Effects of harsh parenting and positive parenting practices on youth aggressive behavior: The moderating role of early pubertal timing

Prior research indicates that early pubertal timing is associated with aggressive behavior, particularly in the context of adversity as postulated in the contextual amplification hypothesis. However, few studies have examined harsh parenting as the context for the effect of early pubertal timing. Even fewer studies have tested the interactive effect of early pubertal timing and positive parenting on aggressive behavior. In this study, we tested the proposition that early pubertal timing, contrary to the general conception of it as a vulnerability, indexed susceptibility, and thus early maturing individuals were affected more by their environment in a “for better and for worse” manner. The sample consisted of 411 community‐recruited youth aged 11–12 years (51% boys, 80% African Americans). Participants reported Tanner Stages of pubertal development, aggressive behavior and harsh parenting practice of their parents. Puberty scores were standardized with groups of the same age, sex, and ethnicity, and those that scored the top one‐third were defined as early maturing individuals. Parents reported youth's aggressive behavior and their parenting practices towards the youth, including harsh parenting and positive parenting. Early pubertal timing significantly moderated the relationship between harsh/positive parenting and aggressive behavior. Specifically, harsh parenting was positively associated with aggressive behavior to a larger degree among early maturing individuals than among on‐time/late‐maturing individuals. Positive parenting was inversely associated with aggressive behavior but only among early maturing individuals. This study is the first to document support for early pubertal timing as susceptibility to the environmental influences in relation to aggressive behavior. Theoretical and intervention implications are discussed.

     

Is Neighborhood Context Differently Related to Externalizing Problems and Delinquency for Girls Compared with Boys?

Although a number of reviews of gender differences in conduct problems and delinquency exist, this paper fills a gap in reviewing neighborhood influences on gender differences in conduct problems and delinquency. These influences are known to be important for boys in childhood and adolescence, but cannot be assumed to be influential in the same manner for girls. The paper starts with several conceptualizations of the association between gender, neighborhoods and juvenile delinquency. It then addresses 4 key questions. Is residing in a disadvantaged neighborhood associated with problem behavior in girls? Are neighborhood effects independent of girls' age? Are girls in disadvantaged neighborhoods exposed to more risk factors than girls in advantaged neighborhoods? Can mediating risk factors explain gender differences in neighborhood effects on children's and adolescents' conduct problems and delinquent behavior? Answers to these questions are important to steer research and elucidate aspects of interventions that can be optimized for girls.     

Pubertal timing and self-esteem in adolescents: The mediating role of body-image and social relations

The aim of this study was to investigate the relationship between pubertal timing and self-esteem, and the mediating role of body-image, parent–adolescent interaction and peer support within this relationship. The sample comprised 526 adolescents, aged 10–13. Two measures of pubertal timing were used: perceived pubertal timing and pubertal timing based on Pubertal Development Scale (PDS). There were no differences in self-esteem between groups of adolescents with different pubertal timing based on PDS. Perceived early maturation was related to lower self-esteem in girls, and higher self-esteem in boys. We found evidence that the relationship between girls' perceived pubertal timing and self-esteem is mediated through the following variables: closeness to mother and father, and conflict with mother. In boys, mediators of the relationship between perceived pubertal timing and self-esteem are body-image and excessive control by mother. The results imply that interventions aiming to improve early maturing girls' self-esteem should focus on the interaction with their parents.     

Genetically influenced change in sensation seeking drives the rise of delinquent behavior during adolescence

Sensation seeking is associated with an increased propensity for delinquency, and emerging research on personality change suggests that mean levels of sensation seeking increase substantially from childhood to adolescence. The current study tested whether individual differences in the rate of change of sensation seeking predicted within-person change in delinquent behavior and whether genetically influenced differences in rate of personality change accounted for this association. Sensation seeking and delinquent behavior were assessed biennially between ages 10-11 and 16-17 in a nationally representative sample of 7675 youths from the National Longitudinal Study of Youth: Children and Young Adults (CNLSY). Analyses using latent growth curve modeling found that within-person change in sensation seeking was significantly and positively correlated with within-person change in delinquency from childhood to adolescence. Furthermore, behavioral genetic analyses of a subset of 2562 sibling pairs indicated that there were substantial genetic influences on both initial levels of sensation seeking and change in sensation seeking during early adolescence, with over 80% of individual differences in change due to genetic factors. Finally, these genetically driven increases in sensation seeking were most important for predicting increases in delinquency, whereas environmental paths between sensation seeking and delinquency were not significant. These results suggest that developmental changes in delinquent behaviors during adolescence are driven by a genetically governed process of personality change.     

Self‐Reported Family Socioeconomic Status,the 5‐HTTLPR Genotype,and Delinquent Behavior in a Community‐Based Adolescent Population

Twin and adoption studies have demonstrated a significant contribution of both genetic and environmental factors to antisocial and delinquent behavior. Associations have been reported between the serotonin transporter (5‐HTT) and aggression, and between socioeconomic status (SES), aggression, and serotonergic functions of the brain. We aimed to investigate associations between the 5‐HTTLPR genotype and family SES in relation to delinquent behavior among adolescents. A total of 1,467 17‐ to 18‐year‐old students in the county of Västmanland, Sweden, anonymously completed a questionnaire and gave a saliva sample. Family SES had a U‐shaped relation to delinquency, where adolescents with low and high family SES were the most delinquent. There were curvilinear interactions between the 5‐HTTLPR genotype and family SES in relation to delinquency. Among individuals having high family SES, boys with the LL (homozygous for the long allele) or LS (heterozygous) genotypes and girls with the SS (homozygous for the short allele) or LS (heterozygous) genotypes showed the highest delinquency scores. Among individuals having low family SES, boys with the LL (homozygous for the long allele) genotype and girls with the LS (heterozygous) genotype showed the highest delinquency scores. The present study suggests evidence for an interaction between family SES and the 5‐HTTLPR genotype in relation to juvenile delinquency. Aggr. Behav. 39:52‐63, 2013. © 2012 Wiley Periodicals, Inc.     

青春期开始时间早晚对初中女生自我概念的影响

目的:探讨青春期开始时间早晚对初中女生自我概念发展的影响。方法:整群抽取初一至初三年龄12-14岁的女生共658人,采用青春期发育量表、田纳西自我概念量表进行测量。结果:①绝大多数12至14岁的女生青春期发育状态处于青春期中和青春期晚期（二者合占90%）;②年龄×青春期发育水平的方差分析显示,在自我概念各因子分及总分上,年龄和青春期发育水平的主效应均不显著;③在家庭自我、社会自我、自我认同、自我行动以及自我概念总分上年龄与青春期发育水平交互效应显著,高年龄低青春期发展水平和低年龄高青春期发展水平的初中女生的得分均较低。结论:青春期开始时间早晚可能对初中女生的自我概念的发展有一定影响,青春期开始时间晚或早均不利于女生自我概念的发展。     

Differences in Nonshared Individual,School, and Family Variables Between Delinquent and Nondelinquent Hmong Adolescents

The purpose of the current study was to determine whether there were differences between Hmong immigrant siblings on multiple nonshared individual, school, and family variables. Participants were 58 sibling pairs (29 delinquent pairs and 29 nondelinquent pairs), who ranged in age from 13 to 21 years. There were 11 pairs of boys, 3 pairs of girls, and 15 pairs of opposite-sex siblings. Results of a multivariate analysis of variance indicated that delinquent and nondelinquent sibling groups were significantly different in participation in organized activities, antisocial attitudes, delinquent behaviors, school truancy, school performance, and parents' labeling. The findings have implications for future research on sibling delinquency in Hmong immigrant families and for program development and delivery.     

Differences in nonshared individual, school, and family variables between delinquent and nondelinquent Hmong adolescents

The purpose of the current study was to determine whether there were differences between Hmong immigrant siblings on multiple nonshared individual, school, and family variables. Participants were 58 sibling pairs (29 delinquent pairs and 29 nondelinquent pairs), who ranged in age from 13 to 21 years. There were 11 pairs of boys, 3 pairs of girls, and 15 pairs of opposite-sex siblings. Results of a multivariate analysis of variance indicated that delinquent and nondelinquent sibling groups were significantly different in participation in organized activities, antisocial attitudes, delinquent behaviors, school truancy, school performance, and parents' labeling. The findings have implications for future research on sibling delinquency in Hmong immigrant families and for program development and delivery.     

Genetic and environmental influences on pubertal development: longitudinal data from Finnish twins at ages 11 and 14

To study sources of individual differences in pubertal development, the authors fit a sex-limitation common factor model to data reported, at ages 11 and 14 years, by 1,891 twin pairs on items that comprise the Pubertal Development Scale (PDS; A. C. Petersen, L. Crockett, M. Richards, & A. Boxer, 1988). The model divides variation into a general pubertal factor and item-specific variation and, in addition, decomposes it into constituent sources. In both boys and girls, genetic influences made the largest contribution to variance common to PDS items. Genetic and nonshared environmental factors accounted for variation specific to PDS items in boys, whereas for girls, common environmental influences were added for growth spurt and menarcheal status. For both common and item-specific variation, genetic effects were partially sex specific. Subsidiary analyses found accelerated maturation in both boys and girls who at age 14 were reared in father-absent homes.     

Elevated social anxiety among early maturing girls

Adolescence is a key period in terms of the development of anxiety psychopathology. An emerging literature suggests that early pubertal maturation is associated with enhanced vulnerability for anxiety symptomatology, although few studies have examined this association with regard to social anxiety. Accordingly, the current study was designed to further elucidate the relation between pubertal timing and social anxiety, with a focus on clarifying the role of gender. Participants were 138 adolescents (ages 12-17 years) recruited from the general community. Level of social anxiety was examined as a function of gender and within-sample pubertal timing. As expected, early maturing girls evidenced significantly higher social anxiety, compared with on-time girls and early maturing boys, and no other differences were found as a function of gender or developmental timing. Findings and future directions are discussed in terms of forwarding developmentally sensitive models of social anxiety etiology and prevention.     

Sources of structure: genetic, environmental, and artifactual influences on the covariation of personality traits

The phenotypic structure of personality traits has been well described, but it has not yet been explained causally. Behavior genetic covariance analyses can identify the underlying causes of phenotypic structure; previous behavior genetic research has suggested that the effects from both genetic and nonshared environmental influences mirror the phenotype. However, nonshared environmental effects are usually estimated as a residualterm that may also include systematic bias, such as that introduced by implicit personality theory. To reduce that bias, we supplemented data from Canadian and German twin studies with cross-observer correlations on the Revised NEO Personality Inventory. The hypothesized five-factor structure was found in both the phenotypic and genetic/familial covariances. When the residual covariance was decomposed into true nonshared environmental influences and method bias, only the latter showed the five-factor structure. True nonshared environmental influences are not structured as genetic influences are, although there was some suggestion that they do affect two personality dimensions, Conscientiousness and Love. These data reaffirm the value of behavior genetic analyses for research on the underlying causes of personality traits.     

A longitudinal study of pubertal timing and extreme body change behaviors among adolescent boys and girls

Recent studies have examined the prevalence of disordered eating and other health risk behaviors among adolescent boys and girls. However, these studies generally have not examined predictors of these behaviors, and have not embedded the investigations within a theoretical framework. This study employed a longitudinal design to evaluate the validity of a biosocial model in explaining health risk behaviors related to extreme body change strategies (disordered eating, exercise dependence, use of food supplements, steroid use) among adolescents. The participants were 430 adolescent boys (mean age = 13.33 years) and 451 adolescent girls (mean age = 13.28 years) who completed measures of pubertal timing, perceived popularity with peers, body dissatisfaction, focus on sport, involvement in competitive sport, strategies to lose weight, strategies to increase muscle, disordered eating, use of food supplements and steroids, and exercise dependence. It was found that both early and late maturing girls were at greatest risk of engaging in health risk behaviors, whereas boys demonstrated more variability in the relationship between pubertal timing and extreme body change behaviors. The results of this study provide important insights into the role of pubertal development in the adoption of extreme body change behaviors among adolescents.     

Linked Lives, Faith, and Behavior: Intergenerational Religious Influence on Adolescent Delinquency

Research on religion and delinquency has generally concluded that only minor forms of delinquency are affected by religious commitments. However, parents have not often been the focus of religion and delinquency research. This study explores the influence of parental religious identity and behavior on the serious delinquency of adolescent children. This analysis tested an intergenerational model of religious influence on delinquent behavior. Results suggest parental religious devotion protects girls considerably better than boys. In fact, it may amplify delinquency among boys, at least when controlling for other important influences such as autonomy and family satisfaction. Parents' conservative Protestant affiliation displays consistent negative direct effects on delinquency, but little indirect influence. This study reinforces the importance of considering linked lives in the development of youth, as well as the need to assess both direct and indirect religious influences.     

Sex differences in the causes of self-reported adolescent delinquency

Sex differences in the causes of self-reported adolescent delinquency were examined in full and half siblings born to a nationally representative sample of women in the United States. Qualitative sex differences in the genes that influence delinquency were not detected. Similarly, the proportions of variance in both aggressive and nonaggressive delinquency attributable to genetic and environmental influences did not differ significantly between girls and boys. Nonetheless, total variance in delinquency was greater among boys, and a scalar sex-differences model suggested that genetic and environmental influences on delinquency have less effect on population variation in delinquency among girls. Similarly, a test of the polygenic multiple threshold model suggested that girls require greater causal liability for the expression of delinquency than boys.     

The ABCs of depression: integrating affective, biological, and cognitive models to explain the emergence of the gender difference in depression

In adulthood, twice as many women as men are depressed, a pattern that holds in most nations. In childhood, girls are no more depressed than boys, but more girls than boys are depressed by ages 13 to 15. Although many influences on this emergent gender difference in depression have been proposed, a truly integrated, developmental model is lacking. The authors propose a model that integrates affective (emotional reactivity), biological (genetic vulnerability, pubertal hormones, pubertal timing and development) and cognitive (cognitive style, objectified body consciousness, rumination) factors as vulnerabilities to depression that, in interaction with negative life events, heighten girls' rates of depression beginning in adolescence and account for the gender difference in depression.     

Environmental Contributions to Adolescent Delinquency: A Fresh Look at the Shared Environment

Few genetically-informative studies have attempted to explicitly identify the shared environmental (i.e., those environmental influences that contribute to sibling similarity) factors now known to contribute to adolescent delinquency. The current study therefore examined whether the parent-child relationship served as one source of these shared environmental influences. Participants were 610 adoptive and biological families from the Sibling Interaction and Behavior Study (SIBS). Parents and adolescents reported on their parent-child conflict and parental involvement with child, and adolescents reported on their own delinquent behaviors. We employed structural equation modeling and supplementary multilevel modeling, finding consistent evidence that the association between delinquency and the parent-child relationship is at least partially shared environmental in origin. Such findings provide an important extension of previous twin studies, as they suggest that passive genotype-environment correlations do not explain earlier findings of shared environmental influences on this association.     

Genetic and environmental stability differs in reactive and proactive aggression

The aim of this study was to examine stability and change in genetic and environmental influences on reactive (impulsive and affective) and proactive (planned and instrumental) aggression from childhood to early adolescence. The sample was drawn from an ongoing longitudinal twin study of risk factors for antisocial behavior at the University of Southern California (USC). The twins were measured on two occasions: ages 9–10 years (N=1,241) and 11–14 years (N=874). Reactive and proactive aggressive behaviors were rated by parents. The stability in reactive aggression was due to genetic and nonshared environmental influences, whereas the continuity in proactive aggression was primarily genetically mediated. Change in both reactive and proactive aggression between the two occasions was mainly explained by nonshared environmental influences, although some evidence for new genetic variance at the second occasion was found for both forms of aggression. These results suggest that proactive and reactive aggression differ in their genetic and environmental stability, and provide further evidence for some distinction between reactive and proactive forms of aggression. Aggr. Behav. 35:437–452, 2009. © 2009 Wiley‐Liss, Inc.     

Interactive Effects of Psychosocial Stress and Early Pubertal Timing on Youth Depression and Anxiety: Contextual Amplification in Family and Peer Environments

While off-time pubertal development has emerged as a potential risk factor for both symptoms of depression and anxiety in youth, the literature is mixed and inconsistent as to (1) how early versus late pubertal timing confers risk for both boys and girls, (2) if the conferred risk is distinct between symptoms of anxiety and depression, and (3) under what social contexts (e.g., family environment, peer relationships) off-time pubertal development may emerge as a potent risk factor for these symptoms. The present study examined the impact of perceived pubertal timing on symptoms of anxiety and depression in two distinct psychosocial contexts: parent’s perceptions of their own harsh parenting and parent’s perceptions of their child’s peer problems. The sample consisted of 412 parents (M = 38.6 years old, SD = 7.8, 60.4 % mothers) of children between the ages of 8 and 17 (M = 12.13, SD = 2.97, 45.4 % girls). All constructs were assessed by parent reports. Linear multiple regression analyses revealed that the interaction between earlier pubertal timing and greater peer problems was significantly related to higher youth depressive and anxiety symptoms. The interaction between earlier pubertal timing and greater harsh discipline was significantly related to higher youth anxiety but not depressive symptoms. Youth gender did not qualify findings. Results suggest that the contextual amplification process of early pubertal timing may occur in both high stress family and peer environments and impact both girls and boys.     

Adverse Housing Conditions and Early‐Onset Delinquency

Housing constitutes an important health resource for children. Research has revealed that, when housing conditions are unfavorable, they can interfere with child health, academic performance, and cognition. Little to no research, however, has considered whether adverse housing conditions and early‐onset delinquency are significantly associated with one another. This study explores the associations between structural and non‐structural housing conditions and delinquent involvement during childhood. Data from the Fragile Families and Child Wellbeing Study (FFCWS) were employed in this study. Each adverse housing condition was significantly associated with early‐onset delinquency. Even so, disarray and deterioration were only significantly linked to early delinquent involvement in the presence of health/safety hazards. The predicted probability of early‐onset delinquency among children exposed to housing risks in the presence of health/safety hazards was nearly three times as large as the predicted probability of early‐onset delinquency among children exposed only to disarray and/or deterioration, and nearly four times as large as the predicted probability of early‐onset delinquency among children exposed to none of the adverse housing conditions. The findings suggest that minimizing housing‐related health/safety hazards among at‐risk subsets of the population may help to alleviate other important public health concerns—particularly early‐onset delinquency. Addressing household health/safety hazards may represent a fruitful avenue for public health programs aimed at the prevention of early‐onset delinquency.     

Delinquent peer affiliation and conduct problems: A twin study

Many putative environmental risks correlate with individuals' genotypes. The association between delinquent peer affiliation and conduct problems may occur because of shared genetic liability. Five hundred fifty three monozygotic and 558 dizygotic twin pairs, aged 11 to 18 years, were assessed for delinquent peer affiliation and conduct problems. The authors investigated whether genes contribute to both delinquent peer affiliation and the correlation between delinquent peer affiliations and conduct problems. Delinquent peer affiliation was influenced by genetic, shared environmental, and nonshared environmental factors; genetic factors also contributed to the correlation between delinquent peer affiliations and conduct problems, providing evidence for genotype-environment correlation. The magnitude of the genetic variance of conduct problems was contextually dependent on levels of delinquent peer affiliation and was greater at higher levels of delinquent peer affiliation.