脑成像在心理学研究领域的价值

现在普遍使用的脑成像技术给心理学研究增加了新的数据和资料。和任何新的方法一样,我们需要决定如何以适当的方式应用这项技术。这项技术如何以现有的方法所不能的方式帮助回答理论问题？这项技术最好是作为因变量还是作为预测变量来使用？它如何与其它感兴趣的心理变量相关？这种新的成像技术有助于我们了解大脑的运作及其与心理学的关系。研究人员需要弄清楚如何利用这项技术提供的信息加深对心理现象的理解。     

Understanding Biology in Religious Experience: The Biogenetic Structuralist Approach of Eugene d'Aquili and Andrew Newberg

What are the biological bases of religious experience? Are there biological constraints upon or determinants of religious narratives and practices? How does understanding the biology of religious experience inform the ongoing reconstruction of religious rituals and myths? In The Mystical Mind, Eugene d’Aquili and Andrew Newberg address these central questions and others from a distinct perspective called biogenetic structuralism. They propose a model of how brain activity gives rise to mystical experiential states, examine how neurobiological responses to rhythmic behavior form religious ritual, and point toward the development of a megatheology, or a theological system appealing to the widest scope of religious world‐views. This paper is a critical review of d’Aquili and Newberg's exciting work.     

The Impact of Motor Activity and Inactivity on the Brain: Implications for the Prevention and Treatment of Nervous- System Disorders

ABSTRACT— Since Donald Hebb's pioneering observations in the 1940s, much research has focused on the effects of variations in physical activity and environmental complexity on behavioral performance and brain structure. Beneficial effects on brain health have been linked to physical fitness, skilled training, and exposure to complex environments, though in rodents these effects may be negated by sudden changes in social structure. Such manipulations can alleviate the deficits associated with several nervous-system disorders and aging. But how increased activity produces its beneficial effects is still not fully understood. How does unskilled physical activity (e.g., repetitive exercise) compare to training in skilled activities or exposure to complex environments? In injury states, is task-specific training a better rehabilitative strategy than general exercise? How do changes in motor activity affect specific brain regions, and can the intensity and timing of therapeutic movement be adjusted to produce optimal outcomes? Are the beneficial effects of motor enrichment banked over periods of inactivity and can they be called upon with booster training to treat a later neurological disorder? Are there circumstances in which increased activity is harmful? Enrichment of physical activity shows promise as an easy and healthful means for improving or restoring brain function, and questions like these are now being investigated so that the full potential of increased activity may be harnessed.     

Young children''s naive theory of biology

This article aimed at investigating the nature of young children's naive theory of biology by reviewing a large number of studies conducted in our laboratories. More specifically, we tried to answer the following five critical questions. What components does young children's knowledge system for biological phenomena (or naive biology) have? What functions does it have in children's lives? How is it acquired in ontogenesis? How does its early version change as children grow older? Is it universal across cultures and through history? We propose that young children's biological knowledge system has at least three components, that is, knowledge needed to specify the target objects of biology, ways of inferring attributes or behaviors of biological kinds, and a non-intentional causal explanatory framework, and that these three constitute a form of biology, which is adaptive in children's lives. We also claim that the core of naive biology is acquired based on specific cognitive constraints as well as the general mechanism of personification and the resultant vitalistic causality, but it is differently instantiated and elaborated through activity-based experiences in the surrounding culture.     

A Clinical-Empirical Model of Personality: Life after the Mischelian Ice Age and the NEO-Lithic Era

ABSTRACT A theory of personality should lead to both accurate prediction and interpretive understanding. Aside from its empirical uses, a personality theory should provide a grammar that allows personality psychologists to infer meaning from overt behavior with more sophistication than a layperson, and the best laboratory for testing the interpretive utility of a personality theory remains the clinic. With respect to the appropriate data for constructing and evaluating theories of personality, an overreliance on questionnaire data is problematic for several reasons: It assumes that understanding people requires no training, it mistakes research on the conscious self-concept for research on personality, it conflates implicit and explicit knowledge, it fails to address defensive biases, and it lacks interrater reliability. Consideration of both empirical and clinical data points to three questions that define the elements of personality necessary for a comprehensive assessment of an individual: (a) What psychological resources–cognitive, affective, and behavioral dispositions–does the individual have at his or her disposal? (b) What does the person wish for, fear, and value, and how do these motives combine and conflict? (c) How does the person experience the self and others, and to what extent can the individual enter into intimate relationships?     

Children's and adults' understanding of the impact of nutrition on biological and psychological processes

Four studies examined children's and adults' beliefs about the impact of nutrition on growth and mood states. In Studies 1 and 2, 271 participants (preschoolers through adults) judged the impact of healthy and unhealthy nutrition on height and weight. In Studies 3 and 4, 267 participants judged the impact of healthy and unhealthy nutrition on positive and negative mood states. The results suggest that young children demonstrate a co‐existence of an ontologically distinct theory of biology as well as a theory of cross‐domain interaction when reasoning about the impact of food on biological and psychological processes.     

When the rules are reversed: Action-monitoring consequences of reversing stimulus–response mappings

How does switching tasks affect our ability to monitor and adapt our behavior? Largely independent lines of research have examined how individuals monitor their actions and adjust to errors, on the one hand, and how they are able to switch between two or more tasks, on the other. Few studies, however, have explored how these two aspects of cognitive?Cbehavioral flexibility interact. That is, how individuals monitor their actions when task rules are switched remains unknown. The present study sought to address this question by examining the action-monitoring consequences of response switching??a form of task switching that involves switching the response that is associated with a particular stimulus. We recorded event-related brain potentials (ERPs) while participants performed a modified letter flanker task in which the stimulus?Cresponse (S?CR) mappings were reversed between blocks. Specifically, we examined three ERPs??the N2, the error-related negativity (ERN), and the error positivity (Pe)??that have been closely associated with action monitoring. The findings revealed that S?CR reversal blocks were associated with dynamic alterations of action-monitoring brain activity: the N2 and ERN were enhanced, whereas the Pe was reduced. Moreover, participants were less likely to adapt their posterror behavior in S?CR reversal blocks. Taken together, these data suggest that response switching results in early enhancements of effortful control mechanisms (N2 and ERN) at the expense of reductions in later response evaluation processes (Pe). Thus, when rules change, our attempts at control are accompanied by less attention to our actions.     

Brain neuron gene expression in the organization of innate and acquired behaviors

In a series of experiments using rats and rabbits, it was determined that regulatory peptides played a role in the mechanisms of learned behavior. Learning a new habit as a conditional avoidance response was accompanied by a considerable increase in brain DNA synthesis, which in turn could result in RNA and protein synthesis activation. The results suggest the following interdependent molecular phenomena: 1) DNA synthesis activation; 2) genome expression of a certain type of oligopeptides; 3) regulation of genome expression of proteins by chromatin nonhistone proteins; 4) regulation of gene expression mechanisms by brain regulatory peptides.     

消费者一定偏爱“笑脸”吗?产品外观中的表情元素对消费者的影响

表情是产品拟人化外观设计中重要元素。产品正面或负面的拟人化“表情” (表达正面或负面情感的表情)会影响到消费者对产品的感知。人际交往相关理论验证了正面表情对消费者的积极影响, 并认为负面表情不利于消费者与产品的互动。但是在现实市场中我们却看到, 消费者有时却更喜爱展现负面表情的产品, 营销领域的产品拟人化研究鲜有关注到产品表情的错位影响。具体而言, 在拟人化营销研究领域, 以下三个研究问题尚待探讨: 产品表情有怎样的具体体现及市场效用; 产品表情对消费者行为和态度影响的心理机制为何; 以及来自于消费者特质和产品因素的调节效应。回答以上问题能够进一步拓展和丰富营销领域的拟人化理论, 同时也将从实践上为产品外观设计提供建议和指导。     

心理学在社会服务体系中作用的思考——以复原力建设为例

在社会心理服务体系建设中, 心理学到底起着什么样的作用?怎样才能更好地构建全面系统的多主体社会心理服务体系?在社会心理服务体系建设中如何推动心理学自身的发展?这些问题一直受到广大心理学者的关注。以复原力为例, 通过对个体、团队以及社区心理复原力建设路径的理论总结, 加上实践案例, 以期为社会心理服务体系建设提供知识借鉴与参考, 并反思心理学在此过程中的作用与角色。心理学专业研究团队可以通过整合其在社会支持体系中的应用, 推动心理学在社会服务体系中的应用。     

Molecular Lamarckism: On the Evolution of Human Intelligence

In modern times, Lamarck's view of evolution, based on inheritance of acquired traits has been superseded by neo-Darwinism, based on random DNA mutations. This article begins with a series of observations suggesting that Lamarckian inheritance is in fact operative throughout Nature. I then launch into a discussion of human intelligence that is the most important feature of human evolution that cannot be easily explained by mutational selection. Thus, we are smarter than demanded by our evolutionary experience as hunter-gatherers. The difficulty lies in the inability of neo-Darwinism to satisfactorily answer the following question: How can a large energy-costly set of genes, each member of which has little apparent benefit when first created individually, all gather into a permanent existence within a short time period in each and every member of a small population (that was dispersed and geographically isolated over a huge planet) who had a low reproductive output, a low rate of beneficial mutations, and a low level of genetic contact? The article concludes with a speculative but far-reaching epigenetic theory of intelligence that does not require DNA mutation as the exclusive source of evolutionary change. Instead, cranial feedback relating brain chemistry, as affected by brain activity including education, with the genome. When it comes to the fast rate of evolution, and the dissemination of the intelligence trait worldwide, cranial feedback could make all the difference.     

Environmental and Behavioral Influences on Gene Activity

The central dogma of molecular biology holds that 'information' flows from the genes to the structure of the proteins that the genes bring about through the formula DNA → RNA → protein. In this view, a set of master genes activates the DNA necessary to produce the appropriate proteins that the organism needs during development. In contrast to this view, probabilistic epigenesis holds that necessarily there are signals from the internal and external environment that activate DNA to produce the appropriate proteins. To support this view, I review a substantial body of evidence showing that external environmental influences on gene activation are normally occurring events in a large variety of organisms, including humans. This demonstrates how genes and environments work together to produce functional organisms, thus extending the model of probabilistic epigenesis.     

Evolutionary Psychology: The Wheat and the Chaff

Evolutionary approaches are on the rise in the social sciences and have the potential to bring an all–encompassing conceptual framework to the study of human behavior. Together with neuroscience, which is digging the grave of mind–body dualism, evolutionary psychology is bound to undermine the still reigning human–animal dualism. If a Darwinian reshaping of the social sciences seems inevitable, even desirable, this should not be looked at as a hostile takeover. The underlying theme of this essay is that it is time for psychologists to join the Darwinian revolution, yet the essay also critically reviews current evolutionary psychology. It questions the loose application of adaptationist thinking and the fragmentation of the genome, behavior, and the brain. From biology we learn that not every species–typical trait is necessarily advantageous, and from neuroscience we learn that not every psychological ability or tendency necessarily needs to have its own specialized brain circuitry. But even if the concept of adaptation is hard to apply, psychologists would do well to start looking at human behavior in the light of evolution.     

Danger! Psychotherapists at work

The rapid growth of psychotherapeutic movements in recent years has led to a free market. Vast numbers of psychotherapists of different theoretical persuasions are now on offer. While such a choice has its obvious advantages, it creates bewildering confusions in the minds of potential clients. How can the clients tell which psychotherapist or psychotherapeutic entepise to opt for? How can they know whether they have made an appropriate choice? How do they know which therapy works best under which conditions? Are there a set of criteria laid down which might guide the client in making a judicious choice? Can they be assured that after a senes of agreed sessions, they will get better? These are extremely serious issues. The paper discusses the practical and ethical implications underlying these issues and offers specific suggestions and guidelines as to how the clients might safeguard their own interests when it comes to choosing an appropriate psychotherapist.     

Psychosis and autism as diametrical disorders of the social brain

Autistic-spectrum conditions and psychotic-spectrum conditions (mainly schizophrenia, bipolar disorder, and major depression) represent two major suites of disorders of human cognition, affect, and behavior that involve altered development and function of the social brain. We describe evidence that a large set of phenotypic traits exhibit diametrically opposite phenotypes in autistic-spectrum versus psychotic-spectrum conditions, with a focus on schizophrenia. This suite of traits is inter-correlated, in that autism involves a general pattern of constrained overgrowth, whereas schizophrenia involves undergrowth. These disorders also exhibit diametric patterns for traits related to social brain development, including aspects of gaze, agency, social cognition, local versus global processing, language, and behavior. Social cognition is thus underdeveloped in autistic-spectrum conditions and hyper-developed on the psychotic spectrum.;>We propose and evaluate a novel hypothesis that may help to explain these diametric phenotypes: that the development of these two sets of conditions is mediated in part by alterations of genomic imprinting. Evidence regarding the genetic, physiological, neurological, and psychological underpinnings of psychotic-spectrum conditions supports the hypothesis that the etiologies of these conditions involve biases towards increased relative effects from imprinted genes with maternal expression, which engender a general pattern of undergrowth. By contrast, autistic-spectrum conditions appear to involve increased relative bias towards effects of paternally expressed genes, which mediate overgrowth. This hypothesis provides a simple yet comprehensive theory, grounded in evolutionary biology and genetics, for understanding the causes and phenotypes of autistic-spectrum and psychotic-spectrum conditions.     

The historical debate on brain and legal responsibility--revisited

The actual discussion on brain research, free will, and legal responsibility repeats all the arguments of a very similar discussion in central Europe 100 years ago, around 1900. Already back then the idea that human perception, emotions, thinking and behavior have a material basis was not new. It is conditioned and limited by the structure and workings of the brain. This applies to sane and sick brains alike. In this debate criminality was made a topic of biology by interpreting it as a symptom of genetic "degeneration". In regard to brain damage in humans with socially deviant behavior, psychiatry claimed responsibility for a large proportion of criminals. This article lays out some important positions of psychiatry and jurisprudence in this debate on the foundations of legal responsibility and their impact up to now. The debate leads to the conclusion that human behavior cannot be described adequately in physical terms of cause and effect. The psychological approach to describe reasons, motives and intentions is substantially more appropriate. This method is also accepted by criminal law.     

Linear genetics,non-linear epigenetics: Complementary approaches to understanding complex diseases

Recent discoveries and rediscoveries in molecular and cell biology, in population and evolutionary biology, and in disease natural history raise new doubts about the ability of genetic analysis alone to predict multifactorial (polygenic) human diseases and other complex phenotypes. These doubts serve to redirect our attention to epigenetic regulation as a second informational system in parallel with the genome. Epigenetic regulation is now viewed by many biologists as a process that includes mechanisms capable of constraining the genome and providing for new patterns of gene expression. Epigenetic networks, both intra- and inter-cellular, provide a basis for nonlinear and chaotic views of cellular and tissue level differentiation and organization, and thus provide a more dynamic approach to understanding the creation of complex phenotypes, even from isogeneic conditions. The reality of regulatory networks within cells inserted, as it were, between genome and phenome, also helps explain the difficulties now encountered when prediction and diagnosis of complex disease omits epigenetic considerations and depends entirely on gene causality. For a complete reference guide, see reviews by R.C. Strohman cited below.     

Synaptogenesis and heritable aspects of executive attention

In humans, changes in brain structure and function can be measured non-invasively during postnatal development. In animals, advanced optical imaging measures can track the formation of synapses during learning and behavior. With the recent progress in these technologies, it is appropriate to begin to assess how the physiological processes of synapse, circuit, and neural network formation relate to the process of cognitive development. Of particular interest is the development of executive function, which develops more gradually in humans. One approach that has shown promise is molecular genetics. The completion of the human genome project and the human genome diversity project make it straightforward to ask whether variation in a particular gene correlates with variation in behavior, brain structure, brain activity, or all of the above. Strategies that unify the wealth of biochemical knowledge pertaining to synapse formation with the functional measures of brain structure and activity may lead to new insights in developmental cognitive psychology.