conditional reasoning performance. The predictions of causal and logical interpretations of the conditional diverge sharply when inferences involving pairs of conditionals—such as if P₁then Q and if P₂then Q—are considered. From a causal perspective, the causal direction of these conditionals is critical: are the P_icauses of Q; or symptoms caused byQ. The rich variety of inference patterns can naturally be modelled by Bayesian networks. A pair of causal conditionals where Q is an effect corresponds to a “collider” structure where the two causes (P_i) converge on a common effect. In contrast, a pair of causal conditionals where Q is a cause corresponds to a network where two effects (P_i) diverge from a common cause. Very different predictions are made by fully explicit or initial mental models interpretations. These predictions were tested in two experiments, each of which yielded data most consistent with causal model theory, rather than with mental models.     

Causal judgements about two causal candidates: Accounting for occurrences,estimating strength,and the importance of interaction judgements

When two possible causes of an outcome are under consideration, contingency information concerns each possible combination of presence and absence of the two causes with occurrences and nonoccurrences of the outcome. White (2008) proposed that such judgements could be predicted by a weighted averaging model integrating these kinds of contingency information. The weights in the model are derived from the hypothesis that causal judgements seek to meet two main aims, accounting for occurrences of the outcome and estimating the strengths of the causes. Here it is shown that the model can explain many but not all relevant published findings. The remainder can be explained by reasoning about interactions between the two causes, by scenario-specific effects, and by variations in cell weight depending on quantity of available information. An experiment is reported that supports this argument. The review and experimental results support the case for a cognitive model of causal judgement in which different kinds of contingency information are utilised to satisfy particular aims of the judgement process.     

Category transfer in sequential causal learning: the unbroken mechanism hypothesis

The goal of the present set of studies is to explore the boundary conditions of category transfer in causal learning. Previous research has shown that people are capable of inducing categories based on causal learning input, and they often transfer these categories to new causal learning tasks. However, occasionally learners abandon the learned categories and induce new ones. Whereas previously it has been argued that transfer is only observed with essentialist categories in which the hidden properties are causally relevant for the target effect in the transfer relation, we here propose an alternative explanation, the unbroken mechanism hypothesis. This hypothesis claims that categories are transferred from a previously learned causal relation to a new causal relation when learners assume a causal mechanism linking the two relations that is continuous and unbroken. The findings of two causal learning experiments support the unbroken mechanism hypothesis.     

Functional explanation and the function of explanation

Teleological explanations (TEs) account for the existence or properties of an entity in terms of a function: we have hearts because they pump blood, and telephones for communication. While many teleological explanations seem appropriate, others are clearly not warranted--for example, that rain exists for plants to grow. Five experiments explore the theoretical commitments that underlie teleological explanations. With the analysis of [Wright, L. (1976). Teleological Explanations. Berkeley, CA: University of California Press] from philosophy as a point of departure, we examine in Experiment 1 whether teleological explanations are interpreted causally, and confirm that TEs are only accepted when the function invoked in the explanation played a causal role in bringing about what is being explained. However, we also find that playing a causal role is not sufficient for all participants to accept TEs. Experiment 2 shows that this is not because participants fail to appreciate the causal structure of the scenarios used as stimuli. In Experiments 3-5 we show that the additional requirement for TE acceptance is that the process by which the function played a causal role must be general in the sense of conforming to a predictable pattern. These findings motivate a proposal, Explanation for Export, which suggests that a psychological function of explanation is to highlight information likely to subserve future prediction and intervention. We relate our proposal to normative accounts of explanation from philosophy of science, as well as to claims from psychology and artificial intelligence.     

Uncovering deterministic causal structures: a Boolean approach

While standard procedures of causal reasoning as procedures analyzing causal Bayesian networks are custom-built for (non-deterministic) probabilistic structures, this paper introduces a Boolean procedure that uncovers deterministic causal structures. Contrary to existing Boolean methodologies, the procedure advanced here successfully analyzes structures of arbitrary complexity. It roughly involves three parts: first, deterministic dependencies are identified in the data; second, these dependencies are suitably minimalized in order to eliminate redundancies; and third, one or—in case of ambiguities—more than one causal structure is assigned to the minimalized deterministic dependencies.     

Children’s developing understanding of the relation between variable causal efficacy and mechanistic complexity

Two experiments investigated 3–4-year-olds’ ability to infer the causal mechanisms for a pair of lights. In both experiments the exterior of the two lights appeared identical. In Experiment 1, one light displayed a stable activation pattern of a single color while the other light displayed a variable pattern of activation by cycling through a series of different colors (i.e., a more varied effect). Children were asked to judge which light had a more complex internal structure. Four-year-olds were more likely to match the light with the more variable effect with a more complex internal mechanism and the light with the more stable effect with a less complex mechanism. Three-year-olds’ responses were at chance. Experiment 2 replicated this finding when the activation patterns of the two lights were described verbally but never demonstrated. Taken together, these results suggest that 4-year-olds appreciate that the variability of an object’s causal efficacy is related to the complexity of its internal mechanistic structure.     

The role of mechanism and covariation information in causal belief updating

The present study is aimed at identifying how prior causal beliefs and covariation information contribute to belief updating when evidence, either compatible or contradictory with those beliefs, is provided. Participants were presented with a cover story with which it was intended to activate or generate a causal belief. Variables related to the prior belief (the type of information, the strength of the cause-effect causal link, and how confident the participant was that the link existed) were assessed. Subsequently, participants were presented with covariational information and were asked to update their beliefs in light of the new evidence. Information reliability, prior belief's causal influence magnitude, and the cause-effect level of contingency portrayed by the new information--but not the type of the prior belief--are shown to directly determine belief updating.     

Adjusting for the correspondence bias: Effects of causal uncertainty, cognitive busyness, and causal strength of situational information

This research examined the conditions under which people who have more chronic doubt about their ability to make sense of social behavior (i.e., are causally uncertain; [Weary and Edwards, 1994] and [Weary and Edwards, 1996]) are more likely to adjust their dispositional inferences for a target’s behaviors. Using a cognitive busyness manipulation within the attitude attribution paradigm, we found in Study 1 that higher causal uncertainty predicted increased correction of dispositional inferences, but only when participants had sufficient attentional resources to devote to the task. In Study 2, we found that higher-causal uncertainty predicted greater inferential correction, but only when the additional information provided a more compelling alternative explanation for the observed behavior. Results of this research are discussed in terms of their relevance to the Causal Uncertainty (Weary & Edwards, 1994) and dispositional inference models.     

Two causal theories of counterfactual conditionals

Bayes nets are formal representations of causal systems that many psychologists have claimed as plausible mental representations. One purported advantage of Bayes nets is that they may provide a theory of counterfactual conditionals, such as If Calvin had been at the party, Miriam would have left early. This article compares two proposed Bayes net theories as models of people's understanding of counterfactuals. Experiments 1-3 show that neither theory makes correct predictions about backtracking counterfactuals (in which the event of the if-clause occurs after the event of the then-clause), and Experiment 4 shows the same is true of forward counterfactuals. An amended version of one of the approaches, however, can provide a more accurate account of these data.     

Second-order conditioning of human causal learning

This article provides the first demonstration of a reliable second-order conditioning (SOC) effect in human causal learning tasks. It demonstrates the human ability to infer relationships between a cause and an effect that were never paired together during training. Experiments 1a and 1b showed a clear and reliable SOC effect, while Experiments 2a and 2b demonstrated that first-order extinction did not affect SOC. These results were similar to those found in animal and human conditioning and suggested that a similar associative mechanism could explain these effects. However, they can also be used to look into the underlying causal mental model people build and store while they are learning this task. From a cognitive view, overall results suggest that an independent rather than a chain causal mental model is stored after second-order learning in human causal tasks.     

Children’s causal inferences from indirect evidence: Backwards blocking and Bayesian reasoning in preschoolers

Previous research suggests that children can infer causal relations from patterns of events. However, what appear to be cases of causal inference may simply reduce to children recognizing relevant associations among events, and responding based on those associations. To examine this claim, in Experiments 1 and 2, children were introduced to a “blicket detector,” a machine that lit up and played music when certain objects were placed upon it. Children observed patterns of contingency between objects and the machine’s activation that required them to use indirect evidence to make causal inferences. Critically, associative models either made no predictions, or made incorrect predictions about these inferences. In general, children were able to make these inferences, but some developmental differences between 3- and 4-year-olds were found. We suggest that children’s causal inferences are not based on recognizing associations, but rather that children develop a mechanism for Bayesian structure learning. Experiment 3 explicitly tests a prediction of this account. Children were asked to make an inference about ambiguous data based on the base rate of certain events occurring. Four-year-olds, but not 3-year-olds were able to make this inference.